A defect in interleukin-10 leads to enhanced malarial disease in Plasmodium chabaudi chabaudi infection in mice.
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Polymorphisms in Plasmodium vivax Circumsporozoite Protein (CSP) Influence Parasite Burden and Cytokine Balance in a Pre-Amazon Endemic Area from BrazilIL-4 inhibits TGF-beta-induced Foxp3+ T cells and, together with TGF-beta, generates IL-9+ IL-10+ Foxp3(-) effector T cellsPlasmodium strain determines dendritic cell function essential for survival from malariaIL-10 from CD4CD25Foxp3CD127 adaptive regulatory T cells modulates parasite clearance and pathology during malaria infection.Gastrointestinal nematode infection exacerbates malaria-induced liver pathology.Malaria parasite infection compromises control of concurrent systemic non-typhoidal Salmonella infection via IL-10-mediated alteration of myeloid cell function.Tumor necrosis factor alpha p55 receptor is important for development of memory responses to blood-stage malaria infection.Tumor necrosis factor alpha-mediated toxic shock in Trypanosoma cruzi-infected interleukin 10-deficient miceHost Resistance to Plasmodium-Induced Acute Immune Pathology Is Regulated by Interleukin-10 Receptor Signaling.A deficiency in gamma interferon or interleukin-10 modulates T-Cell-dependent responses to heat shock protein 60 from Histoplasma capsulatum.Impairment of protective immunity to blood-stage malaria by concurrent nematode infection.Malaria immunity in man and mosquito: insights into unsolved mysteries of a deadly infectious disease.Mycobacterium tuberculosis cell wall released fragments by the action of the human lung mucosa modulate macrophages to control infection in an IL-10-dependent mannerThe spleen CD4+ T cell response to blood-stage Plasmodium chabaudi malaria develops in two phases characterized by different propertiesGranulocyte-macrophage colony-stimulating factor-deficient mice have impaired resistance to blood-stage malaria.Regulation of CD8+ T cell responses to infection with parasitic protozoa.Cytokines in the pathogenesis of and protection against malariaEpitope-specific regulation of immunoglobulin class switching in mice immunized with malarial merozoite surface proteins.The mucosal inflammatory response to non-typhoidal Salmonella in the intestine is blunted by IL-10 during concurrent malaria parasite infectionRole of interleukin 10 transcriptional regulation in inflammation and autoimmune diseaseSoluble ST2 in the fetal inflammatory response syndrome: in vivo evidence of activation of the anti-inflammatory limb of the immune responseChanges in antigen-specific cytokine and chemokine responses to Plasmodium falciparum antigens in a highland area of Kenya after a prolonged absence of malaria exposure.Regulatory role of T cells producing both interferon gamma and interleukin 10 in persistent infectionReduced protective efficacy of a blood-stage malaria vaccine by concurrent nematode infectionγδ T cells acquire effector fates in the thymus and differentiate into cytokine-producing effectors in a Listeria model of infection independently of CD28 costimulation.Lack of endogenous IL-10 enhances production of proinflammatory cytokines and leads to Brucella abortus clearance in mice.In pulmonary paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology.Interleukin-10 and immunity against prokaryotic and eukaryotic intracellular pathogens.Exposure-dependent control of malaria-induced inflammation in children.Disruption of IL-21 signaling affects T cell-B cell interactions and abrogates protective humoral immunity to malaria.IL-27 promotes IL-10 production by effector Th1 CD4+ T cells: a critical mechanism for protection from severe immunopathology during malaria infectionPathology of Plasmodium chabaudi chabaudi infection and mortality in interleukin-10-deficient mice are ameliorated by anti-tumor necrosis factor alpha and exacerbated by anti-transforming growth factor beta antibodies.IFN-γ and IL-21 Double Producing T Cells Are Bcl6-Independent and Survive into the Memory Phase in Plasmodium chabaudi Infection.Cytokine responses of CD4+ T cells during a Plasmodium chabaudi chabaudi (ER) blood-stage infection in mice initiated by the natural route of infectionIL-22 Protects Against Liver Pathology and Lethality of an Experimental Blood-Stage Malaria Infection.IL-10-producing and naturally occurring CD4+ Tregs: limiting collateral damageEnhanced protection to Mycobacterium tuberculosis infection in IL-10-deficient mice is accompanied by early and enhanced Th1 responses in the lung.The effect of declining exposure on T cell-mediated immunity to Plasmodium falciparum - an epidemiological "natural experiment".Tuning microenvironments: induction of regulatory T cells by dendritic cellsInterleukin-10 (IL-10) polymorphisms are associated with IL-10 production and clinical malaria in young children.
P2860
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P2860
A defect in interleukin-10 leads to enhanced malarial disease in Plasmodium chabaudi chabaudi infection in mice.
description
1999 nî lūn-bûn
@nan
1999 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
A defect in interleukin-10 lea ...... di chabaudi infection in mice.
@ast
A defect in interleukin-10 lea ...... di chabaudi infection in mice.
@en
A defect in interleukin-10 lea ...... di chabaudi infection in mice.
@nl
type
label
A defect in interleukin-10 lea ...... di chabaudi infection in mice.
@ast
A defect in interleukin-10 lea ...... di chabaudi infection in mice.
@en
A defect in interleukin-10 lea ...... di chabaudi infection in mice.
@nl
prefLabel
A defect in interleukin-10 lea ...... di chabaudi infection in mice.
@ast
A defect in interleukin-10 lea ...... di chabaudi infection in mice.
@en
A defect in interleukin-10 lea ...... di chabaudi infection in mice.
@nl
P2093
P2860
P921
P1476
A defect in interleukin-10 lea ...... di chabaudi infection in mice.
@en
P2093
P2860
P304
P407
P577
1999-09-01T00:00:00Z