Kallikrein 6 signals through PAR1 and PAR2 to promote neuron injury and exacerbate glutamate neurotoxicity.
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Involvement of Kallikrein-Related Peptidases in Normal and Pathologic ProcessesProteinases, their receptors and inflammatory signalling: the Oxford South Parks Road connectionAstrocyte heterogeneity across the brain and spinal cord occurs developmentally, in adulthood and in response to demyelination.Kallikrein cascades in traumatic spinal cord injury: in vitro evidence for roles in axonopathy and neuron degeneration.Differential expression of multiple kallikreins in a viral model of multiple sclerosis points to unique roles in the innate and adaptive immune response.The thrombin receptor is a critical extracellular switch controlling myelination.Genetic targeting of protease activated receptor 2 reduces inflammatory astrogliosis and improves recovery of function after spinal cord injuryProtein-Based Classifier to Predict Conversion from Clinically Isolated Syndrome to Multiple SclerosisInterplay between exercise and dietary fat modulates myelinogenesis in the central nervous system.Protease-activated receptor-1 deficiency protects against streptozotocin-induced diabetic nephropathy in mice.KLK6 proteolysis is implicated in the turnover and uptake of extracellular alpha-synuclein species.Thrombin Activity and Thrombin Receptor in Rat Glioblastoma Model: Possible Markers and Targets for Intervention?Inhibitors of kallikrein-related peptidases: An overview.Kallikrein Promotes Inflammation in Human Dental Pulp Cells Via Protease-Activated Receptor-1.Targeting the thrombin receptor modulates inflammation and astrogliosis to improve recovery after spinal cord injuryInterference with protease-activated receptor 1 does not reduce damage to subventricular zone cells of immature rodent brain following exposure to blood or blood plasma.Neuro-Coagulopathy: Blood Coagulation Factors in Central Nervous System Diseases.Kallikrein-related peptidase-6 (KLK6) mRNA expression is an independent prognostic tissue biomarker of poor disease-free and overall survival in colorectal adenocarcinoma.Kallikrein-related peptidase 6 exacerbates disease in an autoimmune model of multiple sclerosis.Plasmin reduces fibronectin deposition by mesangial cells in a protease-activated receptor-1 independent manner.Protease activated receptor 2 controls myelin development, resiliency and repair.Implication of the Kallikrein-Kinin system in neurological disorders: Quest for potential biomarkers and mechanisms.Nafamostat Mesilate Improves Neurological Outcome and Axonal Regeneration after Stroke in Rats.The Thrombin Receptor Restricts Subventricular Zone Neural Stem Cell Expansion and Differentiation.Kallikrein-related peptidase 6 orchestrates astrocyte form and function through proteinase activated receptor-dependent mechanisms
P2860
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P2860
Kallikrein 6 signals through PAR1 and PAR2 to promote neuron injury and exacerbate glutamate neurotoxicity.
description
2013 nî lūn-bûn
@nan
2013 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Kallikrein 6 signals through P ...... rbate glutamate neurotoxicity.
@ast
Kallikrein 6 signals through P ...... rbate glutamate neurotoxicity.
@en
type
label
Kallikrein 6 signals through P ...... rbate glutamate neurotoxicity.
@ast
Kallikrein 6 signals through P ...... rbate glutamate neurotoxicity.
@en
prefLabel
Kallikrein 6 signals through P ...... rbate glutamate neurotoxicity.
@ast
Kallikrein 6 signals through P ...... rbate glutamate neurotoxicity.
@en
P2093
P2860
P356
P1476
Kallikrein 6 signals through P ...... rbate glutamate neurotoxicity.
@en
P2093
Hyesook Yoon
Isobel A Scarisbrick
Jianmin Wu
Maja Radulovic
Sachiko I Blaber
P2860
P304
P356
10.1111/JNC.12293
P407
P577
2013-05-27T00:00:00Z