PNKP knockdown by RNA interference inhibits herpes simplex virus-1 replication in astrocytes.

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PNKP knockdown by RNA interference inhibits herpes simplex virus-1 replication in astrocytes.

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2013 nî lūn-bûn

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2013年の論文

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2013年論文

@yue

2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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2013年论文

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PNKP knockdown by RNA interfer ...... s-1 replication in astrocytes.

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PNKP knockdown by RNA interfer ...... s-1 replication in astrocytes.

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PNKP knockdown by RNA interfer ...... s-1 replication in astrocytes.

@en

PNKP knockdown by RNA interfer ...... s-1 replication in astrocytes.

@nl

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PNKP knockdown by RNA interfer ...... s-1 replication in astrocytes.

@en

PNKP knockdown by RNA interfer ...... s-1 replication in astrocytes.

@nl

P1476

PNKP knockdown by RNA interfer ...... s-1 replication in astrocytes.

@en

P2093

Le Sun

http://www.w3.org/2001/XMLSchema#string

Lei Yue

http://www.w3.org/2001/XMLSchema#string

Longding Liu

http://www.w3.org/2001/XMLSchema#string

Min Yan

http://www.w3.org/2001/XMLSchema#string

Qihan Li

http://www.w3.org/2001/XMLSchema#string

Sujie Guo

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Xia Cao

http://www.w3.org/2001/XMLSchema#string

Ying Zhang

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P2860

Molecular cloning of the human gene, PNKP, encoding a polynucleotide kinase 3'-phosphatase and evidence for its role in repair of DNA strand breaks caused by oxidative damage

Interferon lambda inhibits herpes simplex virus type I infection of human astrocytes and neurons

Herpes simplex infections of the nervous system.

Replication of herpes simplex virus DNA.

Tidying up loose ends: the role of polynucleotide kinase/phosphatase in DNA strand break repair.

Herpes simplex virus encephalitis in a patient with complete TLR3 deficiency: TLR3 is otherwise redundant in protective immunity.

Functional genomics reveals an essential and specific role for Stat1 in protection of the central nervous system following herpes simplex virus corneal infection

TLR3 deficiency renders astrocytes permissive to herpes simplex virus infection and facilitates establishment of CNS infection in mice

The role of chemokines during herpes simplex virus-1 infection.

Oligomerization of ICP4 and rearrangement of heat shock proteins may be important for herpes simplex virus type 1 prereplicative site formation.

P2888

s12250-013-3350-5

P304

345-351

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P31

scholarly article

P356

10.1007/S12250-013-3350-5

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P433

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P478

http://www.w3.org/2001/XMLSchema#string

P577

2013-11-06T00:00:00Z

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P698

24213989

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P921

astrocyte