Which in vitro models could be best used to study hepatocyte polarity? - sprookjes - yvandenbroek - TriplyDB

Which in vitro models could be best used to study hepatocyte polarity?

Which in vitro models could be best used to study hepatocyte polarity?

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Efficient generation of hepatic cells from mesenchymal stromal cells by an innovative bio-microfluidic cell culture device.Polarization Restricts Hepatitis C Virus Entry into HepG2 Hepatoma Cells Three-dimensional Huh7 cell culture system for the study of Hepatitis C virus infection A novel mouse model for stable engraftment of a human immune system and human hepatocytes Claudin association with CD81 defines hepatitis C virus entry.Cytokinesis defines a spatial landmark for hepatocyte polarization and apical lumen formation.A comprehensive comparison of transmembrane domains reveals organelle-specific properties.Regulation of bile canalicular network formation and maintenance by AMP-activated protein kinase and LKB1.HepG2 cells expressing microRNA miR-122 support the entire hepatitis C virus life cycle.Systems biology approaches for studying the pathogenesis of non-alcoholic fatty liver disease.Functional 3D human primary hepatocyte spheroids made by co-culturing hepatocytes from partial hepatectomy specimens and human adipose-derived stem cells.Hepatitis C virus receptors claudin-1 and occludin after liver transplantation and influence on early viral kinetics.Demonstration of the presence of the "deleted" MIR122 gene in HepG2 cells.Microengineered cell and tissue systems for drug screening and toxicology applications: Evolution of in-vitro liver technologies.Fabrication of functional 3D hepatic tissues with polarized hepatocytes by stacking endothelial cell sheets in vitro.Hepatitis C virus infection reduces hepatocellular polarity in a vascular endothelial growth factor-dependent manner Hepatocyte polarity Hepatitis c virus genotype 4 replication in the hepatocellular carcinoma cell line HepG2/C3A.A novel helper-dependent adenovirus-based cell culture model for Hepatitis C virus replication and production Primary hepatocytes as targets for hepatitis C virus replication.AKAP350 Is involved in the development of apical "canalicular" structures in hepatic cells HepG2.Low-dose acetaminophen induces early disruption of cell-cell tight junctions in human hepatic cells and mouse liver.α1-antitrypsin deficiency and the hepatocytes - an elegans solution to drug discovery.Chelation therapy in Wilson's disease: from D-penicillamine to the design of selective bioinspired intracellular Cu(I) chelators.Hepatitis C virus entry: beyond receptors.Primary hepatocyte cultures for pharmaco-toxicological studies: at the busy crossroad of various anti-dedifferentiation strategies.Induced pluripotent stem cells as a source of hepatocytes.Entry and release of hepatitis C virus in polarized human hepatocytes.Hepatocyte-like cells derived from induced pluripotent stem cells.The special case of hepatocytes: unique tissue architecture calls for a distinct mode of cell division.Cellular Models and In Vitro Assays for the Screening of modulators of P-gp, MRP1 and BCRP.A sulfur tripod glycoconjugate that releases a high-affinity copper chelator in hepatocytes.Claudin-1 involved in neonatal ichthyosis sclerosing cholangitis syndrome regulates hepatic paracellular permeability.Regulatory subunit I-controlled protein kinase A activity is required for apical bile canalicular lumen development in hepatocytes TNF superfamily members promote hepatitis C virus entry via an NF-κB and myosin light chain kinase dependent pathway.Hepatoma polarization limits CD81 and hepatitis C virus dynamics.IFITM1 is a tight junction protein that inhibits hepatitis C virus entry.Differential basolateral-apical distribution of scavenger receptor, class B, type I in cultured cells and the liver.In vitro systems for the study of hepatitis C virus infection.Build them up and break them down: Tight junctions of cell lines expressing typical hepatocyte polarity with a varied repertoire of claudins.

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P2860

Which in vitro models could be best used to study hepatocyte polarity?

http://www.wikidata.org/entity/Q37189719

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on July 2008

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Which in vitro models could be best used to study hepatocyte polarity?

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Which in vitro models could be best used to study hepatocyte polarity?

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type

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Which in vitro models could be best used to study hepatocyte polarity?

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Which in vitro models could be best used to study hepatocyte polarity?

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Which in vitro models could be best used to study hepatocyte polarity?

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Which in vitro models could be best used to study hepatocyte polarity?

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Biology of the Cell

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Which in vitro models could be best used to study hepatocyte polarity?

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Brigitte Grosse

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Catherine Decaens

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Doris Cassio

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Marjorie Durand

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P2860

A gene encoding a liver-specific ABC transporter is mutated in progressive familial intrahepatic cholestasis

Structural organization of the endoplasmic reticulum.

Par-1 promotes a hepatic mode of apical protein trafficking in MDCK cells

WIF-B cells: an in vitro model for studies of hepatocyte polarity

Expression and subcellular localization of aquaporin water channels in the polarized hepatocyte cell line, WIF-B

Replication of subgenomic hepatitis C virus RNAs in a hepatoma cell line

Radixin deficiency causes conjugated hyperbilirubinemia with loss of Mrp2 from bile canalicular membranes

Trafficking of the bile salt export pump from the Golgi to the canalicular membrane is regulated by the p38 MAP kinase

Differential regulation of rodent hepatocyte and oval cell proliferation by interferon gamma

NH2-terminal signals in ATP7B Cu-ATPase mediate its Cu-dependent anterograde traffic in polarized hepatic cells

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387-398

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scholarly article

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10.1042/BC20070127

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7

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100

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2008-07-01T00:00:00Z

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18549352

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