Oxidative inactivation of mitochondrial aconitase results in iron and H2O2-mediated neurotoxicity in rat primary mesencephalic cultures.
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Protective effects and potential mechanisms of Pien Tze Huang on cerebral chronic ischemia and hypertensive strokeParkinson's Disease: The Mitochondria-Iron LinkMitochondrial involvement and oxidative stress in temporal lobe epilepsyA strategically designed small molecule attacks alpha-ketoglutarate dehydrogenase in tumor cells through a redox processPlasma metabolomics reveals a diagnostic metabolic fingerprint for mitochondrial aconitase (ACO2) deficiencyGlucose 6-phosphate dehydrogenase deficiency increases redox stress and moderately accelerates the development of heart failureOxidative toxicity in neurodegenerative diseases: role of mitochondrial dysfunction and therapeutic strategies.Distinct effects of rotenone, 1-methyl-4-phenylpyridinium and 6-hydroxydopamine on cellular bioenergetics and cell death.NRMT1 knockout mice exhibit phenotypes associated with impaired DNA repair and premature aging.Thioredoxin reductase deficiency potentiates oxidative stress, mitochondrial dysfunction and cell death in dopaminergic cellsAconitase causes iron toxicity in Drosophila pink1 mutantsMitochondrial acetylation and diseases of aging.Altered zinc transport disrupts mitochondrial protein processing/import in fragile X-associated tremor/ataxia syndrome.The role of astrocyte mitochondria in differential regional susceptibility to environmental neurotoxicants: tools for understanding neurodegenerationMitochondrial aconitase knockdown attenuates paraquat-induced dopaminergic cell death via decreased cellular metabolism and release of iron and H₂O₂.Generation of reactive oxygen species in 1-methyl-4-phenylpyridinium (MPP+) treated dopaminergic neurons occurs as an NADPH oxidase-dependent two-wave cascade.Manganese superoxide dismutase: guardian of the powerhouse.A Cell-Permeable Fluorescent Prochelator Responds to Hydrogen Peroxide and Metal Ions by Decreasing Fluorescence.Engineered Trx2p industrial yeast strain protects glycolysis and fermentation proteins from oxidative carbonylation during biomass propagation.Impaired iron status in aging researchGLP-1 Cleavage Product Reverses Persistent ROS Generation After Transient Hyperglycemia by Disrupting an ROS-Generating Feedback LoopEffects of glucose-6-phosphate dehydrogenase deficiency on the metabolic and cardiac responses to obesogenic or high-fructose diets.Alterations in mitochondrial dynamics induced by tebufenpyrad and pyridaben in a dopaminergic neuronal cell culture model.Reduction in mitochondrial iron alleviates cardiac damage during injuryAutophagy and ubiquitin-proteasome system contribute to sperm mitophagy after mammalian fertilizationDesigner aminoglycosides that selectively inhibit cytoplasmic rather than mitochondrial ribosomes show decreased ototoxicity: a strategy for the treatment of genetic diseases.Mitochondria-targeted antioxidants for treatment of Parkinson's disease: preclinical and clinical outcomes.The emerging role of iron dyshomeostasis in the mitochondrial decay of aging.Iron chelation and neuroprotection in neurodegenerative diseases.Curbing cancer's sweet tooth: is there a role for MnSOD in regulation of the Warburg effect?Mitochondria in ageing: there is metabolism beyond the ROS.The hydroxyl radical in plants: from seed to seed.Protective effects of 1α,25-Dihydroxyvitamin D3 on cultured neural cells exposed to catalytic iron.Kaurane diterpenes as mitochondrial alterations preventive agents under experimental oxidative stress conditions.Metabolic enzymes dysregulation in heart failure: the prospective therapy.Inactivation of Aconitase by Tetrahydrobiopterin in DArgic Cells: Relevance to PD.Metabolic Dysfunction in Parkinson's Disease: Bioenergetics, Redox Homeostasis and Central Carbon Metabolism.Hypermetabolism and hypercatabolism of skeletal muscle accompany mitochondrial stress following severe burn trauma.Cardiac-specific overexpression of catalase attenuates paraquat-induced myocardial geometric and contractile alteration: role of ER stress.TCA cycle rewiring fosters metabolic adaptation to oxygen restriction in skeletal muscle from rodents and humans.
P2860
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P2860
Oxidative inactivation of mitochondrial aconitase results in iron and H2O2-mediated neurotoxicity in rat primary mesencephalic cultures.
description
2009 nî lūn-bûn
@nan
2009 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Oxidative inactivation of mito ...... rimary mesencephalic cultures.
@ast
Oxidative inactivation of mito ...... rimary mesencephalic cultures.
@en
type
label
Oxidative inactivation of mito ...... rimary mesencephalic cultures.
@ast
Oxidative inactivation of mito ...... rimary mesencephalic cultures.
@en
prefLabel
Oxidative inactivation of mito ...... rimary mesencephalic cultures.
@ast
Oxidative inactivation of mito ...... rimary mesencephalic cultures.
@en
P2093
P2860
P1433
P1476
Oxidative inactivation of mito ...... rimary mesencephalic cultures.
@en
P2093
David Cantu
Jerome Schaack
Manisha Patel
P2860
P356
10.1371/JOURNAL.PONE.0007095
P407
P577
2009-09-18T00:00:00Z