Identification of differentially expressed genes in cardiac hypertrophy by analysis of expressed sequence tags.
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Heart-specific genes revealed by expressed sequence tag (EST) samplingScaffold Proteins Regulating Extracellular Regulated Kinase Function in Cardiac Hypertrophy and DiseaseSkeletal muscle LIM protein 1 (SLIM1/FHL1) induces alpha 5 beta 1-integrin-dependent myocyte elongationGenomic annotation of 15,809 ESTs identified from pooled early gestation human eyesFour and a half LIM protein 1 binds myosin-binding protein C and regulates myosin filament formation and sarcomere assemblyAnalysis of expressed sequence tags from two starvation, time-of-day-specific libraries of Neurospora crassa reveals novel clock-controlled genes.Identification, characterization, and mapping of expressed sequence tags from an embryonic zebrafish heart cDNA library.NCAM(CD56) and RUNX1(AML1) are up-regulated in human ischemic cardiomyopathy and a rat model of chronic cardiac ischemia.Microarray gene expression profiles in dilated and hypertrophic cardiomyopathic end-stage heart failure.Distinct molecular portraits of human failing hearts identified by dedicated cDNA microarrays.Differential gene expression in abdomens of the malaria vector mosquito, Anopheles gambiae, after sugar feeding, blood feeding and Plasmodium berghei infectionComparative analysis of differentially expressed genes in normal and white spot syndrome virus infected Penaeus monodon.Pressure load: the main factor for altered gene expression in right ventricular hypertrophy in chronic hypoxic rats.Analysis of altered genomic expression profiles in the senescent and diseased myocardium using cDNA microarrays.Mechanotransduction in cardiac hypertrophy and failure.Global gene expression profiling of end-stage dilated cardiomyopathy using a human cardiovascular-based cDNA microarray.Extensive induction of important mediators of fibrosis and dystrophic calcification in desmin-deficient cardiomyopathy.Defining molecular and cellular responses after low and high linear energy transfer radiations to develop biomarkers of carcinogenic risk or therapeutic outcome.Reverse right ventricular structural and extracellular matrix remodeling by estrogen in severe pulmonary hypertensionA novel mechanism involving four-and-a-half LIM domain protein-1 and extracellular signal-regulated kinase-2 regulates titin phosphorylation and mechanics.Cardiac structural and hemodynamic changes associated with physiological heart hypertrophy of pregnancy are reversed postpartum.Gene expression profiling on global cDNA arrays gives hints concerning potential signal transduction pathways involved in cardiac fibrosis of renal failureIdentification of a core set of genes that signifies pathways underlying cardiac hypertrophy.An FHL1-containing complex within the cardiomyocyte sarcomere mediates hypertrophic biomechanical stress responses in miceMatrix Gla protein species and risk of cardiovascular events in type 2 diabetic patients.Genes, proteins and complexes: the multifaceted nature of FHL family proteins in diverse tissues.Key role of ERK1/2 molecular scaffolds in heart pathology.Divergent transcriptional responses to independent genetic causes of cardiac hypertrophy.Whole transcriptome analysis with sequencing: methods, challenges and potential solutions.Identification of an FHL1 protein complex containing ACTN1, ACTN4, and PDLIM1 using affinity purifications and MS-based protein-protein interaction analysisSex dependence and temporal dependence of the left ventricular genomic response to pressure overload.Upregulation of cardiac matrix Gla protein expression in response to hypertrophic stimuli.Kinetic analysis of cardiac transcriptome regulation during chronic high-fat diet in dogs.Novel Muscle-Specific Genes TCAP, TNNI1, and FHL1 in Cattle: SNVs, Linkage Disequilibrium, Combined Genotypes, Association Analysis of Growth Performance, and Carcass Quality Traits and Expression Studies.AT1 blockade abolishes left ventricular hypertrophy in heterozygous cMyBP-C null mice: role of FHL1.Role of the estrogen/estrogen-receptor-beta axis in the genomic response to pressure overload-induced hypertrophy.Transcriptomal analysis of failing and nonfailing human hearts.Dysregulation of FHL1 spliceforms due to an indel mutation produces an Emery-Dreifuss muscular dystrophy plus phenotype.Elucidating the molecular mechanism of cardiac remodeling using a comparative genomic approach.
P2860
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P2860
Identification of differentially expressed genes in cardiac hypertrophy by analysis of expressed sequence tags.
description
2000 nî lūn-bûn
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2000 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Identification of differential ...... is of expressed sequence tags.
@ast
Identification of differential ...... is of expressed sequence tags.
@en
type
label
Identification of differential ...... is of expressed sequence tags.
@ast
Identification of differential ...... is of expressed sequence tags.
@en
prefLabel
Identification of differential ...... is of expressed sequence tags.
@ast
Identification of differential ...... is of expressed sequence tags.
@en
P2093
P356
P1433
P1476
Identification of differential ...... is of expressed sequence tags.
@en
P2093
P356
10.1006/GENO.2000.6171
P577
2000-05-01T00:00:00Z