Functional genomics reveals an essential and specific role for Stat1 in protection of the central nervous system following herpes simplex virus corneal infection
about
Inborn errors of human STAT1: allelic heterogeneity governs the diversity of immunological and infectious phenotypesThe regulation of inflammation by interferons and their STATsHerpes simplex virus serotype and entry receptor availability alter CNS disease in a mouse model of neonatal HSVGlucocorticoid treatment of MCMV infected newborn mice attenuates CNS inflammation and limits deficits in cerebellar development.Genome-wide mouse mutagenesis reveals CD45-mediated T cell function as critical in protective immunity to HSV-1.Von Willebrand Factor Gene Variants Associate with Herpes simplex Encephalitis.Corneal replication is an interferon response-independent bottleneck for virulence of herpes simplex virus 1 in the absence of virion host shutoff.Induction of Multiple miR-200/182 Members in the Brains of Mice Are Associated with Acute Herpes Simplex Virus 1 Encephalitis.mRNA decay during herpes simplex virus (HSV) infections: mutations that affect translation of an mRNA influence the sites at which it is cleaved by the HSV virion host shutoff (Vhs) protein.The Type I Interferon Response Determines Differences in Choroid Plexus Susceptibility between Newborns and Adults in Herpes Simplex Virus Encephalitis.Immune- and Nonimmune-Compartment-Specific Interferon Responses Are Critical Determinants of Herpes Simplex Virus-Induced Generalized Infections and Acute Liver Failure.Cellular Protein WDR11 Interacts with Specific Herpes Simplex Virus Proteins at the trans-Golgi Network To Promote Virus Replication.Innate defense mechanisms against HSV-1 infection in the target tissues, skin and brain.PNKP knockdown by RNA interference inhibits herpes simplex virus-1 replication in astrocytes.Herpes simplex virus 1 counteracts tetherin restriction via its virion host shutoff activity.CNS InfectionsInsights into the pathogenesis of herpes simplex encephalitis from mouse models
P2860
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P2860
Functional genomics reveals an essential and specific role for Stat1 in protection of the central nervous system following herpes simplex virus corneal infection
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Functional genomics reveals an ...... implex virus corneal infection
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Functional genomics reveals an ...... implex virus corneal infection
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type
label
Functional genomics reveals an ...... implex virus corneal infection
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Functional genomics reveals an ...... implex virus corneal infection
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Functional genomics reveals an ...... implex virus corneal infection
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Functional genomics reveals an ...... implex virus corneal infection
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P2093
P2860
P356
P1433
P1476
Functional genomics reveals an ...... implex virus corneal infection
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P2093
Cristian Cilloniz
David A Leib
Pamela Rosato
Tracy Jo Pasieka
Victoria S Carter
P2860
P304
12972-12981
P356
10.1128/JVI.06032-11
P407
P577
2011-10-12T00:00:00Z