Lysyl oxidase activity is dysregulated during impaired alveolarization of mouse and human lungs. - sprookjes - yvandenbroek - TriplyDB

Lysyl oxidase activity is dysregulated during impaired alveolarization of mouse and human lungs.

Lysyl oxidase activity is dysregulated during impaired alveolarization of mouse and human lungs.

http://www.wikidata.org/entity/Q33746190

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The Extracellular Matrix in Bronchopulmonary Dysplasia: Target and Source Interleukin-1 beta influences on lysyl oxidases and matrix metalloproteinases profile of injured anterior cruciate ligament and medial collateral ligament fibroblasts Resolvin D1 and lipoxin A4 improve alveolarization and normalize septal wall thickness in a neonatal murine model of hyperoxia-induced lung injury Chronic lung disease in the preterm infant. Lessons learned from animal models Pseudoexfoliation syndrome-associated genetic variants affect transcription factor binding and alternative splicing of LOXL1.Transglutaminase 2: a new player in bronchopulmonary dysplasia?Reduction of microRNA-206 contributes to the development of bronchopulmonary dysplasia through up-regulation of fibronectin 1.Aberrant signaling pathways of the lung mesenchyme and their contributions to the pathogenesis of bronchopulmonary dysplasia The H2S-generating enzymes cystathionine β-synthase and cystathionine γ-lyase play a role in vascular development during normal lung alveolarization.Transforming growth factor-β regulates endothelin-1 signaling in the newborn mouse lung during hypoxia exposure.Mesenchymal stem cell-mediated reversal of bronchopulmonary dysplasia and associated pulmonary hypertension.Prenatal inflammation exacerbates hyperoxia-induced functional and structural changes in adult mice.Mechanisms of bronchopulmonary dysplasia.VARA attenuates hyperoxia-induced impaired alveolar development and lung function in newborn mice Glucocorticoids recruit Tgfbr3 and Smad1 to shift transforming growth factor-β signaling from the Tgfbr1/Smad2/3 axis to the Acvrl1/Smad1 axis in lung fibroblasts.Deregulation of the lysyl hydroxylase matrix cross-linking system in experimental and clinical bronchopulmonary dysplasia.JNK suppresses pulmonary fibroblast elastogenesis during alveolar development.Update in pediatric lung disease 2009.Recent advances in late lung development and the pathogenesis of bronchopulmonary dysplasia.Altered lung development in bronchopulmonary dysplasia.Advances in paediatric pulmonary vascular disease associated with bronchopulmonary dysplasia.Recent advances in the mechanisms of lung alveolarization and the pathogenesis of bronchopulmonary dysplasia.Collagen and elastin cross-linking is altered during aberrant late lung development associated with hyperoxia.A three-dimensional human model of the fibroblast activation that accompanies bronchopulmonary dysplasia identifies Notch-mediated pathophysiology.Copper Deficiency in the Lungs of TNF-α Transgenic Mice.Extracellular matrix structure and tissue stiffness control postnatal lung development through the lipoprotein receptor-related protein 5/Tie2 signaling system.Systemic hydrogen sulfide administration partially restores normal alveolarization in an experimental animal model of bronchopulmonary dysplasia.The BPD trio? Interaction of dysregulated PDGF, VEGF, and TGF signaling in neonatal chronic lung disease.Transforming growth factor-β stimulates Smad1/5 signaling in pulmonary artery smooth muscle cells and fibroblasts of the newborn mouse through ALK1.Recent advances in our understanding of the mechanisms of late lung development and bronchopulmonary dysplasia.Extracellular matrix in lung development, homeostasis and disease.Alterations in expression of elastogenic and angiogenic genes by different conditions of mechanical ventilation in newborn rat lung.BMP7 regulates lung fibroblast proliferation in newborn rats with bronchopulmonary dysplasia.Perturbations to lysyl oxidase expression broadly influence the transcriptome of lung fibroblasts.Transcriptome profiling of the newborn mouse lung after hypoxia and reoxygenation: hyperoxic reoxygenation affects mTOR signaling pathway, DNA repair, and JNK-pathway regulation.microRNA-122down-regulation may play a role in severe myocardial fibrosis in human aortic stenosis through TGF-β1up-regulation

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P2860

Lysyl oxidase activity is dysregulated during impaired alveolarization of mouse and human lungs.

http://www.wikidata.org/entity/Q33746190

description

2009 nî lūn-bûn

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2009 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2009 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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Lysyl oxidase activity is dysr ...... tion of mouse and human lungs.

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Lysyl oxidase activity is dysr ...... tion of mouse and human lungs.

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Lysyl oxidase activity is dysr ...... tion of mouse and human lungs.

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RCCM.200902-0215OC

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American Journal of Respiratory and Critical Care Medicine

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Lysyl oxidase activity is dysr ...... tion of mouse and human lungs.

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Alexander H Nave

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Arun Kumarasamy

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Eva Dony

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Irene van der Horst

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Irwin Reiss

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Isabelle Schmitt

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Jesse D Roberts

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Oliver Eickelberg

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Ronald R de Krijger

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P2860

Lysyl oxidase is essential for normal development and function of the respiratory system and for the integrity of elastic and collagen fibers in various tissues

A molecular 'signature' of primary breast cancer cultures; patterns resembling tumor tissue

Lysyl oxidases: a novel multifunctional amine oxidase family

Bronchopulmonary dysplasia

Lysyl oxidase: properties, specificity, and biological roles inside and outside of the cell

Elastic fiber homeostasis requires lysyl oxidase-like 1 protein

Pulmonary disease following respirator therapy of hyaline-membrane disease. Bronchopulmonary dysplasia

Inactivation of the lysyl oxidase gene Lox leads to aortic aneurysms, cardiovascular dysfunction, and perinatal death in mice

Vascular endothelial growth factor gene therapy increases survival, promotes lung angiogenesis, and prevents alveolar damage in hyperoxia-induced lung injury: evidence that angiogenesis participates in alveolarization

Oxygen toxicity in the newborn. The effect of prolonged 100 per cent O2 exposure on the lungs of newborn mice.

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Ralph T Schermuly

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