Mutants of Mycobacterium tuberculosis lacking three of the five rpf-like genes are defective for growth in vivo and for resuscitation in vitro.
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Genome sequence of the Fleming strain of Micrococcus luteus, a simple free-living actinobacteriumIndividuality, phenotypic differentiation, dormancy and 'persistence' in culturable bacterial systems: commonalities shared by environmental, laboratory, and clinical microbiologyProtein complexes and proteolytic activation of the cell wall hydrolase RipA regulate septal resolution in mycobacteriaA partner for the resuscitation-promoting factors of Mycobacterium tuberculosisDeletion of the Mycobacterium tuberculosis resuscitation-promoting factor Rv1009 gene results in delayed reactivation from chronic tuberculosisA mycobacterial enzyme essential for cell division synergizes with resuscitation-promoting factorCharacterization of a Clp protease gene regulator and the reaeration response in Mycobacterium tuberculosisWhole genome identification of Mycobacterium tuberculosis vaccine candidates by comprehensive data mining and bioinformatic analysesThe role of resuscitation promoting factors in pathogenesis and reactivation of Mycobacterium tuberculosis during intra-peritoneal infection in mice.Finding of the low molecular weight inhibitors of resuscitation promoting factor enzymatic and resuscitation activity.Interaction and modulation of two antagonistic cell wall enzymes of mycobacteria.The importance of the viable but non-culturable state in human bacterial pathogens.DNA immunization of Mycobacterium tuberculosis resuscitation-promoting factor B elicits polyfunctional CD8(+) T cell responses.The Rip1 protease of Mycobacterium tuberculosis controls the SigD regulon.The extracytoplasmic domain of the Mycobacterium tuberculosis Ser/Thr kinase PknB binds specific muropeptides and is required for PknB localizationIndividual Mycobacterium tuberculosis universal stress protein homologues are dispensable in vitro.Massive pulmonary tuberculosis cavity misdiagnosed as pneumothorax.Latent tuberculosis infection: myths, models, and molecular mechanismsType VI secretion system effectors: poisons with a purpose.Potassium availability triggers Mycobacterium tuberculosis transition to, and resuscitation from, non-culturable (dormant) states.Bacterial growth and cell division: a mycobacterial perspective.Thirty years of viable but nonculturable state research: unsolved molecular mechanisms.Subunit vaccine consisting of multi-stage antigens has high protective efficacy against Mycobacterium tuberculosis infection in mice.Identification of human T-cell responses to Mycobacterium tuberculosis resuscitation-promoting factors in long-term latently infected individuals.Comparative genomics for mycobacterial peptidoglycan remodelling enzymes reveals extensive genetic multiplicityNMR Structure and Dynamics of the Resuscitation Promoting Factor RpfC Catalytic DomainVaccines for tuberculosis: novel concepts and recent progress.Mixed-strain mycobacterium tuberculosis infections and the implications for tuberculosis treatment and controlThe resuscitation-promoting factors of Mycobacterium tuberculosis are required for virulence and resuscitation from dormancy but are collectively dispensable for growth in vitro.Expression, purification and characterization of Mycobacterium tuberculosis RpfE proteinA multistate tuberculosis pharmacometric model: a framework for studying anti-tubercular drug effects in vitro.A Mycobacterium tuberculosis Rpf double-knockout strain exhibits profound defects in reactivation from chronic tuberculosis and innate immunity phenotypes.A Flow Cytometry Method for Rapidly Assessing Mycobacterium tuberculosis Responses to Antibiotics with Different Modes of ActionBacterial peptidoglycan (murein) hydrolases.Tuberculosis genes expressed during persistence and reactivation in the resistant rabbit model.Not to wake a sleeping giant: new insights into host-pathogen interactions identify new targets for vaccination against latent Mycobacterium tuberculosis infection.TB vaccines: current status and future perspectives.A hydrolase of trehalose dimycolate induces nutrient influx and stress sensitivity to balance intracellular growth of Mycobacterium tuberculosis.For better or for worse: the immune response against Mycobacterium tuberculosis balances pathology and protection.Resuscitation promoting factors: a family of microbial proteins in survival and resuscitation of dormant mycobacteria
P2860
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P2860
Mutants of Mycobacterium tuberculosis lacking three of the five rpf-like genes are defective for growth in vivo and for resuscitation in vitro.
description
2005 nî lūn-bûn
@nan
2005 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Mutants of Mycobacterium tuber ...... nd for resuscitation in vitro.
@ast
Mutants of Mycobacterium tuber ...... nd for resuscitation in vitro.
@en
type
label
Mutants of Mycobacterium tuber ...... nd for resuscitation in vitro.
@ast
Mutants of Mycobacterium tuber ...... nd for resuscitation in vitro.
@en
prefLabel
Mutants of Mycobacterium tuber ...... nd for resuscitation in vitro.
@ast
Mutants of Mycobacterium tuber ...... nd for resuscitation in vitro.
@en
P2093
P2860
P1476
Mutants of Mycobacterium tuber ...... nd for resuscitation in vitro.
@en
P2093
Arseny S Kaprelyants
Danielle I Young
Katrina J Downing
Margarita O Shleeva
Michael Young
Vladimir V Mischenko
P2860
P304
P356
10.1128/IAI.73.5.3038-3043.2005
P407
P577
2005-05-01T00:00:00Z