Inhibition of JNK by a peptide inhibitor reduces traumatic brain injury-induced tauopathy in transgenic mice
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c-Jun N-terminal Kinase (JNK) Signaling as a Therapeutic Target for Alzheimer's DiseaseAcute and chronic traumatic encephalopathies: pathogenesis and biomarkers.Linking traumatic brain injury to chronic traumatic encephalopathy: identification of potential mechanisms leading to neurofibrillary tangle developmentAnimal models of traumatic brain injuryChronic effects of mild neurotrauma: putting the cart before the horse?Alzheimer’s disease and chronic traumatic encephalopathy: distinct but possibly overlapping disease entitiesChronic traumatic encephalopathy: clinical-biomarker correlations and current concepts in pathogenesis.Decreased level of olfactory receptors in blood cells following traumatic brain injury and potential association with tauopathyThe pathophysiology of repetitive concussive traumatic brain injury in experimental models; new developments and open questionsIn Silico Modeling of Novel Drug Ligands for Treatment of Concussion Associated Tauopathy.Lithium reduces BACE1 overexpression, β amyloid accumulation, and spatial learning deficits in mice with traumatic brain injury.Rapid accumulation of endogenous tau oligomers in a rat model of traumatic brain injury: possible link between traumatic brain injury and sporadic tauopathiesAmyloid-β Peptides and Tau Protein as Biomarkers in Cerebrospinal and Interstitial Fluid Following Traumatic Brain Injury: A Review of Experimental and Clinical Studies.PTEN/PI3K and MAPK signaling in protection and pathology following CNS injuries.Protective effects of decay-accelerating factor on blast-induced neurotrauma in rats.Evidence of Presynaptic Localization and Function of the c-Jun N-Terminal KinaseJNK inhibitors as anti-inflammatory and neuroprotective agents.Nuclear and cytosolic JNK signalling in neurons.The Pathophysiology of Concussion.Strong Correlation of Genome-Wide Expression after Traumatic Brain Injury In Vitro and In Vivo Implicates a Role for SORLA.Sex and Age Differences in Epinephrine Mechanisms and Outcomes after Brain Injury.A Brief Overview of Tauopathy: Causes, Consequences, and Therapeutic Strategies.Adenosine A2A receptor inactivation alleviates early-onset cognitive dysfunction after traumatic brain injury involving an inhibition of tau hyperphosphorylationCisternostomy: Replacing the age old decompressive hemicraniectomy?Chronic traumatic encephalopathy: A paradigm in search of evidence?Traumatic brain injuries.CNS Injury: Posttranslational Modification of the Tau Protein as a Biomarker.Inhibition of c-Jun N-Terminal Kinase Protects Against Brain Damage and Improves Learning and Memory After Traumatic Brain Injury in Adult Mice.Tau-based therapies in neurodegeneration: opportunities and challenges.Repetitive mild traumatic brain injury augments tau pathology and glial activation in aged hTau mice.Involvement of TG-interacting factor in microglial activation during experimental traumatic brain injury.The Inflammatory Continuum of Traumatic Brain Injury and Alzheimer's Disease.Phosphorylation of Threonine 175 Tau in the Induction of Tau Pathology in Amyotrophic Lateral Sclerosis-Frontotemporal Spectrum Disorder (ALS-FTSD). A Review.
P2860
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P2860
Inhibition of JNK by a peptide inhibitor reduces traumatic brain injury-induced tauopathy in transgenic mice
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
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2012年论文
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name
Inhibition of JNK by a peptide ...... d tauopathy in transgenic mice
@en
type
label
Inhibition of JNK by a peptide ...... d tauopathy in transgenic mice
@en
prefLabel
Inhibition of JNK by a peptide ...... d tauopathy in transgenic mice
@en
P2093
P2860
P1476
Inhibition of JNK by a peptide ...... d tauopathy in transgenic mice
@en
P2093
David L Brody
Hien T Tran
Laura Sanchez
P2860
P304
P356
10.1097/NEN.0B013E3182456AED
P577
2012-02-01T00:00:00Z