Cytokine regulation of MMP-9 in peripheral glia: implications for pathological processes and pain in injured nerve. - sprookjes - yvandenbroek - TriplyDB

Cytokine regulation of MMP-9 in peripheral glia: implications for pathological processes and pain in injured nerve.

Cytokine regulation of MMP-9 in peripheral glia: implications for pathological processes and pain in injured nerve.

http://www.wikidata.org/entity/Q33834853

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Wallerian degeneration: the innate-immune response to traumatic nerve injury NOV/CCN3 attenuates inflammatory pain through regulation of matrix metalloproteinases-2 and -9 Folic Acid Modulates Matrix Metalloproteinase-2 Expression, Alleviates Neuropathic Pain, and Improves Functional Recovery in Spinal Cord-Injured Rats.Transgenic inhibition of glial NF-kappa B reduces pain behavior and inflammation after peripheral nerve injury.MMPs initiate Schwann cell-mediated MBP degradation and mechanical nociception after nerve damage.Matrix metalloproteinase inhibition enhances the rate of nerve regeneration in vivo by promoting dedifferentiation and mitosis of supporting schwann cells The MMP-9/TIMP-1 axis controls the status of differentiation and function of myelin-forming Schwann cells in nerve regeneration.Light-emitting diode therapy induces analgesia and decreases spinal cord and sciatic nerve tumour necrosis factor-α levels after sciatic nerve crush in mice.Targeting anti-inflammatory treatment can ameliorate injury-induced neuropathic pain.Role of matrix metalloproteinases and therapeutic benefits of their inhibition in spinal cord injury.Estrogen in cycling rats alters gene expression in the temporomandibular joint, trigeminal ganglia and trigeminal subnucleus caudalis/upper cervical cord junction Upregulation of inflammatory mediators in a model of chronic pain after spinal cord injury Immunodominant fragments of myelin basic protein initiate T cell-dependent pain.A shed form of LDL receptor-related protein-1 regulates peripheral nerve injury and neuropathic pain in rodents."Listening" and "talking" to neurons: implications of immune activation for pain control and increasing the efficacy of opioids.Pain as a symptom of peripheral nerve sheath tumors: clinical significance and future therapeutic directions.MMP-9 controls Schwann cell proliferation and phenotypic remodeling via IGF-1 and ErbB receptor-mediated activation of MEK/ERK pathway.Leptin derived from adipocytes in injured peripheral nerves facilitates development of neuropathic pain via macrophage stimulation Genome-wide expression profiling of complex regional pain syndrome Spinal matrix metalloproteinase-9 contributes to physical dependence on morphine in mice.CSD, BBB and MMP-9 elevations: animal experiments versus clinical phenomena in migraine.Interleukin-17 impedes Schwann cell-mediated myelination.Effect of subcutaneous treatment with human umbilical cord blood-derived multipotent stem cells on peripheral neuropathic pain in rats.Therapeutic potential of matrix metalloprotease inhibitors in neuropathic pain.N-acetyl-cysteine attenuates remifentanil-induced postoperative hyperalgesia via inhibiting matrix metalloproteinase-9 in dorsal root ganglia.Extrinsic cellular and molecular mediators of peripheral axonal regeneration.The neuroimmunology of degeneration and regeneration in the peripheral nervous system.Matrix metalloproteinases: potential therapeutic target for diabetic neuropathic pain.MMP2-9 cleavage of dystroglycan alters the size and molecular composition of Schwann cell domains.The Glia Response after Peripheral Nerve Injury: A Comparison between Schwann Cells and Olfactory Ensheathing Cells and Their Uses for Neural Regenerative Therapies.Inflammatory Cytokines Are Involved in Focal Demyelination in Leprosy Neuritis.Reciprocal relationship between membrane type 1 matrix metalloproteinase and the algesic peptides of myelin basic protein contributes to chronic neuropathic pain.Distinct roles of matrix metalloproteases in the early- and late-phase development of neuropathic pain Role of Matrix Metalloproteinases 2 in Spinal Cord Injury-Induced Neuropathic Pain.Autotaxin and lysophosphatidic acid1 receptor-mediated demyelination of dorsal root fibers by sciatic nerve injury and intrathecal lysophosphatidylcholine.Transcriptional mechanisms underlying sensitization of peripheral sensory neurons by granulocyte-/granulocyte-macrophage colony stimulating factors.Matrix metalloprotease regulation of neuropathic pain.Tissue Inhibitor of Metalloproteinase-3 Promotes Schwann Cell Myelination.Paeoniflorin attenuates postoperative pain by suppressing Matrix Metalloproteinase-9/2 in mice.Computational Approaches to Matrix Metalloprotease Drug Design.

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Cytokine regulation of MMP-9 in peripheral glia: implications for pathological processes and pain in injured nerve.

http://www.wikidata.org/entity/Q33834853

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2006 nî lūn-bûn

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2006 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2006 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年论文

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Cytokine regulation of MMP-9 i ...... ses and pain in injured nerve.

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Cytokine regulation of MMP-9 i ...... ses and pain in injured nerve.

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Cytokine regulation of MMP-9 i ...... ses and pain in injured nerve.

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J.BBI.2006.10.015

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Brain, Behavior, and Immunity

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Cytokine regulation of MMP-9 i ...... ses and pain in injured nerve.

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Julie Janes

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Robert R Myers

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Sharmila Chattopadhyay

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Veronica Shubayev

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P2860

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

A new mathematical model for relative quantification in real-time RT-PCR

The role of neuroinflammation in neuropathic pain: mechanisms and therapeutic targets

Characterisation of matrix metalloproteinases and the effects of a broad-spectrum inhibitor (BB-1101) in peripheral nerve regeneration

Endoneurial remodeling by TNFalph- and TNFalpha-releasing proteases. A spatial and temporal co-localization study in painful neuropathy.

Matrix metalloproteinase expression and inhibition after sciatic nerve axotomy.

Absence of mechanical allodynia and Abeta-fiber sprouting after sciatic nerve injury in mice lacking membrane-type 5 matrix metalloproteinase.

Matrix metalloproteinases in inflammatory demyelination: targets for treatment.

Comparison of matrix metalloproteinase expression during Wallerian degeneration in the central and peripheral nervous systems.

An experimental model for peripheral neuropathy produced by segmental spinal nerve ligation in the rat.

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