NF-kappaB translocation prevents host cell death after low-dose challenge by Legionella pneumophila. - sprookjes - yvandenbroek - TriplyDB

NF-kappaB translocation prevents host cell death after low-dose challenge by Legionella pneumophila.

NF-kappaB translocation prevents host cell death after low-dose challenge by Legionella pneumophila.

http://www.wikidata.org/entity/Q36228869

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RETRACTED: Molecular characterization of Legionella pneumophila-induced interleukin-8 expression in T cells Legionella pneumophila inhibits macrophage apoptosis by targeting pro-death members of the Bcl2 protein family Caspase Exploitation by Legionella pneumophila Subversion of Cell-Autonomous Immunity and Cell Migration by Legionella pneumophila Effectors Cell biology of infection by Legionella pneumophila Bacterial Type IV secretion systems: versatile virulence machines Type IV secretion-dependent activation of host MAP kinases induces an increased proinflammatory cytokine response to Legionella pneumophila Chemical genetics reveals bacterial and host cell functions critical for type IV effector translocation by Legionella pneumophila A genome-wide in vitro bacterial-infection screen reveals human variation in the host response associated with inflammatory disease Legionella shows a diverse secondary metabolism dependent on a broad spectrum Sfp-type phosphopantetheinyl transferase Legionella pneumophila type II secretion dampens the cytokine response of infected macrophages and epithelia Legionella pneumophila Strain 130b Evades Macrophage Cell Death Independent of the Effector SidF in the Absence of Flagellin Mechanisms of Legionella pneumophila-induced interleukin-8 expression in human lung epithelial cells.Distinct TLR- and NLR-mediated transcriptional responses to an intracellular pathogen.A Legionella pneumophila effector protein encoded in a region of genomic plasticity binds to Dot/Icm-modified vacuoles LnaB: a Legionella pneumophila activator of NF-kappaB.Inhibition of host vacuolar H+-ATPase activity by a Legionella pneumophila effector.Type three secretion system-mediated escape of Burkholderia pseudomallei into the host cytosol is critical for the activation of NFκB Molecular pathogenesis of infections caused by Legionella pneumophila Comprehensive identification of protein substrates of the Dot/Icm type IV transporter of Legionella pneumophila.Cooperation between multiple microbial pattern recognition systems is important for host protection against the intracellular pathogen Legionella pneumophila.The frustrated host response to Legionella pneumophila is bypassed by MyD88-dependent translation of pro-inflammatory cytokines.Spontaneous Irs1 passenger mutation linked to a gene-targeted SerpinB2 allele.The protein SdhA maintains the integrity of the Legionella-containing vacuole Targeting eEF1A by a Legionella pneumophila effector leads to inhibition of protein synthesis and induction of host stress response Alveolar macrophages and neutrophils are the primary reservoirs for Legionella pneumophila and mediate cytosolic surveillance of type IV secretion.The TLR4 D299G and T399I SNPs are constitutively active to up-regulate expression of Trif-dependent genes.Apoptosis-associated speck-like protein (ASC) controls Legionella pneumophila infection in human monocytes.The transcription factor C/EBP-β mediates constitutive and LPS-inducible transcription of murine SerpinB2.Exploitation of evolutionarily conserved amoeba and mammalian processes by Legionella.Striking a balance: modulation of host cell death pathways by legionella pneumophila.Targeting One of its Own: Expanding Roles of Substrates of the Legionella Pneumophila Dot/Icm Type IV Secretion System.Replication of Legionella Pneumophila in Human Cells: Why are We Susceptible?Immune Control of Legionella Infection: An in vivo Perspective.Asc-dependent and independent mechanisms contribute to restriction of legionella pneumophila infection in murine macrophages.Post-translational modifications are key players of the Legionella pneumophila infection strategy A Legionella pneumophila-translocated substrate that is required for growth within macrophages and protection from host cell death Minimization of the Legionella pneumophila genome reveals chromosomal regions involved in host range expansion.The transcriptome of Legionella pneumophila-infected human monocyte-derived macrophages.The Legionella pneumophila effector SidJ is required for efficient recruitment of endoplasmic reticulum proteins to the bacterial phagosome.

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P2860

NF-kappaB translocation prevents host cell death after low-dose challenge by Legionella pneumophila.

http://www.wikidata.org/entity/Q36228869

description

2006 nî lūn-bûn

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NF-kappaB translocation preven ...... nge by Legionella pneumophila.

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NF-kappaB translocation preven ...... nge by Legionella pneumophila.

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Large-scale identification and characterization of human genes that activate NF-kappaB and MAPK signaling pathways

IRAK (Pelle) family member IRAK-2 and MyD88 as proximal mediators of IL-1 signaling

The prosurvival Bcl-2 homolog Bfl-1/A1 is a direct transcriptional target of NF-kappaB that blocks TNFalpha-induced apoptosis

Caffeic acid phenethyl ester is a potent and specific inhibitor of activation of nuclear transcription factor NF-kappa B

Flagellin-deficient Legionella mutants evade caspase-1- and Naip5-mediated macrophage immunity

Type IV secretion: intercellular transfer of macromolecules by systems ancestrally related to conjugation machines

Reactive oxygen species promote TNFalpha-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases

IAP family proteins--suppressors of apoptosis

Recognition of cytosolic DNA activates an IRF3-dependent innate immune response

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