Role of reactive nitrogen and oxygen intermediates in gamma interferon-stimulated murine macrophage bactericidal activity against Burkholderia pseudomallei
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Post-exposure therapeutic efficacy of COX-2 inhibition against Burkholderia pseudomalleiInactivation of [Fe-S] metalloproteins mediates nitric oxide-dependent killing of Burkholderia mallei.Phenotypic and functional characterization of human memory T cell responses to Burkholderia pseudomallei.Innate immune responses of pulmonary epithelial cells to Burkholderia pseudomallei infection.Murine macrophages use oxygen- and nitric oxide-dependent mechanisms to synthesize S-nitroso-albumin and to kill extracellular trypanosomes.Immunotherapy markedly increases the effectiveness of antimicrobial therapy for treatment of Burkholderia pseudomallei infectionObligatory role of gamma interferon for host survival in a murine model of infection with Burkholderia pseudomallei.Interaction of Bartonella henselae with the murine macrophage cell line J774: infection and proinflammatory response.Neutrophil elastase causes tissue damage that decreases host tolerance to lung infection with burkholderia species.Inflammasome-dependent pyroptosis and IL-18 protect against Burkholderia pseudomallei lung infection while IL-1β is deleteriousInteraction of Interferon gamma-induced reactive oxygen species with ceftazidime leads to synergistic killing of intracellular Burkholderia pseudomallei.Phagocyte NADPH oxidase, but not inducible nitric oxide synthase, is essential for early control of Burkholderia cepacia and chromobacterium violaceum infection in miceOropharyngeal aspiration of Burkholderia mallei and Burkholderia pseudomallei in BALB/c mice.Quorum sensing negatively regulates multinucleate cell formation during intracellular growth of Burkholderia pseudomallei in macrophage-like cellsCaspase-1-dependent and -independent cell death pathways in Burkholderia pseudomallei infection of macrophages.Combining vaccination and postexposure CpG therapy provides optimal protection against lethal sepsis in a biodefense model of human melioidosis.Role of inducible nitric oxide synthase and NADPH oxidase in early control of Burkholderia pseudomallei infection in mice.Cationic liposomes extend the immunostimulatory effect of CpG oligodeoxynucleotide against Burkholderia pseudomallei infection in BALB/c mice.The Bacterial Mfd Protein Prevents DNA Damage Induced by the Host Nitrogen Immune Response in a NER-Independent but RecBC-Dependent PathwayiNOS activity is critical for the clearance of Burkholderia mallei from infected RAW 264.7 murine macrophages.Immunostimulatory CpG oligodeoxynucleotide confers protection in a murine model of infection with Burkholderia pseudomallei.Human polymorphonuclear neutrophil responses to Burkholderia pseudomallei in healthy and diabetic subjects.Protection from pneumonic infection with burkholderia species by inhalational immunotherapyInvolvement of signal regulatory protein α, a negative regulator of Toll-like receptor signaling, in impairing the MyD88-independent pathway and intracellular killing of Burkholderia pseudomallei-infected mouse macrophages.Burkholderia pseudomallei-induced expression of suppressor of cytokine signaling 3 and cytokine-inducible src homology 2-containing protein in mouse macrophages: a possible mechanism for suppression of the response to gamma interferon stimulationDefense Mechanisms of Hepatocytes Against Burkholderia pseudomallei.The bacterial DNA repair protein Mfd confers resistance to the host nitrogen immune response.Involvement of beta interferon in enhancing inducible nitric oxide synthase production and antimicrobial activity of Burkholderia pseudomallei-infected macrophages.Induction of iNOS expression and antimicrobial activity by interferon (IFN)-beta is distinct from IFN-gamma in Burkholderia pseudomallei-infected mouse macrophages.Kinetic studies of the production of nitric oxide (NO) and tumour necrosis factor-alpha (TNF-alpha) in macrophages stimulated with Burkholderia pseudomallei endotoxin.Sodium nitrite-mediated killing of the major cystic fibrosis pathogens Pseudomonas aeruginosa, Staphylococcus aureus, and Burkholderia cepacia under anaerobic planktonic and biofilm conditions.Sterile-α- and armadillo motif-containing protein inhibits the TRIF-dependent downregulation of signal regulatory protein α to interfere with intracellular bacterial elimination in Burkholderia pseudomallei-infected mouse macrophages.Production of reactive oxygen species and expression of inducible nitric oxide synthase in rat isolated Kupffer cells stimulated by Leptospira interrogans and Borrelia burgdorferiProtection against heterologous Burkholderia pseudomallei strains by dendritic cell immunization.
P2860
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P2860
Role of reactive nitrogen and oxygen intermediates in gamma interferon-stimulated murine macrophage bactericidal activity against Burkholderia pseudomallei
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Role of reactive nitrogen and ...... inst Burkholderia pseudomallei
@ast
Role of reactive nitrogen and ...... inst Burkholderia pseudomallei
@en
type
label
Role of reactive nitrogen and ...... inst Burkholderia pseudomallei
@ast
Role of reactive nitrogen and ...... inst Burkholderia pseudomallei
@en
prefLabel
Role of reactive nitrogen and ...... inst Burkholderia pseudomallei
@ast
Role of reactive nitrogen and ...... inst Burkholderia pseudomallei
@en
P2093
P2860
P1476
Role of reactive nitrogen and ...... inst Burkholderia pseudomallei
@en
P2093
P2860
P304
P407
P577
1997-10-01T00:00:00Z