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How exercise may amend metabolic disturbances in diabetic cardiomyopathy

How exercise may amend metabolic disturbances in diabetic cardiomyopathy

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Mitochondrial health, the epigenome and healthspan Mitochondrial respiration and ROS emission during β-oxidation in the heart: An experimental-computational study.Pathophysiological Fundamentals of Diabetic Cardiomyopathy.Exercise-induced alterations in pancreatic oxidative stress and mitochondrial function in type 2 diabetic Goto-Kakizaki rats.High-Intensity Exercise Reduces Cardiac Fibrosis and Hypertrophy but Does Not Restore the Nitroso-Redox Imbalance in Diabetic Cardiomyopathy Sprint interval training decreases left-ventricular glucose uptake compared to moderate-intensity continuous training in subjects with type 2 diabetes or prediabetes.Moderate exercise training does not prevent the reduction in myocardial L-type Ca2+ channels protein expression at obese rats Beneficial effects of exercise training in heart failure are lost in male diabetic rats.Exercise of obese mice induces cardioprotection and oxygen sparing in hearts exposed to high-fat load.Exercise Amaliorates Metabolic Disturbances and Oxidative Stress in Diabetic Cardiomyopathy: Possible Underlying Mechanisms.An "Exercise" in Cardiac Metabolism.

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How exercise may amend metabolic disturbances in diabetic cardiomyopathy

http://www.wikidata.org/entity/Q26863272

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2015 nî lūn-bûn

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2015 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2015 թվականի հունիսին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

How exercise may amend metabolic disturbances in diabetic cardiomyopathy

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How exercise may amend metabolic disturbances in diabetic cardiomyopathy

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How exercise may amend metabolic disturbances in diabetic cardiomyopathy

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type

label

How exercise may amend metabolic disturbances in diabetic cardiomyopathy

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How exercise may amend metabolic disturbances in diabetic cardiomyopathy

@en

How exercise may amend metabolic disturbances in diabetic cardiomyopathy

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How exercise may amend metabolic disturbances in diabetic cardiomyopathy

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How exercise may amend metabolic disturbances in diabetic cardiomyopathy

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How exercise may amend metabolic disturbances in diabetic cardiomyopathy

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Antioxidants & Redox Signaling

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How exercise may amend metabolic disturbances in diabetic cardiomyopathy

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Anne D. Hafstad

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Ellen Aasum

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Neoma Boardman

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Creative Commons Attribution-NonCommercial

P2860

Exercise training enhanced myocardial endothelial nitric oxide synthase (eNOS) function in diabetic Goto-Kakizaki (GK) rats

Nrf2 is critical in defense against high glucose-induced oxidative damage in cardiomyocytes

Antioxidant supplements for prevention of mortality in healthy participants and patients with various diseases

Exercise-induced BCL2-regulated autophagy is required for muscle glucose homeostasis

Antioxidants prevent health-promoting effects of physical exercise in humans

Role of nitrosative stress and peroxynitrite in the pathogenesis of diabetic complications. Emerging new therapeutical strategies

Endoplasmic reticulum stress and Nox-mediated reactive oxygen species signaling in the peripheral vasculature: potential role in hypertension

Nitric oxide synthases in heart failure

Interplay of oxidative, nitrosative/nitrative stress, inflammation, cell death and autophagy in diabetic cardiomyopathy

The cardiac phenotype induced by PPARalpha overexpression mimics that caused by diabetes mellitus

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10.1089/ARS.2015.6304

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