sameAs
Ghrelin expression in the mouse pancreas defines a unique multipotent progenitor populationIdentification of Protor as a novel Rictor-binding component of mTOR complex-2Insights into the biochemical and genetic basis of glucokinase activation from naturally occurring hypoglycemia mutationsDeletion of PPARgamma in adipose tissues of mice protects against high fat diet-induced obesity and insulin resistanceFat cell-specific ablation of rictor in mice impairs insulin-regulated fat cell and whole-body glucose and lipid metabolismCytosolic phosphoenolpyruvate carboxykinase does not solely control the rate of hepatic gluconeogenesis in the intact mouse livermTOR complex 2 is required for the development of prostate cancer induced by Pten loss in micePancreas-specific Cre driver lines and considerations for their prudent useDysregulation of the norepinephrine transporter sustains cortical hypodopaminergia and schizophrenia-like behaviors in neuronal rictor null miceRictor/mTORC2 is essential for maintaining a balance between beta-cell proliferation and cell sizeTissue-specific deletion of Foxa2 in pancreatic beta cells results in hyperinsulinemic hypoglycemiapdx-1 function is specifically required in embryonic beta cells to generate appropriate numbers of endocrine cell types and maintain glucose homeostasisMultiallelic disruption of the rictor gene in mice reveals that mTOR complex 2 is essential for fetal growth and viabilityAnks4b, a novel target of HNF4α protein, interacts with GRP78 protein and regulates endoplasmic reticulum stress-induced apoptosis in pancreatic β-cellsLoss of Myt1 function partially compromises endocrine islet cell differentiation and pancreatic physiological function in the mouseDual roles for glucokinase in glucose homeostasis as determined by liver and pancreatic beta cell-specific gene knock-outs using Cre recombinaseTargeted disruption of hepatic frataxin expression causes impaired mitochondrial function, decreased life span and tumor growth in miceTissue-specific regulation of two functional malic enzyme mRNAs by triiodothyronineGenetic rescue of Cdk4 null mice restores pancreatic beta-cell proliferation but not homeostatic cell numberPtf1a determines horizontal and amacrine cell fates during mouse retinal developmentMind bomb 1 is required for pancreatic β-cell formationImpaired insulin secretion and glucose tolerance in beta cell-selective Ca(v)1.2 Ca2+ channel null miceConditional inactivation of the MEN1 gene leads to pancreatic and pituitary tumorigenesis but does not affect normal development of these tissuesDopamine biosynthesis is selectively abolished in substantia nigra/ventral tegmental area but not in hypothalamic neurons in mice with targeted disruption of the Nurr1 geneEarly pancreatic development requires the vertebrate Suppressor of Hairless (RBPJ) in the PTF1 bHLH complexTargeted deletion of a cis-regulatory region reveals differential gene dosage requirements for Pdx1 in foregut organ differentiation and pancreas formation.Mice lacking alpha-calcitonin gene-related peptide exhibit normal cardiovascular regulation and neuromuscular developmentMuscle-specific deletion of rictor impairs insulin-stimulated glucose transport and enhances Basal glycogen synthase activity.A Rictor-Myo1c complex participates in dynamic cortical actin events in 3T3-L1 adipocytesIsolation and characterization of the mouse cytosolic phosphoenolpyruvate carboxykinase (GTP) gene: evidence for tissue-specific hypersensitive sites.Cell-specific roles of glucokinase in glucose homeostasis.Setd5 is essential for mammalian development and the co-transcriptional regulation of histone acetylation.Inactivation of the hepatic cytochrome P450 system by conditional deletion of hepatic cytochrome P450 reductase.Disruption of PPARgamma signaling results in mouse prostatic intraepithelial neoplasia involving active autophagy.Energy homeostasis and gastrointestinal endocrine differentiation do not require the anorectic hormone peptide YY.Alterations in the regulation of androgen-sensitive Cyp 4a monooxygenases cause hypertensionHepatocyte-specific mutation establishes retinoid X receptor alpha as a heterodimeric integrator of multiple physiological processes in the liver.Hepatocyte nuclear factor 3beta (Foxa2) is dispensable for maintaining the differentiated state of the adult hepatocytePhosphoenolpyruvate carboxykinase is necessary for the integration of hepatic energy metabolism.Replacement of Rbpj with Rbpjl in the PTF1 complex controls the final maturation of pancreatic acinar cells.
P50
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P50
description
American biologist
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հետազոտող
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Mark Magnuson
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Mark Magnuson
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Mark Magnuson
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Mark Magnuson
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Mark Magnuson
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Mark Magnuson
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Mark Magnuson
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Mark Magnuson
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Mark Magnuson
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Mark Magnuson
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Mark Magnuson
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Mark Magnuson
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Mark A Magnuson
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Mark A Magnuson
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Mark A. Magnuson
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Mark A. Magnuson
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Mark Magnuson
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Mark Magnuson
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Mark Magnuson
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Mark Magnuson
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Mark Magnuson
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Mark Magnuson
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P106
P1153
7005306800
P21
P31
P496
0000-0002-8824-6499