The role of nuclear lamin B1 in cell proliferation and senescence. - sprookjes - yvandenbroek - TriplyDB

The role of nuclear lamin B1 in cell proliferation and senescence.

The role of nuclear lamin B1 in cell proliferation and senescence.

http://www.wikidata.org/entity/Q35642054

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Old cells, new tricks: chromatin structure in senescence Breaching the nuclear envelope in development and disease Aging and longevity in the simplest animals and the quest for immortality Nuclear lamins and oxidative stress in cell proliferation and longevity Cell-based screen for altered nuclear phenotypes reveals senescence progression in polyploid cells after Aurora kinase B inhibition.Physiological and Pathological Aging Affects Chromatin Dynamics, Structure and Function at the Nuclear Edge Subcellular fractionation and localization studies reveal a direct interaction of the fragile X mental retardation protein (FMRP) with nucleolin The hallmarks of aging Autophagy mediates degradation of nuclear lamina Defects of Lipid Synthesis Are Linked to the Age-Dependent Demyelination Caused by Lamin B1 Overexpression Temsirolimus Partially Rescues the Hutchinson-Gilford Progeria Cellular Phenotype Lamin b1 polymorphism influences morphology of the nuclear envelope, cell cycle progression, and risk of neural tube defects in mice Differential Predictive Roles of A- and B-Type Nuclear Lamins in Prostate Cancer Progression Lysosome-mediated processing of chromatin in senescence.Higher-order unfolding of satellite heterochromatin is a consistent and early event in cell senescence Expression of progerin in aging mouse brains reveals structural nuclear abnormalities without detectible significant alterations in gene expression, hippocampal stem cells or behavior.Lamin-like analogues in plants: the characterization of NMCP1 in Allium cepa Interphase phosphorylation of lamin A.A comparison of oncogene-induced senescence and replicative senescence: implications for tumor suppression and aging.Lamin B1 levels modulate differentiation into neurons during embryonic corticogenesis Loss of lamin B1 results in prolongation of S phase and decondensation of chromosome territories The role of senescent cells in ageing.Lamin B1 overexpression increases nuclear rigidity in autosomal dominant leukodystrophy fibroblasts.Aging, cellular senescence, and cancer Three-dimensional genome architecture: players and mechanisms.Progerin, the protein responsible for the Hutchinson-Gilford progeria syndrome, increases the unrepaired DNA damages following exposure to ionizing radiation.Phytochemical-induced nucleolar stress results in the inhibition of breast cancer cell proliferation.Age-associated loss of lamin-B leads to systemic inflammation and gut hyperplasia.Long noncoding RNA MALAT1 controls cell cycle progression by regulating the expression of oncogenic transcription factor B-MYB.Changes in the proteomic profiles of mouse brain after infection with cyst-forming Toxoplasma gondii.Are there roles for brain cell senescence in aging and neurodegenerative disorders?Subcellular distribution and activity of mechanistic target of rapamycin in aged retinal pigment epithelium.Role of lamin b1 in chromatin instability Sulforaphane enhances progerin clearance in Hutchinson-Gilford progeria fibroblasts.Embryonic expression of the common progeroid lamin A splice mutation arrests postnatal skin development Replicative senescence is associated with nuclear reorganization and with DNA methylation at specific transcription factor binding sites Overexpression of Lamin B Receptor Results in Impaired Skin Differentiation.Altered Protein Expression in the Ileum of Mice Associated with the Development of Chronic Infections with Echinostoma caproni (Trematoda).Blood and immune cell engineering: Cytoskeletal contractility and nuclear rheology impact cell lineage and localization: Biophysical regulation of hematopoietic differentiation and trafficking.Lamin B1 loss is a senescence-associated biomarker.

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The role of nuclear lamin B1 in cell proliferation and senescence.

http://www.wikidata.org/entity/Q35642054

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

The role of nuclear lamin B1 in cell proliferation and senescence.

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The role of nuclear lamin B1 in cell proliferation and senescence.

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type

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The role of nuclear lamin B1 in cell proliferation and senescence.

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The role of nuclear lamin B1 in cell proliferation and senescence.

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The role of nuclear lamin B1 in cell proliferation and senescence.

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The role of nuclear lamin B1 in cell proliferation and senescence.

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Genes & Development

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The role of nuclear lamin B1 in cell proliferation and senescence

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Anne E Goldman

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Catherine A Lucas

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Dale K Shumaker

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Navdeep S Chandel

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Robert B Hamanaka

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Robert D Goldman

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Takeshi Shimi

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Veronika Butin-Israeli

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P2860

The mutant form of lamin A that causes Hutchinson-Gilford progeria is a biomarker of cellular aging in human skin

Alteration of nuclear lamin organization inhibits RNA polymerase II-dependent transcription

Nuclear lamins A and B1: different pathways of assembly during nuclear envelope formation in living cells

A novel role for high-mobility group a proteins in cellular senescence and heterochromatin formation

Feedback between p21 and reactive oxygen production is necessary for cell senescence

Prelamin A acts to accelerate smooth muscle cell senescence and is a novel biomarker of human vascular aging

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

Reversal of the cellular phenotype in the premature aging disease Hutchinson-Gilford progeria syndrome

Hutchinson-Gilford progeria mutant lamin A primarily targets human vascular cells as detected by an anti-Lamin A G608G antibody.

Regulation of late G1/S phase transition and APC Cdh1 by reactive oxygen species

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2579-2593

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scholarly article

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10.1101/GAD.179515.111

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24

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25

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Steven T. Kosak

Stephen A. Adam

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2011-12-08T00:00:00Z

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51866124

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22155925

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3248680

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