Dlk1 is necessary for proper skeletal muscle development and regeneration. - sprookjes - yvandenbroek - TriplyDB

Dlk1 is necessary for proper skeletal muscle development and regeneration.

Dlk1 is necessary for proper skeletal muscle development and regeneration.

http://www.wikidata.org/entity/Q33760580

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Meat Science and Muscle Biology Symposium: stem cell niche and postnatal muscle growth Ephrin-A3 promotes and maintains slow muscle fiber identity during postnatal development and reinnervation.Park7 expression influences myotube size and myosin expression in muscle PPARδ regulates satellite cell proliferation and skeletal muscle regeneration Localizing transcriptional regulatory elements at the mouse Dlk1 locus Consequences of MEGF10 deficiency on myoblast function and Notch1 interactions.Small RNAome profiling from human skeletal muscle: novel miRNAs and their targets associated with cancer cachexia.Comparative analyses by sequencing of transcriptomes during skeletal muscle development between pig breeds differing in muscle growth rate and fatness.Barx2 and Pax7 have antagonistic functions in regulation of wnt signaling and satellite cell differentiation.Delta-like 1 homolog (dlk1): a marker for rhabdomyosarcomas implicated in skeletal muscle regeneration.Gene coexpression networks reveal key drivers of phenotypic divergence in porcine muscle Differential pathways to adult metabolic dysfunction following poor nutrition at two critical developmental periods in sheep.Identification of telocytes in skeletal muscle interstitium: implication for muscle regeneration Early life nutrition modulates muscle stem cell number: implications for muscle mass and repair.The role of Notch signaling in muscle progenitor cell depletion and the rapid onset of histopathology in muscular dystrophy.Heterogeneous activation of a slow myosin gene in proliferating myoblasts and differentiated single myofibers Inducible Deletion of Protein Kinase Map4k4 in Obese Mice Improves Insulin Sensitivity in Liver and Adipose Tissues Runx1 Transcription Factor Is Required for Myoblasts Proliferation during Muscle Regeneration Intramuscular adipose is derived from a non-Pax3 lineage and required for efficient regeneration of skeletal muscles Nitric oxide sustains long-term skeletal muscle regeneration by regulating fate of satellite cells via signaling pathways requiring Vangl2 and cyclic GMP.Low levels of Survival Motor Neuron protein are sufficient for normal muscle function in the SMNΔ7 mouse model of SMA.Early myogenic responses to acute exercise before and after resistance training in young men.Regulation of DLK1 by the maternally expressed miR-379/miR-544 cluster may underlie callipyge polar overdominance inheritance.Myostatin facilitates slow and inhibits fast myosin heavy chain expression during myogenic differentiation.Data supporting possible implication of APOBEC2 in self-renewal functions of myogenic stem satellite cells: Toward understanding the negative regulation of myoblast differentiation MEF2A regulates the Gtl2-Dio3 microRNA mega-cluster to modulate WNT signaling in skeletal muscle regeneration Conditional deletions refine the embryonic requirement for Dlk1.Notch Signaling Mediates Skeletal Muscle Atrophy in Cancer Cachexia Caused by Osteosarcoma.The paternally imprinted DLK1-GTL2 locus is differentially methylated in embryonal and alveolar rhabdomyosarcomas.Integrated Analysis of Long Non-coding RNAs (LncRNAs) and mRNA Expression Profiles Reveals the Potential Role of LncRNAs in Skeletal Muscle Development of the Chicken Deletion of the Ste20-like kinase SLK in skeletal muscle results in a progressive myopathy and muscle weakness.Genes contributing to genetic variation of muscling in sheep.Exercise-associated DNA methylation change in skeletal muscle and the importance of imprinted genes: a bioinformatics meta-analysis.New insights into polar overdominance in callipyge sheep.Effects of transforming growth factor-β1 treatment on muscle regeneration and adipogenesis in glycerol-injured muscle.Membrane-bound delta-like 1 homolog (Dlk1) promotes while soluble Dlk1 inhibits myogenesis in C2C12 cells.KLF7 Regulates Satellite Cell Quiescence in Response to Extracellular Signaling.Prenatal notch1 receptor blockade by protein delta homolog 1 (DLK1) modulates adipocyte size in vivo.The testicular transcriptome associated with spermatogonia differentiation initiated by gonadotrophin stimulation in the juvenile rhesus monkey (Macaca mulatta).Constitutive Notch activation upregulates Pax7 and promotes the self-renewal of skeletal muscle satellite cells.

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Dlk1 is necessary for proper skeletal muscle development and regeneration.

http://www.wikidata.org/entity/Q33760580

description

2010 nî lūn-bûn

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2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Dlk1 is necessary for proper skeletal muscle development and regeneration.

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Dlk1 is necessary for proper skeletal muscle development and regeneration.

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type

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Dlk1 is necessary for proper skeletal muscle development and regeneration.

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Dlk1 is necessary for proper skeletal muscle development and regeneration.

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Dlk1 is necessary for proper skeletal muscle development and regeneration.

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Dlk1 is necessary for proper skeletal muscle development and regeneration.

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JOURNAL.PONE.0015055

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Dlk1 is necessary for proper skeletal muscle development and regeneration.

@en

P2093

Aleksey Yevtodiyenko

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Christopher A Bidwell

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Jennifer V Schmidt

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Jolena N Waddell

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Sanjay K Gupta

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P2860

A novel human muscle factor related to but distinct from MyoD1 induces myogenic conversion in 10T1/2 fibroblasts

Pref-1 regulates mesenchymal cell commitment and differentiation through Sox9

PRDM16 controls a brown fat/skeletal muscle switch

Expression of PEG11 and PEG11AS transcripts in normal and callipyge sheep

Gene dosage effects of the imprinted delta-like homologue 1 (dlk1/pref1) in development: implications for the evolution of imprinting

dlk, a putative mammalian homeotic gene differentially expressed in small cell lung carcinoma and neuroendocrine tumor cell line

The role of the epidermal growth factor-like protein dlk in cell differentiation

Pref-1, a protein containing EGF-like repeats, inhibits adipocyte differentiation

Pref-1 interacts with fibronectin to inhibit adipocyte differentiation

Premature myogenic differentiation and depletion of progenitor cells cause severe muscle hypotrophy in Delta1 mutants

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10.1371/JOURNAL.PONE.0015055

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2993959

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