Identification of the CD4(+) T cell as a major pathogenic factor in ischemic acute renal failure. - sprookjes - yvandenbroek - TriplyDB

Identification of the CD4(+) T cell as a major pathogenic factor in ischemic acute renal failure.

Identification of the CD4(+) T cell as a major pathogenic factor in ischemic acute renal failure.

http://www.wikidata.org/entity/Q36169286

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Guidance cue netrin-1 and the regulation of inflammation in acute and chronic kidney disease Ischemia and reperfusion injury in renal transplantation: hemodynamic and immunological paradigms Mechanisms of Cisplatin nephrotoxicity Proximal tubule-derived colony stimulating factor-1 mediates polarization of renal macrophages and dendritic cells, and recovery in acute kidney injury Crosstalk between the unfolded protein response and NF-κB-mediated inflammation in the progression of chronic kidney disease CSF-1 signaling mediates recovery from acute kidney injury.Therapeutic Targets of Human AKI: Harmonizing Human and Animal AKI Sphingosine 1-Phosphate Receptor 3-Deficient Dendritic Cells Modulate Splenic Responses to Ischemia-Reperfusion Injury.Both PD-1 ligands protect the kidney from ischemia reperfusion injury.Renal protection from ischemia mediated by A2A adenosine receptors on bone marrow-derived cells.Expanding role of T cells in acute kidney injury.Role of leukocytes in the pathogenesis of acute kidney injury Dendritic cell sphingosine 1-phosphate receptor-3 regulates Th1-Th2 polarity in kidney ischemia-reperfusion injury.Pathogenesis of acute kidney injury: foundation for clinical practice.Activation of sphingosine-1-phosphate 1 receptor in the proximal tubule protects against ischemia-reperfusion injury.Regulatory T cells contribute to the protective effect of ischemic preconditioning in the kidney.Immune mechanisms and novel pharmacological therapies of acute kidney injury.Regulatory T cells suppress innate immunity in kidney ischemia-reperfusion injury Targeting sphingosine 1 phosphate receptor type 1 receptors in acute kidney injury.Sphingosine-1-phosphate receptors: biology and therapeutic potential in kidney disease Use of ultrasound to assess renal reperfusion and P-selectin expression following unilateral renal ischemia Hydrodynamic Isotonic Fluid Delivery Ameliorates Moderate-to-Severe Ischemia-Reperfusion Injury in Rat Kidneys.CD4 T cells promote tissue inflammation via CD40 signaling without de novo activation in a murine model of liver ischemia/reperfusion injury.B cells limit repair after ischemic acute kidney injury.T cells in organ ischemia reperfusion injury.Kidney-lung crosstalk in the critically ill patient.Immunosuppressive effects of erythropoietin on human alloreactive T cells Near-term anti-CD25 monoclonal antibody administration protects murine liver from ischemia-reperfusion injury due to reduced numbers of CD4+ T cells Dendritic cells facilitate accumulation of IL-17 T cells in the kidney following acute renal obstruction.Macrophages expressing heme oxygenase-1 improve renal function in ischemia/reperfusion injury.Lymphocyte modulation with FTY720 improves hemorrhagic shock survival in swine Renal ischaemia reperfusion injury: a mouse model of injury and regeneration.Ischemic preconditioning in the liver is independent of regulatory T cell activity.Role of adhesion molecules and dendritic cells in rat hepatic/renal ischemia-reperfusion injury and anti-adhesive intervention with anti-P-selectin lectin-EGF domain monoclonal antibody.The TIM-1:TIM-4 pathway enhances renal ischemia-reperfusion injury Surgical sepsis and organ crosstalk: the role of the kidney Acute kidney injury.CD11c⁺ cells partially mediate the renoprotective effect induced by bone marrow-derived mesenchymal stem cells.Dietary restriction and fasting arrest B and T cell development and increase mature B and T cell numbers in bone marrow.Sulfatide-reactive natural killer T cells abrogate ischemia-reperfusion injury.

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P2860

Identification of the CD4(+) T cell as a major pathogenic factor in ischemic acute renal failure.

http://www.wikidata.org/entity/Q36169286

description

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Identification of the CD4(+) T ...... ischemic acute renal failure.

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Identification of the CD4(+) T ...... ischemic acute renal failure.

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Identification of the CD4(+) T ...... ischemic acute renal failure.

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Identification of the CD4(+) T ...... ischemic acute renal failure.

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Identification of the CD4(+) T ...... ischemic acute renal failure.

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Identification of the CD4(+) T ...... ischemic acute renal failure.

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Identification of the CD4(+) T ...... ischemic acute renal failure.

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A El Ghandour

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F Daniels

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H Rabb

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M J Burne

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M P O'Donnell

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R B Colvin

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S Mauiyyedi

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P2860

A(2A) adenosine receptor-mediated inhibition of renal injury and neutrophil adhesion.

Hypothesis: is renal allograft rejection initiated by the response to injury sustained during the transplant process?

Intercellular adhesion molecule-1-deficient mice are protected against ischemic renal injury.

CD4(+) T-lymphocytes mediate ischemia/reperfusion-induced inflammatory responses in mouse liver.

The role of the B7 costimulatory pathway in experimental cold ischemia/reperfusion injury.

Endothelial cells exposed to anoxia/reoxygenation are hyperadhesive to T-lymphocytes: kinetics and molecular mechanisms.

The cytokine-adhesion molecule cascade in ischemia/reperfusion injury of the rat kidney. Inhibition by a soluble P-selectin ligand

An emerging paradigm shift on the role of leukocyte adhesion molecules.

Acute renal failure: prevailing challenges and prospects for the future.

Leukocytes, cell adhesion molecules and ischemic acute renal failure.

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