Pathology of Plasmodium chabaudi chabaudi infection and mortality in interleukin-10-deficient mice are ameliorated by anti-tumor necrosis factor alpha and exacerbated by anti-transforming growth factor beta antibodies.
about
Complex genetic control of susceptibility to malaria: positional cloning of the Char9 locusEvolution of parasite virulence when host responses cause diseaseEffector memory Th1 CD4 T cells are maintained in a mouse model of chronic malariaPolymorphisms in Plasmodium vivax Circumsporozoite Protein (CSP) Influence Parasite Burden and Cytokine Balance in a Pre-Amazon Endemic Area from BrazilThe contribution of Plasmodium chabaudi to our understanding of malariaRegulation of immunopathogenesis during Plasmodium and Toxoplasma infections: more parallels than distinctions?Plasmodium strain determines dendritic cell function essential for survival from malariaIL-10 from CD4CD25Foxp3CD127 adaptive regulatory T cells modulates parasite clearance and pathology during malaria infection.Distinct clinical and immunologic profiles in severe malarial anemia and cerebral malaria in ZambiaThrombocytopenia May Mediate Disease Severity in Plasmodium falciparum Malaria Through Reduced Transforming Growth Factor Beta-1 Regulation of Proinflammatory and Anti-inflammatory Cytokines.Distinct roles for FOXP3 and FOXP3 CD4 T cells in regulating cellular immunity to uncomplicated and severe Plasmodium falciparum malariaGastrointestinal nematode infection exacerbates malaria-induced liver pathology.Host Resistance to Plasmodium-Induced Acute Immune Pathology Is Regulated by Interleukin-10 Receptor Signaling.Interleukin 10- and Fcgamma receptor-deficient mice resolve Leishmania mexicana lesionsThe spleen CD4+ T cell response to blood-stage Plasmodium chabaudi malaria develops in two phases characterized by different propertiesCirculating soluble endoglin levels in pregnant women in Cameroon and Malawi--associations with placental malaria and fetal growth restriction.Estrogen and progesterone affect responses to malaria infection in female C57BL/6 miceProtein kinase C θ deficiency increases resistance of C57BL/6J mice to Plasmodium berghei infection-induced cerebral malaria.Role of interleukin 10 transcriptional regulation in inflammation and autoimmune diseaseElevated levels of the Plasmodium yoelii homologue of macrophage migration inhibitory factor attenuate blood-stage malaria.Sequestration and histopathology in Plasmodium chabaudi malaria are influenced by the immune response in an organ-specific mannerEpitope-based vaccines with the Anaplasma marginale MSP1a functional motif induce a balanced humoral and cellular immune response in miceInvolvement of gonadal steroids and gamma interferon in sex differences in response to blood-stage malaria infection.Diminished organelle motion in murine Kupffer cells during the erythrocytic stage of malaria.Nitric oxide is involved in the upregulation of IFN-γ and IL-10 mRNA expression by CD8⁺ T cells during the blood stages of P. chabaudi AS infection in CBA/Ca miceMacrophages are critical for cross-protective immunity conferred by Babesia microti against Babesia rodhaini infection in miceIL-27 promotes IL-10 production by effector Th1 CD4+ T cells: a critical mechanism for protection from severe immunopathology during malaria infectionIFN-γ and IL-21 Double Producing T Cells Are Bcl6-Independent and Survive into the Memory Phase in Plasmodium chabaudi Infection.Parasitemia Induces High Plasma Levels of Interleukin-17 (IL-17) and Low Levels of Interleukin-10 (IL-10) and Transforming Growth Factor-ß (TGF-ß) in Pregnant Mice Infected with Malaria.Dendritic cells, pro-inflammatory responses, and antigen presentation in a rodent malaria infection.The immunopathology of experimental visceral leishmaniasis.Does malaria suffer from lack of memory?IL-22 Protects Against Liver Pathology and Lethality of an Experimental Blood-Stage Malaria Infection.Immunity, immunoregulation and the ecology of trichuriasis and ascariasis.Cell-mediated effector molecules and complicated malaria.Regulation of immune response by Plasmodium-infected red blood cells.Insights into deregulated TNF and IL-10 production in malaria: implications for understanding severe malarial anaemia.Activation of transforming growth factor beta by malaria parasite-derived metalloproteinases and a thrombospondin-like moleculePriming of CD4+ T cells and development of CD4+ T cell memory; lessons for malaria.Ascaris co-infection does not alter malaria-induced anaemia in a cohort of Nigerian preschool children.
