Inhibiting mitochondrial permeability transition pore opening at reperfusion protects against ischaemia-reperfusion injury.
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Inhibition of the Mitochondrial Permeability Transition for Cytoprotection: Direct versus Indirect MechanismsThe Role of Mitochondrial Functional Proteins in ROS Production in Ischemic Heart DiseasesMitochondrial dynamics in cardiovascular health and diseaseRole of the MPTP in conditioning the heart - translatability and mechanismMyocardial reperfusion injury: looking beyond primary PCILow-pressure reperfusion alters mitochondrial permeability transitionRole of epoxyeicosatrienoic acids in protecting the myocardium following ischemia/reperfusion injuryAttenuation of doxorubicin-induced cardiotoxicity by mdivi-1: a mitochondrial division/mitophagy inhibitorRegulation of the transcription factor EB-PGC1α axis by beclin-1 controls mitochondrial quality and cardiomyocyte death under stressA novel estrogen receptor GPER inhibits mitochondria permeability transition pore opening and protects the heart against ischemia-reperfusion injury.Protein Kinase Signaling at the Crossroads of Myocyte Life and Death in Ischemic Heart Disease.Is there a role for ischaemic conditioning in cardiac surgery?High Sensitivity of SIRT3 Deficient Hearts to Ischemia-Reperfusion Is Associated with Mitochondrial Abnormalities.Pyrroloquinoline quinone preserves mitochondrial function and prevents oxidative injury in adult rat cardiac myocytes.Mechanism for resveratrol-induced cardioprotection against reperfusion injury involves glycogen synthase kinase 3beta and mitochondrial permeability transition pore.The cardioprotective effect of necrostatin requires the cyclophilin-D component of the mitochondrial permeability transition poreMitochondrial free [Ca2+] increases during ATP/ADP antiport and ADP phosphorylation: exploration of mechanismsMitochondrial depolarization underlies delay in permeability transition by preconditioning with isoflurane: roles of ROS and Ca2+.Mitochondrial cyclophilin-D as a critical mediator of ischaemic preconditioning.Mitochondrial morphology and cardiovascular diseaseThe small chill: mild hypothermia for cardioprotection?Low-intensity aerobic interval training attenuates pathological left ventricular remodeling and mitochondrial dysfunction in aortic-banded miniature swineIschemic preconditioning inhibits mitochondrial permeability transition pore opening through the PTEN/PDE4 signaling pathwayThe role of mitochondria in protection of the heart by preconditioning.Minocycline protects cardiac myocytes against simulated ischemia–reperfusion injury by inhibiting poly(ADP-ribose) polymerase-1.Tumor necrosis factor receptor-associated factor 2 mediates mitochondrial autophagy.Ischemia/reperfusion injury and cardioprotective mechanisms: Role of mitochondria and reactive oxygen speciesLeptin, the obesity-associated hormone, exhibits direct cardioprotective effectsPyrrolidinyl caffeamide against ischemia/reperfusion injury in cardiomyocytes through AMPK/AKT pathwaysChronic testosterone replacement exerts cardioprotection against cardiac ischemia-reperfusion injury by attenuating mitochondrial dysfunction in testosterone-deprived rats.Mechanisms underlying acute protection from cardiac ischemia-reperfusion injuryG Protein-Coupled Estrogen Receptor 1 Mediates Acute Estrogen-Induced Cardioprotection via MEK/ERK/GSK-3β Pathway after Ischemia/Reperfusion.Short-and long-term effects of ischemic postconditioning in STEMI patients: a meta-analysis.Mitochondrial cyclophilin-D as a potential therapeutic target for post-myocardial infarction heart failureMorphine-Induced Preconditioning: Involvement of Protein Kinase A and Mitochondrial Permeability Transition PoreTargeted Modification of Mitochondrial ROS Production Converts High Glucose-Induced Cytotoxicity to Cytoprotection: Effects on Anesthetic Preconditioning.Cyclosporin A and cardioprotection: from investigative tool to therapeutic agent.Age-dependent reductions in mitochondrial respiration are exacerbated by calcium in the female rat heartPreconditioning and postconditioning: the essential role of the mitochondrial permeability transition pore.Astragaloside IV inhibits oxidative stress-induced mitochondrial permeability transition pore opening by inactivating GSK-3β via nitric oxide in H9c2 cardiac cells.
P2860
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P2860
Inhibiting mitochondrial permeability transition pore opening at reperfusion protects against ischaemia-reperfusion injury.
description
2003 nî lūn-bûn
@nan
2003 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Inhibiting mitochondrial perme ...... ischaemia-reperfusion injury.
@ast
Inhibiting mitochondrial perme ...... ischaemia-reperfusion injury.
@en
type
label
Inhibiting mitochondrial perme ...... ischaemia-reperfusion injury.
@ast
Inhibiting mitochondrial perme ...... ischaemia-reperfusion injury.
@en
prefLabel
Inhibiting mitochondrial perme ...... ischaemia-reperfusion injury.
@ast
Inhibiting mitochondrial perme ...... ischaemia-reperfusion injury.
@en
P50
P1476
Inhibiting mitochondrial perme ...... ischaemia-reperfusion injury.
@en
P304
P356
10.1016/J.CARDIORES.2003.09.025
P577
2003-12-01T00:00:00Z