Gene expression changes in foam cells and the role of chemokine receptor CCR7 during atherosclerosis regression in ApoE-deficient mice. - sprookjes - yvandenbroek - TriplyDB

Gene expression changes in foam cells and the role of chemokine receptor CCR7 during atherosclerosis regression in ApoE-deficient mice.

Gene expression changes in foam cells and the role of chemokine receptor CCR7 during atherosclerosis regression in ApoE-deficient mice.

http://www.wikidata.org/entity/Q33236495

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Induction of dendritic cell-like phenotype in macrophages during foam cell formation Scavenger receptor SR-BI in macrophage lipid metabolism High density lipoprotein and metabolic disease: Potential benefits of restoring its functional properties MIF and CXCL12 in Cardiovascular Diseases: Functional Differences and Similarities Chemokines and their receptors in Atherosclerosis Macrophages in vascular inflammation--From atherosclerosis to vasculitis Hypoxia in murine atherosclerotic plaques and its adverse effects on macrophages Regulation of atherogenesis by chemokines and chemokine receptors Regression of atherosclerosis: insights from animal and clinical studies Epigenome-guided analysis of the transcriptome of plaque macrophages during atherosclerosis regression reveals activation of the Wnt signaling pathway Vitamin D deficiency induces high blood pressure and accelerates atherosclerosis in mice Divergent JAM-C Expression Accelerates Monocyte-Derived Cell Exit from Atherosclerotic Plaques Novel concepts in HDL pharmacology.Endoplasmic reticulum stress controls M2 macrophage differentiation and foam cell formation Inflammatory cell recruitment in cardiovascular disease: murine models and potential clinical applications Connexin40 imparts conduction heterogeneity to atrial tissue Atherosclerosis in LDLR-knockout mice is inhibited, but not reversed, by the PPARgamma ligand pioglitazone Regression of atherosclerosis is characterized by broad changes in the plaque macrophage transcriptome Statins promote the regression of atherosclerosis via activation of the CCR7-dependent emigration pathway in macrophages Deletion of ABCA1 and ABCG1 impairs macrophage migration because of increased Rac1 signaling.ACAT inhibition reduces the progression of preexisting, advanced atherosclerotic mouse lesions without plaque or systemic toxicity.HDL induces the expression of the M2 macrophage markers arginase 1 and Fizz-1 in a STAT6-dependent process.An inducible and reversible mouse genetic rescue system.Leukocyte Trafficking in Cardiovascular Disease: Insights from Experimental Models Rapid regression of atherosclerosis with MTP inhibitor treatment.Mechanisms that regulate macrophage burden in atherosclerosis.Global chemokine expression in systemic sclerosis (SSc): CCL19 expression correlates with vascular inflammation in SSc skin.Inflammation and its resolution as determinants of acute coronary syndromes.HDL promotes rapid atherosclerosis regression in mice and alters inflammatory properties of plaque monocyte-derived cells.Genetic network identifies novel pathways contributing to atherosclerosis susceptibility in the innominate artery Association of a rheumatoid arthritis susceptibility variant at the CCL21 locus with premature mortality in inflammatory polyarthritis patients HDL-replacement therapy: mechanism of action, types of agents and potential clinical indications.Genes involved in systemic and arterial bed dependent atherosclerosis--Tampere Vascular study miR33 inhibition overcomes deleterious effects of diabetes mellitus on atherosclerosis plaque regression in mice.A systems biology approach to understanding atherosclerosis.LXR promotes the maximal egress of monocyte-derived cells from mouse aortic plaques during atherosclerosis regression.Tumor necrosis factor receptor-associated factor 1 (TRAF1) deficiency attenuates atherosclerosis in mice by impairing monocyte recruitment to the vessel wall Regulation and consequences of monocytosis Increased microvascularization and vessel permeability associate with active inflammation in human atheromata.Characterization of CCL19 and CCL21 in rheumatoid arthritis.

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Gene expression changes in foam cells and the role of chemokine receptor CCR7 during atherosclerosis regression in ApoE-deficient mice.

http://www.wikidata.org/entity/Q33236495

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2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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Gene expression changes in foa ...... ession in ApoE-deficient mice.

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Gene expression changes in foa ...... ession in ApoE-deficient mice.

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Proceedings of the National Academy of Sciences of the United States of America

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Gene expression changes in foa ...... ession in ApoE-deficient mice.

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Eugene Trogan

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Frank Tacke

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George H Rothblat

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Gwendalyn J Randolph

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Jonathan E Feig

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Snjezana Dogan

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Véronique Angeli

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P2860

Scavenger receptor class B type I-mediated cholesteryl ester-selective uptake and efflux of unesterified cholesterol. Influence of high density lipoprotein size and structure

PPARgamma promotes monocyte/macrophage differentiation and uptake of oxidized LDL

Dendritic cells and the control of immunity

The leukotriene C(4) transporter MRP1 regulates CCL19 (MIP-3beta, ELC)-dependent mobilization of dendritic cells to lymph nodes

A PPAR gamma-LXR-ABCA1 pathway in macrophages is involved in cholesterol efflux and atherogenesis

Reciprocal regulation of inflammation and lipid metabolism by liver X receptors

ATP-binding cassette transporters G1 and G4 mediate cellular cholesterol efflux to high-density lipoproteins

CCR7 coordinates the primary immune response by establishing functional microenvironments in secondary lymphoid organs

The peroxisome proliferator-activated receptor-gamma is a negative regulator of macrophage activation

Laser capture microdissection analysis of gene expression in macrophages from atherosclerotic lesions of apolipoprotein E-deficient mice.

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10.1073/PNAS.0511043103

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103

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Edward A Fisher

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7242007

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16537455

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atherosclerosis

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1450154

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