Elevated glucose inhibits VEGF-A-mediated endocardial cushion formation: modulation by PECAM-1 and MMP-2.
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The role of immunoglobulin superfamily cell adhesion molecules in cancer metastasisEndocardial and epicardial epithelial to mesenchymal transitions in heart development and diseaseProteomic-based detection of a protein cluster dysregulated during cardiovascular development identifies biomarkers of congenital heart defectsLigand-specific function of transforming growth factor beta in epithelial-mesenchymal transition in heart development2,3,7,8-Tetrachlorodibenzo-p-dioxin exposure prevents cardiac valve formation in developing zebrafishMatrix metalloproteinase 2-integrin alpha(v)beta3 binding is required for mesenchymal cell invasive activity but not epithelial locomotion: a computational time-lapse study.Vascular endothelial growth factor receptor signaling is required for cardiac valve formation in zebrafish.Arsenic exposure perturbs epithelial-mesenchymal cell transition and gene expression in a collagen gel assayA novel role for PECAM-1 (CD31) in regulating haematopoietic progenitor cell compartmentalization between the peripheral blood and bone marrowGenetic variation in VEGF does not contribute significantly to the risk of congenital cardiovascular malformation.Heart valve development: endothelial cell signaling and differentiationActivated protein C enhances cell motility of endothelial cells and MDA-MB-231 breast cancer cells by intracellular signal transduction.Inhibition of lung tumor growth in nude mice by siRNA(CD31) targeting PECAM-1.VEGF-A and Semaphorin3A: modulators of vascular sympathetic innervation.Identification of microRNA-93 as a novel regulator of vascular endothelial growth factor in hyperglycemic conditions.Allelic Interaction between CRELD1 and VEGFA in the Pathogenesis of Cardiac Atrioventricular Septal Defects.Acute actions and novel targets of matrix metalloproteinases in the heart and vasculatureMaternal diabetes induces congenital heart defects in mice by altering the expression of genes involved in cardiovascular developmentLeptin affects endocardial cushion formation by modulating EMT and migration via Akt signaling cascades.Endocardial-to-mesenchymal transformation and mesenchymal cell colonization at the onset of human cardiac valve developmentMechanical strain activates a program of genes functionally involved in paracrine signaling of angiogenesis.Specific local cardiovascular changes of Nepsilon-(carboxymethyl)lysine, vascular endothelial growth factor, and Smad2 in the developing embryos coincide with maternal diabetes-induced congenital heart defects.A novel matrix metalloproteinase 2 (MMP2) terminal hemopexin domain mutation in a family with multicentric osteolysis with nodulosis and arthritis with cardiac defects.Matrix metalloproteinase-2 and myocardial oxidative stress injury: beyond the matrix.Type 2 diabetes mellitus induces congenital heart defects in murine embryos by increasing oxidative stress, endoplasmic reticulum stress, and apoptosis.CD44 regulation of endothelial cell proliferation and apoptosis via modulation of CD31 and VE-cadherin expression.What chick and mouse models have taught us about the role of the endocardium in congenital heart disease.The importance of sphingolipids and reactive oxygen species in cardiovascular development.Meta-analysis of gene expression in human pancreatic islets after in vitro expansion.Frs2alpha-deficiency in cardiac progenitors disrupts a subset of FGF signals required for outflow tract morphogenesis.Synergic and antagonistic relationship between MMP-2 and MMP-9 with fibrosis and inflammation in Chagas' cardiomyopathy.MiR-23b and miR-199a impair epithelial-to-mesenchymal transition during atrioventricular endocardial cushion formation.PECAM-1 affects GSK-3beta-mediated beta-catenin phosphorylation and degradation.PECAM-1: a multifaceted regulator of megakaryocytopoiesis.Opposing actions of Notch1 and VEGF in post-natal cardiac valve endothelial cellsCardiac malformations are associated with altered expression of vascular endothelial growth factor and endothelial nitric oxide synthase genes in embryos of diabetic mice.CD44 regulates vascular endothelial barrier integrity via a PECAM-1 dependent mechanism.Activin-A in diabetes-induced cardiac malformations in embryos.Endoplasmic reticulum stress in maternal diabetes-induced cardiac malformations during critical cardiogenesis period.Understanding heart development and congenital heart defects through developmental biology: a segmental approach.
P2860
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P2860
Elevated glucose inhibits VEGF-A-mediated endocardial cushion formation: modulation by PECAM-1 and MMP-2.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh
2003年學術文章
@zh-hant
name
Elevated glucose inhibits VEGF ...... dulation by PECAM-1 and MMP-2.
@ast
Elevated glucose inhibits VEGF ...... dulation by PECAM-1 and MMP-2.
@en
type
label
Elevated glucose inhibits VEGF ...... dulation by PECAM-1 and MMP-2.
@ast
Elevated glucose inhibits VEGF ...... dulation by PECAM-1 and MMP-2.
@en
prefLabel
Elevated glucose inhibits VEGF ...... dulation by PECAM-1 and MMP-2.
@ast
Elevated glucose inhibits VEGF ...... dulation by PECAM-1 and MMP-2.
@en
P2093
P2860
P356
P1476
Elevated glucose inhibits VEGF ...... dulation by PECAM-1 and MMP-2.
@en
P2093
Emese Pinter
Joseph A Madri
Josephine M Enciso
Sandra Canosa
Todd D Camenisch
P2860
P304
P356
10.1083/JCB.200209014
P407
P577
2003-02-01T00:00:00Z