Methods for defining distinct bioenergetic profiles in platelets, lymphocytes, monocytes, and neutrophils, and the oxidative burst from human blood. - sprookjes - yvandenbroek - TriplyDB

Methods for defining distinct bioenergetic profiles in platelets, lymphocytes, monocytes, and neutrophils, and the oxidative burst from human blood.

Methods for defining distinct bioenergetic profiles in platelets, lymphocytes, monocytes, and neutrophils, and the oxidative burst from human blood.

http://www.wikidata.org/entity/Q36906968

about

Classical ROS-dependent and early/rapid ROS-independent release of Neutrophil Extracellular Traps triggered by Leishmania parasites.Teaching the basics of autophagy and mitophagy to redox biologists--mechanisms and experimental approaches Noninvasive diagnostics of mitochondrial disorders in isolated lymphocytes with high resolution respirometry Redox biology and the interface between bioenergetics, autophagy and circadian control of metabolism Bioenergetic adaptation in response to autophagy regulators during rotenone exposure Determining Maximum Glycolytic Capacity Using Extracellular Flux Measurements Monocyte bioenergetic function is associated with body composition in virologically suppressed HIV-infected women Platelet bioenergetic screen in sickle cell patients reveals mitochondrial complex V inhibition, which contributes to platelet activation.Impairment of mitochondrial respiration following ex vivo cyanide exposure in peripheral blood mononuclear cells.Comprehensive measurement of respiratory activity in permeabilized cells using extracellular flux analysis Mitochondria in monocytes and macrophages-implications for translational and basic research Metabolic remodeling of white adipose tissue in obesity Aging and energetics' 'Top 40' future research opportunities 2010-2013.The Bioenergetic Health Index: a new concept in mitochondrial translational research Metabolic plasticity in resting and thrombin activated platelets Hemoglobin-associated oxidative stress in the pericardial compartment of postoperative cardiac surgery patients.Feasibility outcomes of a presurgical randomized controlled trial exploring the impact of caloric restriction and increased physical activity versus a wait-list control on tumor characteristics and circulating biomarkers in men electing prostatectom Altered Mitochondrial Function, Mitochondrial DNA and Reduced Metabolic Flexibility in Patients With Diabetic Nephropathy.Discovery of GSK2795039, a Novel Small Molecule NADPH Oxidase 2 Inhibitor.Decreased Bioenergetic Health Index in monocytes isolated from the pericardial fluid and blood of post-operative cardiac surgery patients.Advances in the quantification of mitochondrial function in primary human immune cells through extracellular flux analysis.Correlates of Peripheral Blood Mitochondrial DNA Content in a General Population Modification of platelet proteins by 4-hydroxynonenal: Potential Mechanisms for inhibition of aggregation and metabolism Mapping the Human Platelet Lipidome Reveals Cytosolic Phospholipase A2 as a Regulator of Mitochondrial Bioenergetics during Activation.Monocyte Mitochondrial Function in Calcium Oxalate Stone Formers.Blood cell respirometry is associated with skeletal and cardiac muscle bioenergetics: Implications for a minimally invasive biomarker of mitochondrial health.Prevailing vitamin D status influences mitochondrial and glycolytic bioenergetics in peripheral blood mononuclear cells obtained from adults.Insulin resistance in HIV-infected youth is associated with decreased mitochondrial respiration A review of the mitochondrial and glycolytic metabolism in human platelets and leukocytes: implications for their use as bioenergetic biomarkers Relation of mitochondrial oxygen consumption in peripheral blood mononuclear cells to vascular function in type 2 diabetes mellitus.An Optimized Protocol to Analyze Glycolysis and Mitochondrial Respiration in Lymphocytes.Winter to summer change in vitamin D status reduces systemic inflammation and bioenergetic activity of human peripheral blood mononuclear cells.The bioenergetics of inflammation: insights into obesity and type 2 diabetes.Energy metabolic pathways control the fate and function of myeloid immune cells.Pleiotropic effects of 4-hydroxynonenal on oxidative burst and phagocytosis in neutrophils.Beyond COX-1: the effects of aspirin on platelet biology and potential mechanisms of chemoprevention.The Bioenergetic Health Index is a sensitive measure of oxidative stress in human monocytes.Microbial stimulation of different Toll-like receptor signalling pathways induces diverse metabolic programmes in human monocytes.Measurement of Mitochondrial Respiration and Motility in Acute Care: Sepsis, Trauma, and Poisoning.A Metabolic Shift toward Pentose Phosphate Pathway Is Necessary for Amyloid Fibril- and Phorbol 12-Myristate 13-Acetate-induced Neutrophil Extracellular Trap (NET) Formation

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Methods for defining distinct bioenergetic profiles in platelets, lymphocytes, monocytes, and neutrophils, and the oxidative burst from human blood.

http://www.wikidata.org/entity/Q36906968

description

2013 nî lūn-bûn

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Methods for defining distinct ...... dative burst from human blood.

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Balu K Chacko

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Michelle S Johnson

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Philip A Kramer

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Robert W Hardy

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Saranya Ravi

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Scott W Ballinger

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Victor M Darley-Usmar

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P2860

Mitochondrial dysfunction and mitophagy activation in blood mononuclear cells of fibromyalgia patients: implications in the pathogenesis of the disease

Mitochondrial dysfunction in ataxia-telangiectasia

Novel mitochondrial targets for neuroprotection

Mitochondrial genomic contribution to mitochondrial dysfunction in Alzheimer's disease.

Importance of the bioenergetic reserve capacity in response to cardiomyocyte stress induced by 4-hydroxynonenal.

Chronic granulomatous disease. Report on a national registry of 368 patients.

Convergence of hormones, inflammation, and energy-related factors: a novel pathway of cancer etiology.

Is Crohn's disease an immunodeficiency? A hypothesis suggesting possible early events in the pathogenesis of Crohn's disease.

Altered mitochondrial membrane potential, mass, and morphology in the mononuclear cells of humans with type 2 diabetes.

Prevention of diabetic nephropathy in Ins2(+/)⁻(AkitaJ) mice by the mitochondria-targeted therapy MitoQ.

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