Quality control of DNA break metabolism: in the 'end', it's a good thing.
about
ATM and Artemis promote homologous recombination of radiation-induced DNA double-strand breaks in G2When stem cells grow old: phenotypes and mechanisms of stem cell agingRepair of strand breaks by homologous recombinationThe Shu complex, which contains Rad51 paralogues, promotes DNA repair through inhibition of the Srs2 anti-recombinase.Localization of recombination proteins and Srs2 reveals anti-recombinase function in vivo.Regulation of homologous recombination in eukaryotes.The achene mucilage hydrated in desert dew assists seed cells in maintaining DNA integrity: adaptive strategy of desert plant Artemisia sphaerocephala.Synthetic lethality between gene defects affecting a single non-essential molecular pathway with reversible steps.Crystal structure of the Mre11-Rad50-ATPγS complex: understanding the interplay between Mre11 and Rad50LIA5 is required for nuclear reorganization and programmed DNA rearrangements occurring during tetrahymena macronuclear differentiationA genomic screen revealing the importance of vesicular trafficking pathways in genome maintenance and protection against genotoxic stress in diploid Saccharomyces cerevisiae cellsPlk1 and CK2 act in concert to regulate Rad51 during DNA double strand break repair.High-throughput strategies for the discovery and engineering of enzymes for biocatalysis.The emerging role of nuclear architecture in DNA repair and genome maintenance.DNA-damage repair; the good, the bad, and the ugly.Biochemical analysis of human PIF1 helicase and functions of its N-terminal domain.Association between the XRCC6 Promoter rs2267437 polymorphism and cancer risk: evidence based on the current literatureTelomere heterogeneity: taking advantage of stochastic events.TEN1 is essential for CDC13-mediated telomere capping.The emerging role of HP1 in the DNA damage response.Taming the tiger by the tail: modulation of DNA damage responses by telomeres.When the caps fall off: responses to telomere uncapping in yeast.Abnormalities of DNA repair mechanisms in common hematological malignancies.Mediators of homologous DNA pairing.Regulation of recombination and genomic maintenance.Transient expression of RAD51 in the late G2-phase is required for cell cycle progression in synchronous Physarum cells.
P2860
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P2860
Quality control of DNA break metabolism: in the 'end', it's a good thing.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
@tr
scientific article published on February 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Quality control of DNA break metabolism: in the 'end', it's a good thing.
@en
Quality control of DNA break metabolism: in the 'end', it's a good thing.
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type
label
Quality control of DNA break metabolism: in the 'end', it's a good thing.
@en
Quality control of DNA break metabolism: in the 'end', it's a good thing.
@nl
prefLabel
Quality control of DNA break metabolism: in the 'end', it's a good thing.
@en
Quality control of DNA break metabolism: in the 'end', it's a good thing.
@nl
P2860
P356
P1433
P1476
Quality control of DNA break metabolism: in the 'end', it's a good thing.
@en
P2093
Claire Wyman
Roland Kanaar
P2860
P304
P356
10.1038/EMBOJ.2008.11
P407
P577
2008-02-01T00:00:00Z