STAT6 transcription factor is a facilitator of the nuclear receptor PPARγ-regulated gene expression in macrophages and dendritic cells
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Is the Mouse a Good Model of Human PPARγ-Related Metabolic Diseases?Redox Changes Induced by General Anesthesia in Critically Ill Patients with Multiple TraumasEosinophils Reduce Chronic Inflammation in Adipose Tissue by Secreting Th2 Cytokines and Promoting M2 Macrophages PolarizationMinireview: Conversing with chromatin: the language of nuclear receptorsMitochondria in the regulation of innate and adaptive immunityMechanisms driving macrophage diversity and specialization in distinct tumor microenvironments and parallelisms with other tissuesMetabolic reprogramming in macrophages and dendritic cells in innate immunityMetabolic control of dendritic cell activation and function: recent advances and clinical implicationsReprogramming of lysosomal gene expression by interleukin-4 and Stat6Macrophage plasticity and polarization: in vivo veritasPPARγ agonists regulate tobacco smoke-induced Toll like receptor 4 expression in alveolar macrophages.Macrophages in homeostatic immune function.Kruppel-like transcription factor 6 regulates inflammatory macrophage polarization.Mechanisms of inflammatory responses in obese adipose tissue.The active enhancer network operated by liganded RXR supports angiogenic activity in macrophages.A decade of progress in adipose tissue macrophage biology.A neurodegeneration-specific gene-expression signature of acutely isolated microglia from an amyotrophic lateral sclerosis mouse modelThe biology of nematode- and IL4Rα-dependent murine macrophage polarization in vivo as defined by RNA-Seq and targeted lipidomics.PPARs are a unique set of fatty acid regulated transcription factors controlling both lipid metabolism and inflammationThe evolving biology of microglia in Alzheimer's disease.Peroxisome proliferator-activated receptor γ deficiency in T cells accelerates chronic rejection by influencing the differentiation of CD4+ T cells and alternatively activated macrophagesEvidence-based translation for the genomic responses of murine models for the study of human immunity.The PPAR-γ antagonist GW9662 elicits differentiation of M2c-like cells and upregulation of the MerTK/Gas6 axis: a key role for PPAR-γ in human macrophage polarizationInhibition of PPARγ in myeloid-lineage cells induces systemic inflammation, immunosuppression, and tumorigenesis.Monoamine oxidase A (MAO-A): a signature marker of alternatively activated monocytes/macrophagesCarboxypeptidase-M is regulated by lipids and CSFs in macrophages and dendritic cells and expressed selectively in tissue granulomas and foam cellsMacrophage PPARγ and impaired wound healing in type 2 diabetesM2 Macrophage Polarization Mediates Anti-inflammatory Effects of Endothelial Nitric Oxide Signaling.Activation of natural killer T cells promotes M2 Macrophage polarization in adipose tissue and improves systemic glucose tolerance via interleukin-4 (IL-4)/STAT6 protein signaling axis in obesity.Modulation of macrophage efferocytosis in inflammation.miR-142-5p and miR-130a-3p are regulated by IL-4 and IL-13 and control profibrogenic macrophage program.Adipose tissue signaling by nuclear receptors in metabolic complications of obesityMacrophage PPARγ, a Lipid Activated Transcription Factor Controls the Growth Factor GDF3 and Skeletal Muscle Regeneration.Negative Immune Regulator TIPE2 Promotes M2 Macrophage Differentiation through the Activation of PI3K-AKT Signaling PathwayHigh salt reduces the activation of IL-4- and IL-13-stimulated macrophages.Tuberculosis is associated with expansion of a motile, permissive and immunomodulatory CD16(+) monocyte population via the IL-10/STAT3 axis.JAK and STAT signaling molecules in immunoregulation and immune-mediated disease.Systemic analysis of PPARγ in mouse macrophage populations reveals marked diversity in expression with critical roles in resolution of inflammation and airway immunitySelenoprotein Expression in Macrophages Is Critical for Optimal Clearance of Parasitic Helminth Nippostrongylus brasiliensis.Nitro-oleic acid modulates classical and regulatory activation of macrophages and their involvement in pro-fibrotic responses.
P2860
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P2860
STAT6 transcription factor is a facilitator of the nuclear receptor PPARγ-regulated gene expression in macrophages and dendritic cells
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
STAT6 transcription factor is ...... acrophages and dendritic cells
@ast
STAT6 transcription factor is ...... acrophages and dendritic cells
@en
STAT6 transcription factor is ...... acrophages and dendritic cells
@nl
type
label
STAT6 transcription factor is ...... acrophages and dendritic cells
@ast
STAT6 transcription factor is ...... acrophages and dendritic cells
@en
STAT6 transcription factor is ...... acrophages and dendritic cells
@nl
prefLabel
STAT6 transcription factor is ...... acrophages and dendritic cells
@ast
STAT6 transcription factor is ...... acrophages and dendritic cells
@en
STAT6 transcription factor is ...... acrophages and dendritic cells
@nl
P2093
P2860
P50
P1433
P1476
STAT6 transcription factor is ...... acrophages and dendritic cells
@en
P2093
Attila Pap
Attila Szanto
Balazs Dezso
Melinda Oros
Szilard Poliska
Yaacov Barak
Zsuzsanna S Nagy
P2860
P304
P356
10.1016/J.IMMUNI.2010.11.009
P407
P577
2010-11-01T00:00:00Z