Recently transposed Alu repeats result from multiple source genes.
about
A human Alu RNA-binding protein whose expression is associated with accumulation of small cytoplasmic Alu RNAMolecular archeology of L1 insertions in the human genome.Exonization of AluYa5 in the human ACE gene requires mutations in both 3' and 5' splice sites and is facilitated by a conserved splicing enhancer.Whole-genome analysis of Alu repeat elements reveals complex evolutionary historyEvidence that most human Alu sequences were inserted in a process that ceased about 30 million years agoOrigin of the Alu family: a family of Alu-like monomers gave birth to the left and the right arms of the Alu elementsA trinucleotide repeat-associated increase in the level of Alu RNA-binding protein occurred during the same period as the major Alu amplification that accompanied anthropoid evolutionTFIIF facilitates dissociation of RNA polymerase II from noncoding RNAs that lack a repression domainUnder the genomic radar: the stealth model of Alu amplificationLaboratory methods for the analysis of primate mobile elements.A population genetic study of the evolution of SINEs. I. Polymorphism with regard to the presence or absence of an element.Details of retropositional genome dynamics that provide a rationale for a generic division: the distinct branching of all the pacific salmon and trout (Oncorhynchus) from the Atlantic salmon and trout (Salmo).A population genetic study of the evolution of SINEs. II. Sequence evolution under the master copy modelRNA polymerase III promoter and terminator elements affect Alu RNA expression.The role of recombination in the origin and evolution of Alu subfamiliesLamellar spacing in cuboid hydroxyapatite scaffolds regulates bone formation by human bone marrow stromal cellsFusion of a free left Alu monomer and a free right Alu monomer at the origin of the Alu family in the primate genomesRodent BC1 RNA gene as a master gene for ID element amplification.Evolutionary selection against change in many Alu repeat sequences interspersed through primate genomesThe subset of mouse B1 (Alu-equivalent) sequences expressed as small processed cytoplasmic transcriptsSpecies-specific amplification of tRNA-derived short interspersed repetitive elements (SINEs) by retroposition: a process of parasitization of entire genomes during the evolution of salmonids.African origin of human-specific polymorphic Alu insertions.The emergence of new DNA repeats and the divergence of primates.The decline in human Alu retroposition was accompanied by an asymmetric decrease in SRP9/14 binding to dimeric Alu RNA and increased expression of small cytoplasmic Alu RNA.Activation of RNA polymerase III transcription of human Alu repetitive elements by adenovirus type 5: requirement for the E1b 58-kilodalton protein and the products of E4 open reading frames 3 and 6.Multiple dispersed loci produce small cytoplasmic Alu RNA.Developmental differences in methylation of human Alu repeatsThe RNA polymerase III terminator used by a B1-Alu element can modulate 3' processing of the intermediate RNA productPatterns of insertions and their covariation with substitutions in the rat, mouse, and human genomes.Creation of new bone by the percutaneous injection of human bone marrow stromal cell and HA/TCP suspensionsDifferential binding of human nuclear proteins to Alu subfamilies.Proposed roles for DNA methylation in Alu transcriptional repression and mutational inactivation.A new subfamily of recently retroposed human Alu repeats.Wrapping of genomic polydA.polydT tracts around nucleosome core particles.Amplification dynamics of human-specific (HS) Alu family members.Human Alu subfamilies and their methylation revealed by blot hybridizationAn Alu element retroposition in two families with Huntington disease defines a new active Alu subfamily.Molecular analysis of the chromosomal breakpoint and fusion transcripts in the acute lymphoblastic SEM cell line with chromosomal translocation t(4;11).An evolution-based hypothesis on the origin and mechanisms of autoimmune disease.Evolutionary dynamics of selfish DNA explains the abundance distribution of genomic subsequences.
P2860
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P2860
Recently transposed Alu repeats result from multiple source genes.
description
1990 nî lūn-bûn
@nan
1990 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1990 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
name
Recently transposed Alu repeats result from multiple source genes.
@ast
Recently transposed Alu repeats result from multiple source genes.
@en
type
label
Recently transposed Alu repeats result from multiple source genes.
@ast
Recently transposed Alu repeats result from multiple source genes.
@en
prefLabel
Recently transposed Alu repeats result from multiple source genes.
@ast
Recently transposed Alu repeats result from multiple source genes.
@en
P2093
P2860
P356
P1476
Recently transposed Alu repeats result from multiple source genes
@en
P2093
P2860
P304
P356
10.1093/NAR/18.20.6019
P407
P577
1990-10-01T00:00:00Z