T cell hyperactivity in lupus as a consequence of hyperstimulatory antigen-presenting cells.
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Dendritic Cells in Systemic Lupus Erythematosus: From Pathogenic Players to Therapeutic ToolsEnvironment versus genetics in autoimmunity: a geneticist's perspectiveTLR ligands up-regulate Trex1 expression in murine conventional dendritic cells through type I Interferon and NF-κB-dependent signaling pathwaysDemethylzeylasteral (T-96) Treatment Ameliorates Mice Lupus Nephritis Accompanied by Inhibiting Activation of NF-κB PathwayEstrogen receptor alpha modulates Toll-like receptor signaling in murine lupusImmune dysregulation accelerates atherosclerosis and modulates plaque composition in systemic lupus erythematosusDysregulated CD4+ T cells from SLE-susceptible mice are sufficient to accelerate atherosclerosis in LDLr-/- mice.The lupus susceptibility locus Sle3 is not sufficient to accelerate atherosclerosis in lupus-susceptible low density lipoprotein receptor-deficient mice.Heat shock protein 90 inhibition by 17-DMAG lessens disease in the MRL/lpr mouse model of systemic lupus erythematosusAutoimmune effector memory T cells: the bad and the goodType I interferons produced by resident renal cells may promote end-organ disease in autoantibody-mediated glomerulonephritis.Dendritic cells in systemic lupus erythematosusMyeloid cells, BAFF, and IFN-gamma establish an inflammatory loop that exacerbates autoimmunity in Lyn-deficient mice.Dendritic cells in lupus are not required for activation of T and B cells but promote their expansion, resulting in tissue damageMurine models of systemic lupus erythematosusLatent membrane protein 1, the EBV-encoded oncogenic mimic of CD40, accelerates autoimmunity in B6.Sle1 miceAbnormal costimulatory phenotype and function of dendritic cells before and after the onset of severe murine lupusThree checkpoints in lupus development: central tolerance in adaptive immunity, peripheral amplification by innate immunity and end-organ inflammation.OX40/OX40L in systemic lupus erythematosus: association with disease activity and lupus nephritis.Manipulating autophagic processes in autoimmune diseases: a special focus on modulating chaperone-mediated autophagy, an emerging therapeutic target.TNF-α and TGF-β counter-regulate PD-L1 expression on monocytes in systemic lupus erythematosus.Genetic and cellular dissection of the activation of AM14 rheumatoid factor B cells in a mouse model of lupusShared signaling networks active in B cells isolated from genetically distinct mouse models of lupus.Murine gammaherpesvirus 68 infection protects lupus-prone mice from the development of autoimmunity.Alpha 1 Antitrypsin Inhibits Dendritic Cell Activation and Attenuates Nephritis in a Mouse Model of Lupus.Macrophages from lupus-prone MRL mice have a conditional signaling abnormality that leads to dysregulated expression of numerous genes.SLE: translating lessons from model systems to human diseaseB cell TLR7 expression drives anti-RNA autoantibody production and exacerbates disease in systemic lupus erythematosus-prone mice.Urine VCAM-1 as a marker of renal pathology activity index in lupus nephritisModulating proximal cell signaling by targeting Btk ameliorates humoral autoimmunity and end-organ disease in murine lupus.Blockade of CD354 (TREM-1) Ameliorates Anti-GBM-Induced NephritisAntigen presenting cell abnormalities in the Cln3(-/-) mouse model of juvenile neuronal ceroid lipofuscinosisAutoimmunity stimulated by adoptively transferred dendritic cells is initiated by both alphabeta and gammadelta T cells but does not require MyD88 signaling.Genetics of clinical expression in SLE.Kallikrein genes are associated with lupus and glomerular basement membrane-specific antibody-induced nephritis in mice and humans.Identification of IRAK1 as a risk gene with critical role in the pathogenesis of systemic lupus erythematosus.Apigenin, a non-mutagenic dietary flavonoid, suppresses lupus by inhibiting autoantigen presentation for expansion of autoreactive Th1 and Th17 cellsPTPN22 deficiency cooperates with the CD45 E613R allele to break tolerance on a non-autoimmune background.Deficiency of type I IFN receptor in lupus-prone New Zealand mixed 2328 mice decreases dendritic cell numbers and activation and protects from disease.Dendritic cell function in lupus: Independent contributors or victims of aberrant immune regulation.
P2860
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P2860
T cell hyperactivity in lupus as a consequence of hyperstimulatory antigen-presenting cells.
description
2005 nî lūn-bûn
@nan
2005 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
name
T cell hyperactivity in lupus ...... tory antigen-presenting cells.
@en
type
label
T cell hyperactivity in lupus ...... tory antigen-presenting cells.
@en
prefLabel
T cell hyperactivity in lupus ...... tory antigen-presenting cells.
@en
P2093
P2860
P356
P1476
T cell hyperactivity in lupus ...... tory antigen-presenting cells.
@en
P2093
Chandra Mohan
Edward K Wakeland
Eric S Sobel
Jiankun Zhu
Xuebin Liu
P2860
P304
P356
10.1172/JCI23049
P407
P577
2005-06-09T00:00:00Z