Mitochondrial reactive oxygen species trigger hypoxia-inducible factor-dependent extension of the replicative life span during hypoxia - sprookjes - yvandenbroek - TriplyDB

Mitochondrial reactive oxygen species trigger hypoxia-inducible factor-dependent extension of the replicative life span during hypoxia

Mitochondrial reactive oxygen species trigger hypoxia-inducible factor-dependent extension of the replicative life span during hypoxia

http://www.wikidata.org/entity/Q35950224

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MicroRNA-210 regulates mitochondrial free radical response to hypoxia and krebs cycle in cancer cells by targeting iron sulfur cluster protein ISCU Conjoint propagation and differentiation of human embryonic stem cells to cardiomyocytes in a defined microcarrier spinner culture The Tumorigenic Roles of the Cellular REDOX Regulatory Systems Nuclear lamins and oxidative stress in cell proliferation and longevity mTOR activation is a biomarker and a central pathway to autoimmune disorders, cancer, obesity, and aging Declining NAD(+) induces a pseudohypoxic state disrupting nuclear-mitochondrial communication during aging The "two-faced" effects of reactive oxygen species and the lipid peroxidation product 4-hydroxynonenal in the hallmarks of cancer Nrf2 is the key to chemotherapy resistance in MCF7 breast cancer cells under hypoxia ROS function in redox signaling and oxidative stress Signal transduction by reactive oxygen species Physiological roles of mitochondrial reactive oxygen species Strain-dependent oxidant release in articular cartilage originates from mitochondria Kinome siRNA screen identifies SMG-1 as a negative regulator of hypoxia-inducible factor-1alpha in hypoxia Genome-wide fitness and expression profiling implicate Mga2 in adaptation to hydrogen peroxide.The HIF-1 hypoxia-inducible factor modulates lifespan in C. elegans.Bistability of mitochondrial respiration underlies paradoxical reactive oxygen species generation induced by anoxia Neuroprotective effect of peroxiredoxin 6 against hypoxia-induced retinal ganglion cell damage.Leonurine Improves Age-Dependent Impaired Angiogenesis: Possible Involvement of Mitochondrial Function and HIF-1α Dependent VEGF Activation.Endothelin-1 induces a glycolytic switch in pulmonary arterial endothelial cells via the mitochondrial translocation of endothelial nitric oxide synthase.Macrophage migration inhibitory factor-knockout mice are long lived and respond to caloric restriction.Spatiotemporal intracellular nitric oxide signaling captured using internalized, near-infrared fluorescent carbon nanotube nanosensors.Mitochondrial reactive oxygen species regulate cellular signaling and dictate biological outcomes.Mitochondrial localization of telomeric protein TIN2 links telomere regulation to metabolic control Controlled DNA "damage" and repair in hypoxic signaling Regulation of life span by mitochondrial respiration: the HIF-1 and ROS connection.SirT3 suppresses hypoxia inducible factor 1α and tumor growth by inhibiting mitochondrial ROS production.Gd-metallofullerenol nanomaterial as non-toxic breast cancer stem cell-specific inhibitor Hypoxia leads to Na,K-ATPase downregulation via Ca(2+) release-activated Ca(2+) channels and AMPK activation The role of hypoxia inducible factor-1 alpha in bypassing oncogene-induced senescence Two sides to every story: the HIF-dependent and HIF-independent functions of pVHL.The role of nuclear lamin B1 in cell proliferation and senescence.Carbon source metabolism and its regulation in cancer cells.Hormetic effect of rotenone in primary human fibroblasts.Glucotoxicity induces glucose-6-phosphatase catalytic unit expression by acting on the interaction of HIF-1α with CREB-binding protein The twisted survivin connection to angiogenesis Oxygen versus Reactive Oxygen in the Regulation of HIF-1α: The Balance Tips O2 sensing, mitochondria and ROS signaling: The fog is lifting Exogenous low dose hydrogen peroxide increases hypoxia-inducible factor-1alpha protein expression and induces preconditioning protection against ischemia in primary cortical neurons.Hyperoxia-induced premature senescence requires p53 and pRb, but not mitochondrial matrix ROS.A role of mitochondrial complex II defects in genetic models of Huntington's disease expressing N-terminal fragments of mutant huntingtin

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P2860

Mitochondrial reactive oxygen species trigger hypoxia-inducible factor-dependent extension of the replicative life span during hypoxia

http://www.wikidata.org/entity/Q35950224

description

2007 nî lūn-bûn

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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2007年论文

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2007年论文

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name

Mitochondrial reactive oxygen ...... ative life span during hypoxia

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Mitochondrial reactive oxygen ...... ative life span during hypoxia

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type

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Mitochondrial reactive oxygen ...... ative life span during hypoxia

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Mitochondrial reactive oxygen ...... ative life span during hypoxia

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Mitochondrial reactive oxygen ...... ative life span during hypoxia

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Mitochondrial reactive oxygen ...... ative life span during hypoxia

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Molecular and Cellular Biology

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Mitochondrial reactive oxygen ...... ative life span during hypoxia

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Eric L Bell

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James Eisenbart

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Navdeep S Chandel

http://www.w3.org/2001/XMLSchema#string

Paul T Schumacker

http://www.w3.org/2001/XMLSchema#string

Tatyana A Klimova

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P2860

Hypoxia extends the life span of vascular smooth muscle cells through telomerase activation.

Activation of vascular endothelial growth factor gene transcription by hypoxia-inducible factor 1

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

Extension of life-span by introduction of telomerase into normal human cells

A biomarker that identifies senescent human cells in culture and in aging skin in vivo

Crucial role of p53-dependent cellular senescence in suppression of Pten-deficient tumorigenesis

Targeting HIF-1 for cancer therapy

A new mathematical model for relative quantification in real-time RT-PCR

HIF-1 and human disease: one highly involved factor

A critical role for histone H2AX in recruitment of repair factors to nuclear foci after DNA damage

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5737-5745

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10.1128/MCB.02265-06

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16

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27

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6273885

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17562866

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1952129

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