Normal pregnancy: mechanisms underlying the paradox of a ouabain-resistant state with elevated endogenous ouabain, suppressed arterial sodium calcium exchange, and low blood pressure
about
Na+-K+-ATPase, a new class of plasma membrane receptorsOuabain-digoxin antagonism in rat arteries and neuronesNeuroendocrine humoral and vascular components in the pressor pathway for brain angiotensin II: a new axis in long term blood pressure control.Wistar rats resistant to the hypertensive effects of ouabain exhibit enhanced cardiac vagal activity and elevated plasma levels of calcitonin gene-related peptide.Ouabain inhibits placental sFlt1 production by repressing HSP27-dependent HIF-1α pathway.Reduction in maternal circulating ouabain impairs offspring growth and kidney developmentArterial α2-Na+ pump expression influences blood pressure: lessons from novel, genetically engineered smooth muscle-specific α2 mice.Cinobufotalin impedes Sw.71 cytotrophoblast cell line function via cell cycle arrest and apoptotic signaling.Personalized Therapy of Hypertension: the Past and the Future.Preliminary study of the effects of furosemide on blood pressure during late-onset pre-eclampsia in patients with high cardiac output.Salt, aldosterone and extrarenal Na+ - sensitive responses in pregnancy.Na,K-ATPase alpha isoforms at the blood-cerebrospinal fluid-trigeminal nerve and blood-retina interfaces in the rat.Endogenous Ouabain: Recent Advances and ControversiesNanomolar ouabain augments Ca2+ signalling in rat hippocampal neurones and glia.Regulation of the sodium pump during cardiomyocyte adaptation to pregnancy.The pump, the exchanger, and the holy spirit: origins and 40-year evolution of ideas about the ouabain-Na+ pump endocrine system.Pivotal role of α2 Na+ pumps and their high affinity ouabain binding site in cardiovascular health and disease.Prevention of apoptosis averts glomerular tubular disconnection and podocyte loss in proteinuric kidney disease.
P2860
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P2860
Normal pregnancy: mechanisms underlying the paradox of a ouabain-resistant state with elevated endogenous ouabain, suppressed arterial sodium calcium exchange, and low blood pressure
description
2012 nî lūn-bûn
@nan
2012 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2012年の論文
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2012年学术文章
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2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
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name
Normal pregnancy: mechanisms u ...... change, and low blood pressure
@ast
Normal pregnancy: mechanisms u ...... change, and low blood pressure
@en
Normal pregnancy: mechanisms u ...... change, and low blood pressure
@nl
type
label
Normal pregnancy: mechanisms u ...... change, and low blood pressure
@ast
Normal pregnancy: mechanisms u ...... change, and low blood pressure
@en
Normal pregnancy: mechanisms u ...... change, and low blood pressure
@nl
prefLabel
Normal pregnancy: mechanisms u ...... change, and low blood pressure
@ast
Normal pregnancy: mechanisms u ...... change, and low blood pressure
@en
Normal pregnancy: mechanisms u ...... change, and low blood pressure
@nl
P2093
P2860
P1476
Normal pregnancy: mechanisms u ...... change, and low blood pressure
@en
P2093
Brandiese E Jacobs
John M Hamlyn
Maria V Pulina
Vera A Golovina
P2860
P304
P356
10.1152/AJPHEART.00532.2011
P577
2012-01-13T00:00:00Z