COOH-terminal truncations promote proteasome-dependent degradation of mature cystic fibrosis transmembrane conductance regulator from post-Golgi compartments.
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Misfolding diverts CFTR from recycling to degradation: quality control at early endosomesProtein homeostasis at the plasma membraneRoles for the ubiquitin-proteasome pathway in protein quality control and signaling in the retina: implications in the pathogenesis of age-related macular degenerationAn HRD/DER-independent ER quality control mechanism involves Rsp5p-dependent ubiquitination and ER-Golgi transportModulation of mature cystic fibrosis transmembrane regulator protein by the PDZ domain protein CALRegulation of cystic fibrosis transmembrane regulator trafficking and protein expression by a Rho family small GTPase TC10Involvement of the Cdc42 pathway in CFTR post-translational turnover and in its plasma membrane stability in airway epithelial cellsTracking of quantum dot-labeled CFTR shows near immobilization by C-terminal PDZ interactionsSingle-particle tracking of membrane protein diffusion in a potential: simulation, detection, and application to confined diffusion of CFTR Cl- channels.4'-Methyl-4,5'-bithiazole-based correctors of defective delta F508-CFTR cellular processing.Clinical and molecular characterization of S1118F-CFTR.The CFTR frameshift mutation 3905insT and its effect at transcript and protein level.Endocytic trafficking routes of wild type and DeltaF508 cystic fibrosis transmembrane conductance regulatorQuality control for unfolded proteins at the plasma membrane.Microparticle-mediated transfer of the viral receptors CAR and CD46, and the CFTR channel in a CHO cell model confers new functions to target cells.Effects of C-terminal deletions on cystic fibrosis transmembrane conductance regulator function in cystic fibrosis airway epitheliaRecognition of a single transmembrane degron by sequential quality control checkpoints.Secretory pathway quality control operating in Golgi, plasmalemmal, and endosomal systems.Ubiquitin-proteasome pathway and cellular responses to oxidative stress.Hepatic cytochrome P450 degradation: mechanistic diversity of the cellular sanitation brigade.Cigarette smoke exposure induces CFTR internalization and insolubility, leading to airway surface liquid dehydration.Endocytic trafficking of CFTR in health and disease.Derlin-dependent retrograde transport from endosomes to the Golgi apparatus.Enhancement of ubiquitin conjugation activity reduces intracellular aggregation of V76D mutant γD-crystallin.Reduced PDZ interactions of rescued ΔF508CFTR increases its cell surface mobility.The Bro1-related protein HD-PTP/PTPN23 is required for endosomal cargo sorting and multivesicular body morphogenesis.Cellular proteolytic systems in P450 degradation: evolutionary conservation from Saccharomyces cerevisiae to mammalian liver.Revisiting the role of cystic fibrosis transmembrane conductance regulator and counterion permeability in the pH regulation of endocytic organelles.The emerging role of the ubiquitin proteasome in pulmonary biology and disease.Protein quality control at the plasma membrane.Identification of interdependent signals required for anterograde traffic of the ATP-binding cassette transporter protein Yor1p.CFTR Folding Consortium: methods available for studies of CFTR folding and correctionAdipocyte-derived lipids increase angiotensin-converting enzyme (ACE) expression and modulate macrophage phenotype.Cystic fibrosis transmembrane conductance regulator-independent phagosomal acidification in macrophages.The role of the C terminus and Na+/H+ exchanger regulatory factor in the functional expression of cystic fibrosis transmembrane conductance regulator in nonpolarized cells and epithelia.The cytoplasmic C-terminus of the sulfonylurea receptor is important for KATP channel function but is not key for complex assembly or trafficking.The triage of damaged proteins: degradation by the ubiquitin-proteasome pathway or repair by molecular chaperones.QUANTUM DOT SINGLE MOLECULE TRACKING REVEALS A WIDE RANGE OF DIFFUSIVE MOTIONS OF MEMBRANE TRANSPORT PROTEINS.Removal of Pex3p is an important initial stage in selective peroxisome degradation in Hansenula polymorpha.Rapid degradation of solid-phase bound peptides by the 20S proteasome.
P2860
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P2860
COOH-terminal truncations promote proteasome-dependent degradation of mature cystic fibrosis transmembrane conductance regulator from post-Golgi compartments.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh
2001年學術文章
@zh-hant
name
COOH-terminal truncations prom ...... from post-Golgi compartments.
@ast
COOH-terminal truncations prom ...... from post-Golgi compartments.
@en
type
label
COOH-terminal truncations prom ...... from post-Golgi compartments.
@ast
COOH-terminal truncations prom ...... from post-Golgi compartments.
@en
prefLabel
COOH-terminal truncations prom ...... from post-Golgi compartments.
@ast
COOH-terminal truncations prom ...... from post-Golgi compartments.
@en
P2093
P2860
P356
P1476
COOH-terminal truncations prom ...... from post-Golgi compartments.
@en
P2093
G L Lukacs
M Benharouga
P2860
P304
P356
10.1083/JCB.153.5.957
P407
P577
2001-05-01T00:00:00Z