about
Caspase-Mediated Anti-Apoptotic Effect of Ginsenoside Rg5, a Main Rare Ginsenoside, on Acetaminophen-Induced Hepatotoxicity in Mice.Nephroprotective Effects of Anthocyanin from the Fruits of Panax ginseng (GFA) on Cisplatin-Induced Acute Kidney Injury in Mice.Saponins (Ginsenosides) from the Leaves of Panax quinquefolius Ameliorated Acetaminophen-Induced Hepatotoxicity in Mice.Response to the Comments on Caspase-Mediated Anti-Apoptotic Effect of Ginsenoside Rg5, a Main Rare Ginsenoside, on Acetaminophen-Induced Hepatotoxicity in Mice.Dietary α-Mangostin Provides Protective Effects against Acetaminophen-Induced Hepatotoxicity in Mice via Akt/mTOR-Mediated Inhibition of Autophagy and Apoptosis.The Liver Protection Effects of Maltol, a Flavoring Agent, on Carbon Tetrachloride-Induced Acute Liver Injury in Mice via Inhibiting Apoptosis and Inflammatory ResponseSaponins Ameliorate Cisplatin-Induced Acute Nephrotoxicity through the NF-κB-Mediated Inflammation and PI3K/Akt/Apoptosis Signaling PathwaysPseudoginsengenin DQ Exhibits Therapeutic Effects in Cisplatin-Induced Acute Kidney Injury via Sirt1/NF-κB and Caspase Signaling Pathway without Compromising Its Antitumor Activity in MiceMaltol Improves APAP-Induced Hepatotoxicity by Inhibiting Oxidative Stress and Inflammation Response via NF-κB and PI3K/Akt Signal PathwaysMaltol Mitigates Thioacetamide-induced Liver Fibrosis through TGF-β1-mediated Activation of PI3K/Akt Signaling PathwayArginyl-fructosyl-glucose, a Major Maillard Reaction Product of Red Ginseng, Attenuates Cisplatin-Induced Acute Kidney Injury by Regulating Nuclear Factor κB and Phosphatidylinositol 3-Kinase/Protein Kinase B Signaling PathwaysGinsenoside Rb3 provides protective effects against cisplatin-induced nephrotoxicity via regulation of AMPK-/mTOR-mediated autophagy and inhibition of apoptosis in vitro and in vivoMaltol (3-Hydroxy-2-methyl-4-pyrone) Slows d-Galactose-Induced Brain Aging Process by Damping the Nrf2/HO-1-Mediated Oxidative Stress in MiceRed ginseng protects against cisplatin-induced intestinal toxicity by inhibiting apoptosis and autophagy via the PI3K/AKT and MAPK signaling pathwaysProtective effect of ginsenoside Rk1, a major rare saponin from black ginseng, on cisplatin-induced nephrotoxicity in HEK-293 cells
P50
Q47719913-ACE31F3B-D903-4AF4-A94B-BEF8E6F1478CQ47898167-5DB54ADB-3B85-4B0F-B9D6-1BE9EAA3E678Q48248734-916FDD9F-DD92-4C67-866A-2E24D4C8F26CQ50188125-54D593F9-F424-48A5-BCE1-58E6C2B77081Q55421884-64445495-A0E3-4453-83C9-B5B4392D8CC2Q58696577-3A52935D-5033-438C-93A5-12FDEDCDE803Q58716165-C7DB2A56-F825-4894-8B0D-9D94FEC45E07Q59813799-8BDBBD1D-8101-4781-9AE8-CA2FF48D92D4Q90230789-CF6B1918-56B7-43A0-BE05-DA5A2616C2C6Q91051276-4F6AF107-39C4-42E8-8561-0AEFAC65AD83Q91689703-2329EF1B-A24D-4124-93B0-67B57CFA2FBFQ92062088-BFA1B4E1-5D9C-4F82-AB02-8F4FD068F379Q92955298-F2BB7129-E9EB-4CF6-AEAB-74A5446DAA1EQ94476426-590898B8-F523-4BAA-A95C-BCBDFEDD03EEQ94571610-5FAEE2A7-3083-46E9-AF92-ECFB06065421
P50
description
researcher (ORCID 0000-0002-2988-4298)
@en
name
Wei Li
@en
type
label
Wei Li
@en
prefLabel
Wei Li
@en
P31
P496
0000-0002-2988-4298