Upregulation of Na+ and Ca2+ transporters in arterial smooth muscle from ouabain-induced hypertensive rats.
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Specialized Functional Diversity and Interactions of the Na,K-ATPaseSalt sensitivity, endogenous ouabain and hypertensionOuabain-digoxin antagonism in rat arteries and neuronesRegulation of hypothalamic renin-angiotensin system and oxidative stress by aldosteroneKnockout of Na+/Ca2+ exchanger in smooth muscle attenuates vasoconstriction and L-type Ca2+ channel current and lowers blood pressureSignaling mechanisms that link salt retention to hypertension: endogenous ouabain, the Na(+) pump, the Na(+)/Ca(2+) exchanger and TRPC proteins.Central neuronal activation and pressor responses induced by circulating ANG II: role of the brain aldosterone-"ouabain" pathway.Upregulation of Na+/Ca2+ exchanger and TRPC6 contributes to abnormal Ca2+ homeostasis in arterial smooth muscle cells from Milan hypertensive rats.Normal pregnancy: mechanisms underlying the paradox of a ouabain-resistant state with elevated endogenous ouabain, suppressed arterial sodium calcium exchange, and low blood pressureNeuroendocrine humoral and vascular components in the pressor pathway for brain angiotensin II: a new axis in long term blood pressure control.Wistar rats resistant to the hypertensive effects of ouabain exhibit enhanced cardiac vagal activity and elevated plasma levels of calcitonin gene-related peptide.Why isn't endogenous ouabain more widely accepted?Endogenous ouabain: a link between sodium intake and hypertensionTRPC channels in smooth muscle cellsAttenuated renal vascular responses to acute angiotensin II infusion in smooth muscle-specific Na+/Ca2+ exchanger knockout mice.Endogenous cardiotonic steroids in kidney failure: a review and an hypothesis.Hypertension from chronic central sodium chloride in mice is mediated by the ouabain-binding site on the Na,K-ATPase α₂-isoform.How NaCl raises blood pressure: a new paradigm for the pathogenesis of salt-dependent hypertension.A role of the sodium pump in spreading ischemia in rats.Increased arterial smooth muscle Ca2+ signaling, vasoconstriction, and myogenic reactivity in Milan hypertensive ratsTransient receptor potential channels in the vasculature.Arterial α2-Na+ pump expression influences blood pressure: lessons from novel, genetically engineered smooth muscle-specific α2 mice.Nanomolar ouabain increases NCX1 expression and enhances Ca2+ signaling in human arterial myocytes: a mechanism that links salt to increased vascular resistance?Endogenous ouabain and the renin-angiotensin-aldosterone system: distinct effects on Na handling and blood pressure in human hypertension.Vasorelaxation of goat mesenteric artery is mediated by endothelial Na(+)-K(+)-ATPase.Activation of c-SRC underlies the differential effects of ouabain and digoxin on Ca(2+) signaling in arterial smooth muscle cellsAn elevation in physical coupling of type 1 inositol 1,4,5-trisphosphate (IP3) receptors to transient receptor potential 3 (TRPC3) channels constricts mesenteric arteries in genetic hypertensionPreoperative endogenous ouabain predicts acute kidney injury in cardiac surgery patientsLivin' with NCX and lovin' it: a 45 year romance.Increased constrictor tone induced by ouabain treatment in rats.Polymorphisms, hypertension and thiazide diuretics.TRPC Channels and Stroke.Update on angiotensin II: new endocrine connections between the brain, adrenal glands and the cardiovascular system.Endogenous Ouabain: Recent Advances and ControversiesCross talk between plasma membrane Na(+)/Ca (2+) exchanger-1 and TRPC/Orai-containing channels: key players in arterial hypertension.DIDS and the Janus-faced Na⁺-K⁺-activated ATPase. Focus on "DIDS inhibits Na-K-ATPase activity in porcine nonpigmented ciliary epithelial cells by a Src family kinase-dependent mechanism".A low concentration of ouabain (0.18 microg/kg) enhances hypertension in spontaneously hypertensive rats by inhibiting the Na+ pump and activating the renin-angiotensin system.Overactive bladder mediated by accelerated Ca2+ influx mode of Na+/Ca2+ exchanger in smooth muscle.Central and Peripheral Slow-Pressor Mechanisms Contributing to.How does pressure overload cause cardiac hypertrophy and dysfunction? High-ouabain affinity cardiac Na+ pumps are crucial.
P2860
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P2860
Upregulation of Na+ and Ca2+ transporters in arterial smooth muscle from ouabain-induced hypertensive rats.
description
2009 nî lūn-bûn
@nan
2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Upregulation of Na+ and Ca2+ t ...... ain-induced hypertensive rats.
@ast
Upregulation of Na+ and Ca2+ t ...... ain-induced hypertensive rats.
@en
Upregulation of Na+ and Ca2+ t ...... ain-induced hypertensive rats.
@nl
type
label
Upregulation of Na+ and Ca2+ t ...... ain-induced hypertensive rats.
@ast
Upregulation of Na+ and Ca2+ t ...... ain-induced hypertensive rats.
@en
Upregulation of Na+ and Ca2+ t ...... ain-induced hypertensive rats.
@nl
prefLabel
Upregulation of Na+ and Ca2+ t ...... ain-induced hypertensive rats.
@ast
Upregulation of Na+ and Ca2+ t ...... ain-induced hypertensive rats.
@en
Upregulation of Na+ and Ca2+ t ...... ain-induced hypertensive rats.
@nl
P2093
P2860
P1476
Upregulation of Na+ and Ca2+ t ...... ain-induced hypertensive rats.
@en
P2093
Alessandra Zulian
Amparo Mazzocco-Spezzia
Italia Papparella
John M Hamlyn
Maria V Pulina
Mordecai P Blaustein
Olga Beskina
Sergey G Baryshnikov
Vera A Golovina
P2860
P304
P356
10.1152/AJPHEART.00784.2009
P577
2009-11-06T00:00:00Z