Targeting binding partners of the CBFβ-SMMHC fusion protein for the treatment of inversion 16 acute myeloid leukemia. - sprookjes - yvandenbroek - TriplyDB

Targeting binding partners of the CBFβ-SMMHC fusion protein for the treatment of inversion 16 acute myeloid leukemia.

Targeting binding partners of the CBFβ-SMMHC fusion protein for the treatment of inversion 16 acute myeloid leukemia.

http://www.wikidata.org/entity/Q37662400

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Myeloid neoplasms with concurrent BCR-ABL1 and CBFB rearrangements: A series of 10 cases of a clinically aggressive neoplasm.Preleukemia and Leukemia-Initiating Cell Activity in inv(16) Acute Myeloid Leukemia.

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Targeting binding partners of the CBFβ-SMMHC fusion protein for the treatment of inversion 16 acute myeloid leukemia.

http://www.wikidata.org/entity/Q37662400

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 17 August 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Targeting binding partners of ...... ion 16 acute myeloid leukemia.

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Targeting binding partners of ...... ion 16 acute myeloid leukemia.

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Targeting binding partners of ...... ion 16 acute myeloid leukemia.

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Targeting binding partners of ...... ion 16 acute myeloid leukemia.

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ONCOTARGET.11357

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Targeting binding partners of ...... ion 16 acute myeloid leukemia.

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Lisa Richter

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R Katherine Hyde

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Yiqian Wang

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Creative Commons Attribution 2.5 Unported

P2860

Structural basis for the heterodimeric interaction between the acute leukaemia-associated transcription factors AML1 and CBFbeta

Fusion between transcription factor CBF beta/PEBP2 beta and a myosin heavy chain in acute myeloid leukemia

Fusion of the TEL gene on 12p13 to the AML1 gene on 21q22 in acute lymphoblastic leukemia

The inv(16) fusion protein associates with corepressors via a smooth muscle myosin heavy-chain domain.

Disruption of the Cbfa2 gene causes necrosis and hemorrhaging in the central nervous system and blocks definitive hematopoiesis

The inv(16) encodes an acute myeloid leukemia 1 transcriptional corepressor

Interplay between Transcription Factors and the Epigenome: Insight from the Role of RUNX1 in Leukemia

Solution structure of core binding factor beta and map of the CBF alpha binding site

Prognostic relevance of integrated genetic profiling in acute myeloid leukemia.

Progress towards small molecule inhibitors of the Menin-Mixed Lineage Leukemia (MLL) interaction with in vivo utility

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66255-66266

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10.18632/ONCOTARGET.11357

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2016-10-04T00:00:00Z

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DNA-binding protein

acute myeloid leukemia

genetic variation

transcription factor

neoplasm proteins

mutant proteins

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