Ovine lentivirus is macrophagetropic and does not replicate productively in T lymphocytes.
about
Infection of dendritic cells by the Maedi-Visna lentivirusDuplicated sequence motif in the long terminal repeat of maedi-visna virus extends cell tropism and is associated with neurovirulencePriming with tat-deleted caprine arthritis encephalitis virus (CAEV) proviral DNA or live virus protects goats from challenge with pathogenic CAEV.In vivo monocyte tropism of pathogenic feline immunodeficiency virusesFunctional murine leukemia virus vectors pseudotyped with the visna virus envelope show expanded visna virus cell tropism.Mutations in Ovis aries TMEM154 are associated with lower small ruminant lentivirus proviral concentration in one sheep flockReduced lentivirus susceptibility in sheep with TMEM154 mutations.Pathogenesis of simian immunodeficiency virus pneumonia: an immunopathological response to virus.Ovine lentivirus expression and disease. Virus replication, but not entry, is restricted to macrophages of specific tissuesThe vif gene is essential for efficient replication of caprine arthritis encephalitis virus in goat synovial membrane cells and affects the late steps of the virus replication cycledUTPase-minus caprine arthritis-encephalitis virus is attenuated for pathogenesis and accumulates G-to-A substitutions.Early events in immune evasion by the lentivirus maedi-visna occurring within infected lymphoid tissue.Small-ruminant lentivirus enhances PrPSc accumulation in cultured sheep microglial cells.Detection of caprine arthritis-encephalitis virus by polymerase chain reaction.Development of TaqMan-based qPCR method for detection of caprine arthritis-encephalitis virus (CAEV) infectionRetroviral infections in sheep and goats: small ruminant lentiviruses and host interaction.Propagating and detecting an infectious molecular clone of maedi-visna virus that expresses green fluorescent protein.Lack of functional receptors is the only barrier that prevents caprine arthritis-encephalitis virus from infecting human cells.Molecular biology and pathogenesis of animal lentivirus infections.Initial lentivirus-host interactions within lymph nodes: a study of maedi-visna virus infection in sheepHuman immunodeficiency virus type 1 DNA synthesis, integration, and efficient viral replication in growth-arrested T cells.Simultaneous mutations in CA and Vif of Maedi-Visna virus cause attenuated replication in macrophages and reduced infectivity in vivo.Peripheral ovine progressive pneumonia provirus levels correlate with and predict histological tissue lesion severity in naturally infected sheep.Development and validation of an ovine progressive pneumonia virus quantitative PCR.Comparative Analysis of Tat-Dependent and Tat-Deficient Natural Lentiviruses.Inducible nitric oxide synthase is expressed in joints of goats in the late stage of infection with caprine arthritis encephalitis virus.
P2860
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P2860
Ovine lentivirus is macrophagetropic and does not replicate productively in T lymphocytes.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Ovine lentivirus is macrophage ...... productively in T lymphocytes.
@ast
Ovine lentivirus is macrophage ...... productively in T lymphocytes.
@en
type
label
Ovine lentivirus is macrophage ...... productively in T lymphocytes.
@ast
Ovine lentivirus is macrophage ...... productively in T lymphocytes.
@en
prefLabel
Ovine lentivirus is macrophage ...... productively in T lymphocytes.
@ast
Ovine lentivirus is macrophage ...... productively in T lymphocytes.
@en
P2093
P2860
P1433
P1476
Ovine lentivirus is macrophage ...... productively in T lymphocytes.
@en
P2093
M D Gorrell
M R Brandon
P2860
P304
P407
P577
1992-05-01T00:00:00Z