P2860
Q24679744-73ECE1F5-6A06-4ECB-AB40-18720422BA0DQ27485642-0837510C-D95C-49E5-90DC-540B575D1CABQ28476346-2C4284BA-6FFC-4D97-87AF-E94FADE0D38BQ28550566-AD6E1532-34D9-42BE-B73A-777A5ED51F7CQ29030212-59369F79-6C92-4020-A252-20CF1A3091B4Q30409386-2E820AD8-3106-4528-891A-C3A20C4C2FAFQ33290218-D74B857D-07AE-4080-8D6C-1385A9AFA7A0Q33327498-3938E426-2FEB-4994-A6B9-D8911A9DA19AQ33393958-0300DDE5-4BDA-423B-A582-59563FF6D951Q33422123-E6654AF1-BBAA-4074-AEED-706E37A1EBEFQ33426367-5D6A7FBE-912D-4B1C-AE00-9E9145C4E65FQ33551405-8FB341B8-42F0-4F7D-9795-535E5B3BCF64Q33721737-46A7D00F-D589-47CA-BD45-6DE77EE7083CQ33769299-9F4AC1BD-E515-4B06-A5EC-15165B02B8BDQ33981534-14E3B8EE-DE4C-412E-B6BA-870AB05576C1Q34037890-A996D2B2-43AB-48A1-9DE8-A1E965BC55F7Q34137151-85369FDD-29E9-4935-B13D-46EA685D058CQ34177295-002AE021-692C-4B88-A3F3-CA800921082EQ34262223-EF995735-48A9-4E73-BF2F-BA416B55BECBQ34309728-6F4D3C96-59D4-4DE7-8C64-76C5C2BDF5AFQ34522257-6AA50CA3-F624-4971-90AA-A544D32D0FBEQ34665901-54EC1C62-472E-43B4-9A15-254C9BFA3479Q34681044-6CB0728A-AB23-4F2E-9877-3A0AAEB6BED0Q34699879-AB4A6F71-28DD-4674-A196-28F9F3D3CFA4Q35566860-94118FE1-E75A-41C6-8B83-3B05544AB673Q35665478-6F996133-2350-47E5-B3DC-80AB9D510A55Q35737807-5956D080-FBC5-4710-866A-E43D68421263Q35863341-3E7C0B42-9A38-4F47-98D8-E2D452487F47Q35881400-77D8B5E8-C648-4C7B-A220-534F038CF265Q35884189-97A526EE-FFD3-4651-8056-44F9E7DC973EQ35884243-9A47D164-2B14-4923-B591-F17DE33A72BFQ35884250-1562A7DC-4230-4994-BE7D-2031946014A7Q35928387-B878E0F2-5581-4631-AE49-B9F3966109CAQ36072548-93952D5A-F488-4AA5-AC4C-3F4F03B0E103Q36170491-42EBE2B3-6F2C-426F-AA2C-D9C2936A0773Q36265007-93A0D2FC-49BD-49BC-9528-E877CFD7D625Q36311915-138828A1-6F97-4147-8921-72E49B1A3204Q36371572-EE09E904-BBB3-4EE1-9BDD-8249E8DA135DQ36378330-5D178D42-A7AA-4AD4-91F2-9C3BB4280A54Q36531272-B92A87AC-0DBA-4E10-8F14-B4DAB0BB0E41
P2860
Pathology of Plasmodium chabaudi chabaudi infection and mortality in interleukin-10-deficient mice are ameliorated by anti-tumor necrosis factor alpha and exacerbated by anti-transforming growth factor beta antibodies.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Pathology of Plasmodium chabau ...... growth factor beta antibodies.
@ast
Pathology of Plasmodium chabau ...... growth factor beta antibodies.
@en
type
label
Pathology of Plasmodium chabau ...... growth factor beta antibodies.
@ast
Pathology of Plasmodium chabau ...... growth factor beta antibodies.
@en
prefLabel
Pathology of Plasmodium chabau ...... growth factor beta antibodies.
@ast
Pathology of Plasmodium chabau ...... growth factor beta antibodies.
@en
P2093
P2860
P921
P1476
Pathology of Plasmodium chabau ...... growth factor beta antibodies.
@en
P2093
Fakhreldin Omer
Latifu A Sanni
P2860
P304
P356
10.1128/IAI.71.9.4850-4856.2003
P407
P577
2003-09-01T00:00:00Z