Colony-stimulating factor-1 mediates macrophage-related neural damage in a model for Charcot-Marie-Tooth disease type 1X
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Neuroinflammation in the peripheral nerve: Cause, modulator, or bystander in peripheral neuropathies?Rare Variants in MME, Encoding Metalloprotease Neprilysin, Are Linked to Late-Onset Autosomal-Dominant Axonal PolyneuropathiesEndogenous antibodies contribute to macrophage-mediated demyelination in a mouse model for CMT1B.Cell-Surface and Secreted Isoforms of CSF-1 Exert Opposing Roles in Macrophage-Mediated Neural Damage in Cx32-Deficient Mice.Emerging Roles for CSF-1 Receptor and its Ligands in the Nervous System.Murine therapeutic models for Charcot-Marie-Tooth (CMT) disease.Animal models and therapeutic prospects for Charcot-Marie-Tooth disease.Molecules involved in the crosstalk between immune- and peripheral nerve Schwann cells.Wallerian degeneration: an emerging axon death pathway linking injury and disease.Neuroinflammation as modifier of genetically caused neurological disorders of the central nervous system: Understanding pathogenesis and chances for treatment.Towards a functional pathology of hereditary neuropathies.CSF1R mutations in hereditary diffuse leukoencephalopathy with spheroids are loss of function.Proteolipid protein modulates preservation of peripheral axons and premature death when myelin protein zero is lacking.Targeting the colony stimulating factor 1 receptor alleviates two forms of Charcot-Marie-Tooth disease in mice.Characterization of Endoneurial Fibroblast-like Cells from Human and Rat Peripheral Nerves.Coexistence of Charcot-Marie-Tooth disease type 1A and anti-MAG neuropathy.LITAF (SIMPLE) regulates Wallerian degeneration after injury but is not essential for peripheral nerve development and maintenance: implications for Charcot-Marie-Tooth disease.Characterization of New Transgenic Mouse Models for Two Charcot-Marie-Tooth-Causing HspB1 Mutations using the Rosa26 Locus.CSF-1-activated macrophages are target-directed and essential mediators of Schwann cell dedifferentiation and dysfunction in Cx32-deficient mice.Nonuniform molecular features of myelinating Schwann cells in models for CMT1: distinct disease patterns are associated with NCAM and c-Jun upregulation.Blocking bad.What's the Function of Connexin 32 in the Peripheral Nervous System?
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Colony-stimulating factor-1 mediates macrophage-related neural damage in a model for Charcot-Marie-Tooth disease type 1X
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
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2011年论文
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name
Colony-stimulating factor-1 me ...... ot-Marie-Tooth disease type 1X
@ast
Colony-stimulating factor-1 me ...... ot-Marie-Tooth disease type 1X
@en
type
label
Colony-stimulating factor-1 me ...... ot-Marie-Tooth disease type 1X
@ast
Colony-stimulating factor-1 me ...... ot-Marie-Tooth disease type 1X
@en
prefLabel
Colony-stimulating factor-1 me ...... ot-Marie-Tooth disease type 1X
@ast
Colony-stimulating factor-1 me ...... ot-Marie-Tooth disease type 1X
@en
P2093
P2860
P356
P1433
P1476
Colony-stimulating factor-1 me ...... ot-Marie-Tooth disease type 1X
@en
P2093
E Richard Stanley
Hanna Zieger
Heike Heuer
Janos Groh
Joachim Weis
Rudolf Martini
P2860
P304
P356
10.1093/BRAIN/AWR283
P407
P577
2011-11-16T00:00:00Z