High- and moderate-intensity training normalizes ventricular function and mechanoenergetics in mice with diet-induced obesity.
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How exercise may amend metabolic disturbances in diabetic cardiomyopathyObesity reduces left ventricular strains, torsion, and synchrony in mouse models: a cine displacement encoding with stimulated echoes (DENSE) cardiovascular magnetic resonance study.Evidence for distinct effects of exercise in different cardiac hypertrophic disorders.Cardioprotective effects of voluntary exercise in a rat model: role of matrix metalloproteinase-2.Sake Protein Supplementation Affects Exercise Performance and Biochemical Profiles in Power-Exercise-Trained MiceModerate intensity exercise prevents diabetic cardiomyopathy associated contractile dysfunction through restoration of mitochondrial function and connexin 43 levels in db/db mice.Exercise and diabetes have opposite effects on the assembly and O-GlcNAc modification of the mSin3A/HDAC1/2 complex in the heart.Interval and continuous exercise training produce similar increases in skeletal muscle and left ventricle microvascular density in ratsExercise mediated protection of diabetic heart through modulation of microRNA mediated molecular pathwaysThe "Goldilocks Zone" from a redox perspective-Adaptive vs. deleterious responses to oxidative stress in striated muscle.Branched-Chain Amino Acid Supplementation in Combination with Voluntary Running Improves Body Composition in Female C57BL/6 Mice.Exercise-induced alterations in pancreatic oxidative stress and mitochondrial function in type 2 diabetic Goto-Kakizaki rats.Obesity, Inflammation, and Exercise Training: Relative Contribution of iNOS and eNOS in the Modulation of Vascular Function in the Mouse Aorta.Sprint interval training decreases left-ventricular glucose uptake compared to moderate-intensity continuous training in subjects with type 2 diabetes or prediabetes.Exercise Training Restores Cardiac MicroRNA-1 and MicroRNA-29c to Nonpathological Levels in Obese Rats.Exercise training promotes cardioprotection through oxygen-sparing action in high fat-fed mice.High Intensity Interval Training (HIIT) improves physical performance and frailty in aged mice.Exercise of obese mice induces cardioprotection and oxygen sparing in hearts exposed to high-fat load.High-intensity interval training and calorie restriction promote remodeling of glucose and lipid metabolism in diet-induced obesity.Exercise Amaliorates Metabolic Disturbances and Oxidative Stress in Diabetic Cardiomyopathy: Possible Underlying Mechanisms.Treatment of Diabetic Mice with a Combination of Ketogenic Diet and Aerobic Exercise via Modulations of PPARs Gene Programs.Beneficial Autophagic Activities, Mitochondrial Function, and Metabolic Phenotype Adaptations Promoted by High-Intensity Interval Training in a Rat Model.
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High- and moderate-intensity training normalizes ventricular function and mechanoenergetics in mice with diet-induced obesity.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 14 March 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
High- and moderate-intensity t ...... ice with diet-induced obesity.
@en
High- and moderate-intensity t ...... ice with diet-induced obesity.
@nl
type
label
High- and moderate-intensity t ...... ice with diet-induced obesity.
@en
High- and moderate-intensity t ...... ice with diet-induced obesity.
@nl
prefLabel
High- and moderate-intensity t ...... ice with diet-induced obesity.
@en
High- and moderate-intensity t ...... ice with diet-induced obesity.
@nl
P2093
P2860
P356
P1433
P1476
High- and moderate-intensity t ...... ice with diet-induced obesity.
@en
P2093
Anje C Höper
Anne D Hafstad
Elin Hadler-Olsen
Ellen Aasum
Terje S Larsen
P2860
P304
P356
10.2337/DB12-1580
P407
P577
2013-03-14T00:00:00Z