Role of Nogo-A in neuronal survival in the reperfused ischemic brain - sprookjes - yvandenbroek - TriplyDB

Role of Nogo-A in neuronal survival in the reperfused ischemic brain

Role of Nogo-A in neuronal survival in the reperfused ischemic brain

http://www.wikidata.org/entity/Q33525257

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Myelin-associated glycoprotein modulates apoptosis of motoneurons during early postnatal development via NgR/p75(NTR) receptor-mediated activation of RhoA signaling pathways Neuronal Nogo-A upregulation does not contribute to ER stress-associated apoptosis but participates in the regenerative response in the axotomized adult retina.Effects of neural progenitor cells on post-stroke neurological impairment-a detailed and comprehensive analysis of behavioral tests.Experimental models of brain ischemia: a review of techniques, magnetic resonance imaging, and investigational cell-based therapies.Particular phosphorylation of PI3K/Akt on Thr308 via PDK-1 and PTEN mediates melatonin's neuroprotective activity after focal cerebral ischemia in mice.Targeted siRNA delivery reduces nitric oxide mediated cell death after spinal cord injury Upregulation of the GEF-H1 pathway after transient cerebral ischemia.HMG-CoA Reductase Inhibition Promotes Neurological Recovery, Peri-Lesional Tissue Remodeling, and Contralesional Pyramidal Tract Plasticity after Focal Cerebral Ischemia.Nogo-A knockdown inhibits hypoxia/reoxygenation-induced activation of mitochondrial-dependent apoptosis in cardiomyocytes.Exercise promotes axon regeneration of newborn striatonigral and corticonigral projection neurons in rats after ischemic stroke.Pharmacological inhibition of Rho-kinase (ROCK) signaling enhances cisplatin resistance in neuroblastoma cells.Amino-Nogo-A antagonizes reactive oxygen species generation and protects immature primary cortical neurons from oxidative toxicity.Intracerebroventricularly delivered VEGF promotes contralesional corticorubral plasticity after focal cerebral ischemia via mechanisms involving anti-inflammatory actions.Mechanisms of acute neurovascular protection with AT1 blockade after stroke: Effect of prestroke hypertension.Artery reopening is required for the neurorestorative effects of angiotensin modulation after experimental stroke Delayed administration of a PTEN inhibitor BPV improves functional recovery after experimental stroke Therapeutic antibodies in stroke.Neurodegeneration and Glial Response after Acute Striatal Stroke: Histological Basis for Neuroprotective Studies.Promoting brain remodelling and plasticity for stroke recovery: therapeutic promise and potential pitfalls of clinical translation.The role of Nogo-A in axonal plasticity, regrowth and repair.Therapeutics targeting Nogo-A hold promise for stroke restoration.Correlates of Post-Stroke Brain Plasticity, Relationship to Pathophysiological Settings and Implications for Human Proof-of-Concept Studies Astrocytic JWA expression is essential to dopaminergic neuron survival in the pathogenesis of Parkinson's disease.The Involvement of the Myelin-Associated Inhibitors and Their Receptors in CNS Plasticity and Injury.LOTUS overexpression accelerates neuronal plasticity after focal brain ischemia in mice.Asynchronous therapy targeting Nogo-A enhances neurobehavioral recovery by reducing neuronal loss and promoting neurite outgrowth after cerebral ischemia in mice.M9, a novel region of amino-Nogo-A, attenuates cerebral ischemic injury by inhibiting NADPH oxidase-derived superoxide production in mice.Perilesional treatment with chondroitinase ABC and motor training promote functional recovery after stroke in rats.A novel domain of amino-Nogo-A protects HT22 cells exposed to oxygen glucose deprivation by inhibiting NADPH oxidase activity.Examination of age-dependent effects of fetal ethanol exposure on behavior, hippocampal cell counts, and doublecortin immunoreactivity in rats.Evidence that membrane-bound G protein-coupled melatonin receptors MT1 and MT2 are not involved in the neuroprotective effects of melatonin in focal cerebral ischemia.Time-of-Day Dependent Neuronal Injury After Ischemic Stroke: Implication of Circadian Clock Transcriptional Factor Bmal1 and Survival Kinase AKT.Kir6.1 knockdown aggravates cerebral ischemia/reperfusion-induced neural injury in mice.

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Role of Nogo-A in neuronal survival in the reperfused ischemic brain

http://www.wikidata.org/entity/Q33525257

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2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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name

Role of Nogo-A in neuronal survival in the reperfused ischemic brain

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Role of Nogo-A in neuronal survival in the reperfused ischemic brain

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Role of Nogo-A in neuronal survival in the reperfused ischemic brain.

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Role of Nogo-A in neuronal survival in the reperfused ischemic brain

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Role of Nogo-A in neuronal survival in the reperfused ischemic brain

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Role of Nogo-A in neuronal survival in the reperfused ischemic brain.

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Role of Nogo-A in neuronal survival in the reperfused ischemic brain

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Role of Nogo-A in neuronal survival in the reperfused ischemic brain

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Role of Nogo-A in neuronal survival in the reperfused ischemic brain.

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Journal of Cerebral Blood Flow & Metabolism

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Role of Nogo-A in neuronal survival in the reperfused ischemic brain

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Ayman ElAli

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Claudio L Bassetti

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Dirk M Hermann

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Ertugrul Kilic

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Martin E Schwab

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Ulkan Kilic

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Zeyun Guo

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P2860

Identification of the Nogo inhibitor of axon regeneration as a Reticulon protein

A new role for Nogo as a regulator of vascular remodeling

Glial inhibition of CNS axon regeneration

Nogo-A is a myelin-associated neurite outgrowth inhibitor and an antigen for monoclonal antibody IN-1

Recovery from spinal cord injury mediated by antibodies to neurite growth inhibitors

Neurobehavioral development, adult openfield exploration and swimming navigation learning in mice with a modified beta-amyloid precursor protein gene

Nogo-A and myelin-associated glycoprotein mediate neurite growth inhibition by antagonistic regulation of RhoA and Rac1

Dual neuroprotective signaling mediated by downregulating two distinct phosphatase activities of PTEN.

1,026 experimental treatments in acute stroke

Relationship between metabolic dysfunctions, gene responses and delayed cell death after mild focal cerebral ischemia in mice.

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10.1038/JCBFM.2009.268

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5

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30

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41089452

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20087369

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