Mycobacterium tuberculosis triggers host type I IFN signaling to regulate IL-1β production in human macrophages - sprookjes - yvandenbroek - TriplyDB

Mycobacterium tuberculosis triggers host type I IFN signaling to regulate IL-1β production in human macrophages

Mycobacterium tuberculosis triggers host type I IFN signaling to regulate IL-1β production in human macrophages

http://www.wikidata.org/entity/Q35175767

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Prognostic and Predictive Value of DAMPs and DAMP-Associated Processes in Cancer Duality of lipid mediators in host response against Mycobacterium tuberculosis: good cop, bad cop Mycobacterium tuberculosis and the host cell inflammasome: a complex relationship Differential effects of type I and II interferons on myeloid cells and resistance to intracellular bacterial infections Versatile myeloid cell subsets contribute to tuberculosis-associated inflammation Immunometabolism in Tuberculosis Reverse translation in tuberculosis: neutrophils provide clues for understanding development of active disease Granulomas and Inflammation: Host-Directed Therapies for Tuberculosis.Disease-promoting effects of type I interferons in viral, bacterial, and coinfections Sensing of Mycobacterium tuberculosis and consequences to both host and bacillus Allele-specific induction of IL-1β expression by C/EBPβ and PU.1 contributes to increased tuberculosis susceptibility Cutting edge: Mycobacterium tuberculosis but not nonvirulent mycobacteria inhibits IFN-β and AIM2 inflammasome-dependent IL-1β production via its ESX-1 secretion system Interferon alpha treatment of patients with impaired interferon gamma signaling.Type I interferons in bacterial infections: taming of myeloid cells and possible implications for autoimmunity.Type I IFN induces IL-10 production in an IL-27-independent manner and blocks responsiveness to IFN-γ for production of IL-12 and bacterial killing in Mycobacterium tuberculosis-infected macrophages.Insights into battles between Mycobacterium tuberculosis and macrophages.Mycobacterium tuberculosis activates the DNA-dependent cytosolic surveillance pathway within macrophages.Autophagy in tuberculosis Comparative functional genomics and the bovine macrophage response to strains of the mycobacterium genus.Interleukin-1 and interferon-γ orchestrate β-glucan-activated human dendritic cell programming via IκB-ζ modulation.Transcriptional blood signatures distinguish pulmonary tuberculosis, pulmonary sarcoidosis, pneumonias and lung cancers Defining the range of pathogens susceptible to Ifitm3 restriction using a knockout mouse model Cytokine and lipid mediator networks in tuberculosis Soluble amyloid triggers a myeloid differentiation factor 88 and interferon regulatory factor 7 dependent neuronal type-1 interferon response in vitro Inflammasome activation of IL-18 results in endothelial progenitor cell dysfunction in systemic lupus erythematosus.Innate and adaptive interferons suppress IL-1α and IL-1β production by distinct pulmonary myeloid subsets during Mycobacterium tuberculosis infection.The Cytosolic Sensor cGAS Detects Mycobacterium tuberculosis DNA to Induce Type I Interferons and Activate Autophagy ESX-1 exploits type I IFN-signalling to promote a regulatory macrophage phenotype refractory to IFNγ-mediated autophagy and growth restriction of intracellular mycobacteria.Dynamic roles of type I and type II IFNs in early infection with Mycobacterium tuberculosis.IL-27 Suppresses Antimicrobial Activity in Human Leprosy.Mucosal vaccination with attenuated Mycobacterium tuberculosis induces strong central memory responses and protects against tuberculosis Systematic, multiparametric analysis of Mycobacterium tuberculosis intracellular infection offers insight into coordinated virulence Activation of inflammasome signaling mediates pathology of acute P. aeruginosa pneumonia.Functional genomics identifies type I interferon pathway as central for host defense against Candida albicans Type I interferon suppresses type II interferon-triggered human anti-mycobacterial responses.Anti-dsDNA antibodies bind to TLR4 and activate NLRP3 inflammasome in lupus monocytes/macrophages.Type I Interferon Counters or Promotes Coxiella burnetii Replication Dependent on Tissue.Pathways of IL-1β secretion by macrophages infected with clinical Mycobacterium tuberculosis strains.Role of type I interferons in inflammasome activation, cell death, and disease during microbial infection.Low Dose BCG Infection as a Model for Macrophage Activation Maintaining Cell Viability.

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P2860

Mycobacterium tuberculosis triggers host type I IFN signaling to regulate IL-1β production in human macrophages

http://www.wikidata.org/entity/Q35175767

description

2011 nî lūn-bûn

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2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2011 թվականի հուլիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Mycobacterium tuberculosis tri ...... roduction in human macrophages

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Mycobacterium tuberculosis tri ...... roduction in human macrophages

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Mycobacterium tuberculosis tri ...... roduction in human macrophages

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Mycobacterium tuberculosis tri ...... roduction in human macrophages

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JIMMUNOL.1100926

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Journal of Immunology

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Mycobacterium tuberculosis tri ...... roduction in human macrophages

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Alan Sher

http://www.w3.org/2001/XMLSchema#string

Aleksey Novikov

http://www.w3.org/2001/XMLSchema#string

Carl G Feng

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Giorgio Trinchieri

http://www.w3.org/2001/XMLSchema#string

Katrin D Mayer-Barber

http://www.w3.org/2001/XMLSchema#string

Kevin D Kirschman

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Kevin Shenderov

http://www.w3.org/2001/XMLSchema#string

Marco Cardone

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Robert Thompson

http://www.w3.org/2001/XMLSchema#string

Ronald L Rabin

http://www.w3.org/2001/XMLSchema#string

P2860

Host-detrimental role of Esx-1-mediated inflammasome activation in mycobacterial infection

Type I interferon: friend or foe?

Direct multiplexed measurement of gene expression with color-coded probe pairs

An interferon-inducible neutrophil-driven blood transcriptional signature in human tuberculosis

NOD2, RIP2 and IRF5 play a critical role in the type I interferon response to Mycobacterium tuberculosis.

IL-1beta processing in host defense: beyond the inflammasomes.

Intranasal Poly-IC treatment exacerbates tuberculosis in mice through the pulmonary recruitment of a pathogen-permissive monocyte/macrophage population.

Type I IFN signaling constrains IL-17A/F secretion by gammadelta T cells during bacterial infections.

Regulation of antigen presentation by Mycobacterium tuberculosis: a role for Toll-like receptors

Suppression of TNF-α and IL-1 signaling identifies a mechanism of homeostatic regulation of macrophages by IL-27

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2540-2547

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10.4049/JIMMUNOL.1100926

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5

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187

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51517280

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21784976

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P921

Mycobacterium tuberculosis

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3159798

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