Stromal Senescence By Prolonged CDK4/6 Inhibition Potentiates Tumor Growth.

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Senescent cells: an emerging target for diseases of ageing.Senescence and tumor suppression.NOTCH-mediated non-cell autonomous regulation of chromatin structure during senescence.Non-Cell Autonomous Effects of the Senescence-Associated Secretory Phenotype in Cancer Therapy.

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P2860

Stromal Senescence By Prolonged CDK4/6 Inhibition Potentiates Tumor Growth.

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http://www.wikidata.org/entity/Q39048659

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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2016年论文

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name

Stromal Senescence By Prolonged CDK4/6 Inhibition Potentiates Tumor Growth.

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Stromal Senescence By Prolonged CDK4/6 Inhibition Potentiates Tumor Growth.

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type

Item

label

Stromal Senescence By Prolonged CDK4/6 Inhibition Potentiates Tumor Growth.

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Stromal Senescence By Prolonged CDK4/6 Inhibition Potentiates Tumor Growth.

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Stromal Senescence By Prolonged CDK4/6 Inhibition Potentiates Tumor Growth.

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Stromal Senescence By Prolonged CDK4/6 Inhibition Potentiates Tumor Growth.

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P1476

Stromal Senescence By Prolonged CDK4/6 Inhibition Potentiates Tumor Growth.

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P2093

Christin E Burd

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Christina Y Yu

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Jianying Zhang

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Kyle M LaPak

http://www.w3.org/2001/XMLSchema#string

Rebecca C Hennessey

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Reena Shakya

http://www.w3.org/2001/XMLSchema#string

Xiangnan Guan

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P2860

Immuno-oncology combinations: a review of clinical experience and future prospects

Senescence-associated secretory phenotypes reveal cell-nonautonomous functions of oncogenic RAS and the p53 tumor suppressor

Cellular senescence: when bad things happen to good cells

The senescence-associated secretory phenotype: the dark side of tumor suppression

A human-like senescence-associated secretory phenotype is conserved in mouse cells dependent on physiological oxygen.

The inflammatory network: bridging senescent stroma and epithelial tumorigenesis.

Inflammatory networks during cellular senescence: causes and consequences.

Senescent fibroblasts promote epithelial cell growth and tumorigenesis: a link between cancer and aging.

A complex secretory program orchestrated by the inflammasome controls paracrine senescence.

The meaning of p16(ink4a) expression in tumors: functional significance, clinical associations and future developments

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237-249

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scholarly article

P356

10.1158/1541-7786.MCR-16-0319

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2016-12-30T00:00:00Z

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28039358

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5334447

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Stromal Senescence By Prolonged CDK4/6 Inhibition Potentiates Tumor Growth.

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P2860

P2860

Stromal Senescence By Prolonged CDK4/6 Inhibition Potentiates Tumor Growth.

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P1433

P1476

P2093

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P932