about
Mouse GATA-4: a retinoic acid-inducible GATA-binding transcription factor expressed in endodermally derived tissues and heartToying with fate: Redirecting the differentiation of adrenocortical progenitor cells into gonadal-like tissueTranscription factor GATA-6 is expressed in malignant endoderm of pediatric yolk sac tumors and in teratomasDifferential expression of GATA-4 and GATA-6 in fetal and adult mouse and human adrenal tissueSafety, efficacy, and immune reconstitution after rituximab therapy in pediatric patients with chronic or refractory hematologic autoimmune cytopenias.Inherited bone marrow failure syndromes in adolescents and young adults.Nkx2-5 and Sarcospan genetically interact in the development of the muscular ventricular septum of the heartReview paper: origin and molecular pathology of adrenocortical neoplasmsValidation is a dish oft served coldHigh affinity esterification of eicosanoid precursor fatty acids by plateletsAssignment of the transcription factor GATA4 gene to human chromosome 8 and mouse chromosome 14: Gata4 is a candidate gene for Ds (disorganization).GATA-4, GATA-5, and GATA-6 activate the rat liver fatty acid binding protein gene in concert with HNF-1alpha.Fgf-8 expression in the post-gastrulation mouse suggests roles in the development of the face, limbs and central nervous system.Biosynthesis of coagulation Factor V by a human hepatocellular carcinoma cell line.Laser capture microdissection-reduced representation bisulfite sequencing (LCM-RRBS) maps changes in DNA methylation associated with gonadectomy-induced adrenocortical neoplasia in the mouse.Novel markers of gonadectomy-induced adrenocortical neoplasia in the mouse and ferretClinical outcomes of splenectomy in children: report of the splenectomy in congenital hemolytic anemia registryThe maternal-age-associated risk of congenital heart disease is modifiable.GATA4 is a key regulator of steroidogenesis and glycolysis in mouse Leydig cells.Animal models of adrenocortical tumorigenesisGATA4 is a critical regulator of gonadectomy-induced adrenocortical tumorigenesis in mice.Molecular genetics of congenital diaphragmatic defects.GATA factors in endocrine neoplasiaGonadectomy-induced adrenocortical neoplasia in the domestic ferret (Mustela putorius furo) and laboratory mouse.Role of the GATA family of transcription factors in endocrine development, function, and diseaseThe GATA-4 transcription factor transactivates the cardiac muscle-specific troponin C promoter-enhancer in nonmuscle cells.GATA transcription factors in adrenal development and tumors.Adrenocortical tumorigenesis, luteinizing hormone receptor and transcription factors GATA-4 and GATA-6.Conditional mutagenesis of Gata6 in SF1-positive cells causes gonadal-like differentiation in the adrenal cortex of mice.Cytochrome b5 expression in gonadectomy-induced adrenocortical neoplasms of the domestic ferret (Mustela putorius furo).TERC and TERT gene mutations in patients with bone marrow failure and the significance of telomere length measurements.Hematologic outcomes after total splenectomy and partial splenectomy for congenital hemolytic anemiaA family with Hoyeraal-Hreidarsson syndrome and four variants in two genes of the telomerase core complex.Inositol cyclic phosphates are produced by cleavage of phosphatidylphosphoinositols (polyphosphoinositides) with purified sheep seminal vesicle phospholipase C enzymes.Adrenocortical tumorigenesis in transgenic mice expressing the inhibin alpha-subunit promoter/simian virus 40 T-antigen transgene: relationship between ectopic expression of luteinizing hormone receptor and transcription factor GATA-4.Downregulation of transcription factor GATA4 sensitizes human hepatoblastoma cells to doxorubicin-induced apoptosis.Transcription factor GATA-4 is abundantly expressed in childhood but not in adult liver tumors.The metabolism of phosphoinositide-derived messenger molecules.Enhanced expression of transcription factor GATA-4 in inflammatory bowel disease and its possible regulation by TGF-beta1.Hodgkin disease after renal transplantation in childhood.
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David B. Wilson
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