NOD2, RIP2 and IRF5 play a critical role in the type I interferon response to Mycobacterium tuberculosis.
about
Mycobacteria in Crohn’s disease: how innate immune deficiency may result in chronic inflammationActivation of nucleotide oligomerization domain 2 (NOD2) by human cytomegalovirus initiates innate immune responses and restricts virus replicationNOD2 controls the nature of the inflammatory response and subsequent fate of Mycobacterium tuberculosis and M. bovis BCG in human macrophagesActivation of interferon regulatory factor 5 by site specific phosphorylationCrosstalk between Mycobacterium tuberculosis and the host cellInduction of IFN-β and the Innate Antiviral Response in Myeloid Cells Occurs through an IPS-1-Dependent Signal That Does Not Require IRF-3 and IRF-7Type I interferon: friend or foe?Interaction of a traditional Chinese Medicine (PHY906) and CPT-11 on the inflammatory process in the tumor microenvironmentCentral roles of NLRs and inflammasomes in viral infectionMuramyl dipeptide induces NOD2-dependent Ly6C(high) monocyte recruitment to the lungs and protects against influenza virus infectionEspA acts as a critical mediator of ESX1-dependent virulence in Mycobacterium tuberculosis by affecting bacterial cell wall integrityMeta-analysis identifies 29 additional ulcerative colitis risk loci, increasing the number of confirmed associations to 47Analysis of Gene Expression in an Inbred Line of Soft-Shell Clams (Mya arenaria) Displaying Growth Heterosis: Regulation of Structural Genes and the NOD2 PathwaySynergistic stimulation of type I interferons during influenza virus coinfection promotes Streptococcus pneumoniae colonization in mice.Phagosomal signaling by Borrelia burgdorferi in human monocytes involves Toll-like receptor (TLR) 2 and TLR8 cooperativity and TLR8-mediated induction of IFN-betaInfergen Stimulated Macrophages Restrict Mycobacterium tuberculosis Growth by Autophagy and Release of Nitric Oxide.The Central Role of IFI204 in IFN-β Release and Autophagy Activation during Mycobacterium bovis Infection.Getting "Inside" Type I IFNs: Type I IFNs in Intracellular Bacterial InfectionsCritical role of IRF-5 in the development of T helper 1 responses to Leishmania donovani infection.IFN regulatory factor 5 is required for disease development in the FcgammaRIIB-/-Yaa and FcgammaRIIB-/- mouse models of systemic lupus erythematosusDeletion of the cyclic di-AMP phosphodiesterase gene (cnpB) in Mycobacterium tuberculosis leads to reduced virulence in a mouse model of infection.Type I interferon production induced by Streptococcus pyogenes-derived nucleic acids is required for host protectionRegulation of antigen presentation by Mycobacterium tuberculosis: a role for Toll-like receptorsMycobacterium tuberculosis activates the DNA-dependent cytosolic surveillance pathway within macrophages.Gene expression during the generation and activation of mouse neutrophils: implication of novel functional and regulatory pathways.Mycobacterium tuberculosis and TLR2 agonists inhibit induction of type I IFN and class I MHC antigen cross processing by TLR9Beyond macrophages: the diversity of mononuclear cells in tuberculosis.Autophagy in tuberculosisBacterial programming of host responses: coordination between type I interferon and cell death.Leprosy and the human genomeModifications to the peptidoglycan backbone help bacteria to establish infectionTranscriptional blood signatures distinguish pulmonary tuberculosis, pulmonary sarcoidosis, pneumonias and lung cancersStreptococcus pneumoniae DNA initiates type I interferon signaling in the respiratory tractMycobacterium tuberculosis type VII secreted effector EsxH targets host ESCRT to impair traffickingIRF5 deficiency ameliorates lupus but promotes atherosclerosis and metabolic dysfunction in a mouse model of lupus-associated atherosclerosisInduction of type I interferon signaling determines the relative pathogenicity of Staphylococcus aureus strains.Cytokine and lipid mediator networks in tuberculosisMycobacterium tuberculosis triggers host type I IFN signaling to regulate IL-1β production in human macrophagesA bacterial cyclic dinucleotide activates the cytosolic surveillance pathway and mediates innate resistance to tuberculosis.Disruption of an M. tuberculosis membrane protein causes a magnesium-dependent cell division defect and failure to persist in mice
P2860
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P2860
NOD2, RIP2 and IRF5 play a critical role in the type I interferon response to Mycobacterium tuberculosis.
description
2009 nî lūn-bûn
@nan
2009 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
NOD2, RIP2 and IRF5 play a cri ...... to Mycobacterium tuberculosis.
@ast
NOD2, RIP2 and IRF5 play a cri ...... to Mycobacterium tuberculosis.
@en
type
label
NOD2, RIP2 and IRF5 play a cri ...... to Mycobacterium tuberculosis.
@ast
NOD2, RIP2 and IRF5 play a cri ...... to Mycobacterium tuberculosis.
@en
prefLabel
NOD2, RIP2 and IRF5 play a cri ...... to Mycobacterium tuberculosis.
@ast
NOD2, RIP2 and IRF5 play a cri ...... to Mycobacterium tuberculosis.
@en
P2093
P2860
P1433
P1476
NOD2, RIP2 and IRF5 play a cri ...... to Mycobacterium tuberculosis.
@en
P2093
Amit K Pandey
Christopher M Sassetti
Francois Coulombe
Katherine A Fitzgerald
Marcel A Behr
Michelle A Kelliher
Sarah M Fortune
Yibin Yang
Zhaozhao Jiang
P2860
P304
P356
10.1371/JOURNAL.PPAT.1000500
P577
2009-07-03T00:00:00Z