p130Cas is required for mammary tumor growth and transforming growth factor-beta-mediated metastasis through regulation of Smad2/3 activity.
about
Cas proteins: dodgy scaffolding in breast cancerCAS proteins in normal and pathological cell growth controlNEDD9 crucially regulates TGF-β-triggered epithelial-mesenchymal transition and cell invasion in prostate cancer cells: involvement in cancer progressivenessCas and NEDD9 Contribute to Tumor Progression through Dynamic Regulation of the Cytoskeleton.NSP-CAS Protein Complexes: Emerging Signaling Modules in CancerAssociation of the breast cancer antiestrogen resistance protein 1 (BCAR1) and BCAR3 scaffolding proteins in cell signaling and antiestrogen resistanceLIM and SH3 protein-1 modulates CXCR2-mediated cell migration.Novel regulatory role of neuropilin-1 in endothelial-to-mesenchymal transition and fibrosis in pancreatic ductal adenocarcinomaBreast cancer anti-estrogen resistance 3 inhibits transforming growth factor β/Smad signaling and associates with favorable breast cancer disease outcomesDown-regulation of epithelial cadherin is required to initiate metastatic outgrowth of breast cancer.Fibroblast growth factor receptor splice variants are stable markers of oncogenic transforming growth factor β1 signaling in metastatic breast cancers.P130cas is required for TGF-β1-mediated epithelial-mesenchymal transition in lung cancerTTRAP is a novel component of the non-canonical TRAF6-TAK1 TGF-β signaling pathwayBCAR1 protein plays important roles in carcinogenesis and predicts poor prognosis in non-small-cell lung cancerTGFβ-dependent induction of interleukin-11 and interleukin-8 involves SMAD and p38 MAPK pathways in breast tumor models with varied bone metastases potential.Transforming growth factor-β-induced epithelial-mesenchymal transition facilitates epidermal growth factor-dependent breast cancer progressionLysyl oxidase contributes to mechanotransduction-mediated regulation of transforming growth factor-β signaling in breast cancer cells.The antitumorigenic function of EGFR in metastatic breast cancer is regulated by expression of Mig6.TGFβ-mediated suppression of CD248 in non-cancer cells via canonical Smad-dependent signaling pathways is uncoupled in cancer cellsSignaling mechanisms of the epithelial-mesenchymal transition.Deptor enhances triple-negative breast cancer metastasis and chemoresistance through coupling to survivin expressionThe Cain and Abl of epithelial-mesenchymal transition and transforming growth factor-β in mammary epithelial cellsSilencing β3 Integrin by Targeted ECO/siRNA Nanoparticles Inhibits EMT and Metastasis of Triple-Negative Breast Cancer.PEAK1 Acts as a Molecular Switch to Regulate Context-Dependent TGFβ Responses in Breast Cancer.Forkhead Box F1 promotes breast cancer cell migration by upregulating lysyl oxidase and suppressing Smad2/3 signalingTGF-β upregulates miR-181a expression to promote breast cancer metastasis.Distinct phosphotyrosine-dependent functions of the ShcA adaptor protein are required for transforming growth factor β (TGFβ)-induced breast cancer cell migration, invasion, and metastasisLPA Stimulates the Phosphorylation of p130Cas via Gαi2 in Ovarian Cancer CellsUlinastatin exerts synergistic effects with taxotere and inhibits invasion and metastasis of breast cancer by blocking angiogenesis and the epithelial-mesenchymal transition.The pathophysiology of epithelial-mesenchymal transition induced by transforming growth factor-beta in normal and malignant mammary epithelial cells.TGF-β stimulates Pyk2 expression as part of an epithelial-mesenchymal transition program required for metastatic outgrowth of breast cancer.Noncanonical TGF-β signaling during mammary tumorigenesis.Deconstructing the mechanisms and consequences of TGF-β-induced EMT during cancer progression.The relevance of the TGF-β Paradox to EMT-MET programsTargeted inactivation of β1 integrin induces β3 integrin switching, which drives breast cancer metastasis by TGF-βA critical role for p130Cas in the progression of pulmonary hypertension in humans and rodents.Upregulated WAVE3 expression is essential for TGF-β-mediated EMT and metastasis of triple-negative breast cancer cells.Transforming growth factor-β signaling in tumor initiation, progression and therapy in breast cancer: an update.Neuropilins are multifunctional coreceptors involved in tumor initiation, growth, metastasis and immunity.Roles for crk in cancer metastasis and invasion.
P2860
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P2860
p130Cas is required for mammary tumor growth and transforming growth factor-beta-mediated metastasis through regulation of Smad2/3 activity.
description
2009 nî lūn-bûn
@nan
2009 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
p130Cas is required for mammar ...... egulation of Smad2/3 activity.
@ast
p130Cas is required for mammar ...... egulation of Smad2/3 activity.
@en
p130Cas is required for mammar ...... egulation of Smad2/3 activity.
@nl
type
label
p130Cas is required for mammar ...... egulation of Smad2/3 activity.
@ast
p130Cas is required for mammar ...... egulation of Smad2/3 activity.
@en
p130Cas is required for mammar ...... egulation of Smad2/3 activity.
@nl
prefLabel
p130Cas is required for mammar ...... egulation of Smad2/3 activity.
@ast
p130Cas is required for mammar ...... egulation of Smad2/3 activity.
@en
p130Cas is required for mammar ...... egulation of Smad2/3 activity.
@nl
P2860
P356
P1476
p130Cas is required for mammar ...... regulation of Smad2/3 activity
@en
P2093
Jason A Smith
William P Schiemann
P2860
P304
34145-34156
P356
10.1074/JBC.M109.023614
P407
P577
2009-10-12T00:00:00Z