The maternal-age-associated risk of congenital heart disease is modifiable. - sprookjes - yvandenbroek - TriplyDB

The maternal-age-associated risk of congenital heart disease is modifiable.

The maternal-age-associated risk of congenital heart disease is modifiable.

http://www.wikidata.org/entity/Q35365452

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Obstetric and offspring risks of women's morbid conditions linked to prior anticancer treatments A counterview of 'An investigation of the false discovery rate and the misinterpretation of p-values' by Colquhoun (2014)De Novo and Rare Variants at Multiple Loci Support the Oligogenic Origins of Atrioventricular Septal Heart Defects.Prolonged Cre expression driven by the α-myosin heavy chain promoter can be cardiotoxic.The Hole and the Whole: Lessons from Manipulation of Nipbl Deficiency Mutation of a common amino acid in NKX2.5 results in dilated cardiomyopathy in two large families.The impact of environmental factors on the occurrence of congenital heart disease in the form of hypoplastic left heart syndrome.Association Between DSCR1 Variations and Congenital Heart Disease Susceptibility.Pregnancy at Advanced Maternal Age Affects Behavior and Hippocampal Gene Expression in Mouse Offspring.The effects of voluntary exercise on oocyte quality in a diet-induced obese murine model.Genetics of congenital heart disease Advanced maternal age causes adverse programming of mouse blastocysts leading to altered growth and impaired cardiometabolic health in post-natal life.Maternal Midpregnancy Glucose Levels and Risk of Congenital Heart Disease in Offspring.Maternal exercise upregulates mitochondrial gene expression and increases enzyme activity of fetal mouse hearts.Exercise and epigenetic inheritance of disease risk.The Complex Genetic Basis of Congenital Heart Defects.Decidualisation and placentation defects are a major cause of age-related reproductive decline Trend analysis of mortality rates and causes of death in children under 5 years old in Beijing, China from 1992 to 2015 and forecast of mortality into the future: an entire population-based epidemiological study.Transgenerational cardiology: One way to a baby's heart is through the mother.Congenital heart defects in IVF/ICSI pregnancy: systematic review and meta-analysis.Distinct genetic architectures for syndromic and nonsyndromic congenital heart defects identified by exome sequencing.Race for healthy hearts

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P2860

The maternal-age-associated risk of congenital heart disease is modifiable.

http://www.wikidata.org/entity/Q35365452

description

2015 nî lūn-bûn

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2015 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2015 թվականի ապրիլին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

The maternal-age-associated risk of congenital heart disease is modifiable.

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The maternal-age-associated risk of congenital heart disease is modifiable.

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The maternal-age-associated risk of congenital heart disease is modifiable.

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The maternal-age-associated risk of congenital heart disease is modifiable.

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The maternal-age-associated risk of congenital heart disease is modifiable.

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The maternal-age-associated risk of congenital heart disease is modifiable.

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The maternal-age-associated risk of congenital heart disease is modifiable.

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Adam A Panzer

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Alayna K Hutchinson

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Claire E Schulkey

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Herman Luther

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Mary M Grady

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Megan T Danzo

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Patrick Y Jay

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Rachel A Magnan

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Suk D Regmi

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P2860

Mutations in the cardiac transcription factor NKX2.5 affect diverse cardiac developmental pathways

Congenital heart disease caused by mutations in the transcription factor NKX2-5

Prevention of neural tube defects: results of the Medical Research Council Vitamin Study. MRC Vitamin Study Research Group

Serum levels of choline-containing compounds are associated with aerobic fitness level: the HUNT-study

Whole blood transcriptomics and urinary metabolomics to define adaptive biochemical pathways of high-intensity exercise in 50-60 year old masters athletes

The cardiac homeobox gene Csx/Nkx2.5 lies genetically upstream of multiple genes essential for heart development

The epidemiology of cardiovascular defects, part I: a study based on data from three large registries of congenital malformations.

Detecting maternal-fetal genotype interactions associated with conotruncal heart defects: a haplotype-based analysis with penalized logistic regression.

Modifier genes in mice and humans.

Heterogeneity of genetic modifiers ensures normal cardiac development

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