about
Down-regulation of epithelial cadherin is required to initiate metastatic outgrowth of breast cancer.p130Cas is required for mammary tumor growth and transforming growth factor-beta-mediated metastasis through regulation of Smad2/3 activity.STAT3 and epithelial-mesenchymal transitions in carcinomasCXCL12 chemokine expression and secretion regulates colorectal carcinoma cell anoikis through Bim-mediated intrinsic apoptosis.Fibroblast growth factor receptor splice variants are stable markers of oncogenic transforming growth factor β1 signaling in metastatic breast cancers.Detection of Lysyl Oxidase-Like 2 (LOXL2), a Biomarker of Metastasis from Breast Cancers Using Human Blood Samples.Transforming growth factor-β-induced epithelial-mesenchymal transition facilitates epidermal growth factor-dependent breast cancer progressionThe antitumorigenic function of EGFR in metastatic breast cancer is regulated by expression of Mig6.Deptor enhances triple-negative breast cancer metastasis and chemoresistance through coupling to survivin expressionβ3 integrin-EGF receptor cross-talk activates p190RhoGAP in mouse mammary gland epithelial cells.In vivo dual substrate bioluminescent imaging.Integrin-mediated resistance to epidermal growth factor receptor-targeted therapy: an inflammatory situation.Longitudinal Bioluminescent Quantification of Three Dimensional Cell Growth.Upregulated WAVE3 expression is essential for TGF-β-mediated EMT and metastasis of triple-negative breast cancer cells.FGFR signaling maintains a drug persistent cell population following epithelial-mesenchymal transition.Selective Inhibition of STAT3 Phosphorylation Using a Nuclear-Targeted Kinase Inhibitor.Covalent Targeting of Fibroblast Growth Factor Receptor Inhibits Metastatic Breast Cancer.Rho activation regulates CXCL12 chemokine stimulated actin rearrangement and restitution in model intestinal epithelia.Phosphoproteins in extracellular vesicles as candidate markers for breast cancer.Regulation of epithelial-mesenchymal transition and metastasis by TGF-β, P-bodies, and autophagy.Inhibition of pyruvate carboxylase by 1α,25-dihydroxyvitamin D promotes oxidative stress in early breast cancer progression.Analytical Pipeline for Discovery and Verification of Glycoproteins from Plasma-Derived Extracellular Vesicles as Breast Cancer Biomarkers.Pyruvate carboxylase supports the pulmonary tropism of metastatic breast cancer.The paradoxical functions of EGFR during breast cancer progressionBiased signaling downstream of epidermal growth factor receptor regulates proliferative versus apoptotic response to ligandPBRM1 Regulates Stress Response in Epithelial CellsAutocrine Fibronectin Inhibits Breast Cancer MetastasisTransglutaminase-2 facilitates extracellular vesicle-mediated establishment of the metastatic nicheHyaluronic acid, CD44 and RHAMM regulate myoblast behavior during embryogenesisSpleen Tyrosine Kinase-Mediated Autophagy Is Required for Epithelial-Mesenchymal Plasticity and Metastasis in Breast CancerApplication of a Substrate-Mediated Selection with c-Src Tyrosine Kinase to a DNA-Encoded Chemical LibraryThe Dynamic Relationship of Breast Cancer Cells and Fibroblasts in Fibronectin Accumulation at Primary and Metastatic Tumor Sites
P50
Q30502431-32D61D93-CD7E-4020-9AB6-4F35CF289F15Q33553472-FBEFA9E5-DC99-4094-8C4A-E2EAC4901E35Q33623947-0EF98F33-E308-4D18-92C3-38689ABE15C9Q33705126-28402B13-F945-4432-B816-D8BA3C2C738CQ33739802-FD730231-0DDC-48F4-A966-795A6534FAEDQ33859309-C6764CA7-2209-4BC9-A144-5F51E1F605F3Q34789337-150145EC-90B6-4985-9E41-CEC114D8A0BFQ35023922-79FA32B6-7FF0-4C9F-B051-59595640AA94Q35214539-B9F4FCC0-0622-4E86-988B-91365F9E2948Q35551725-230A5A4F-B76F-415E-BB87-9E3FEC64FE74Q35581797-64542DFD-B80E-4385-B1C7-5EE01DFBE9C5Q36507879-59C7804D-FA22-4697-9395-F3912DA9554DQ37118883-1A839939-0BA8-420E-9165-EF024A4AA9F3Q37464261-69DEEB89-5A39-4523-A56E-1475432AEB96Q37696325-C1E77F9F-93AC-4824-B328-108A5BCC7EB8Q38634605-3265859C-82B4-4CB3-9390-2A81810A063BQ38760333-F2CA8093-85CB-4586-A398-DAD2C21B0F08Q40119156-527DA4BB-71C9-408E-AB08-BE651A4E7788Q41710047-D8714FE5-DA34-4B66-A03B-C2EA2740CB38Q47120340-37D2F341-C82D-455D-9CFB-E08BE7482D42Q47661449-29DB5231-2872-48C6-B5AA-F8BA3281AFAAQ52599203-ACC35435-2C1B-4A57-97AA-F0C5239C382CQ55690571-0263C6A5-EC2C-45A0-8EB1-BDBAAF05E860Q57118645-3D53300A-548D-4FD9-8E93-92867D97358BQ58700321-2C6E95E0-D74A-4827-B4A7-BFAD2517A68AQ64085735-385E4CFD-FD0B-4637-A2AE-EE8ADB095F85Q89206302-6859491B-B4E7-475B-9276-27D5FAB9A2E6Q89701169-176C0A33-36DD-4740-8BD5-986AB07700AEQ91064074-DA21DAEC-5A19-4C15-A4DB-ECB58AC38F32Q91408660-3877A048-A405-42BB-BB24-80F08E4294FCQ92315719-CD49163C-4129-4DDB-AE36-F0E3859098FFQ95327182-23969B17-0855-4512-B922-D8AA0319A1FD
P50
description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Michael K Wendt
@en
Michael K Wendt
@nl
type
label
Michael K Wendt
@en
Michael K Wendt
@nl
prefLabel
Michael K Wendt
@en
Michael K Wendt
@nl
P108
P108
P31
P496
0000-0002-3665-7413