The biogenesis of the MHC class II compartment in human I-cell disease B lymphoblasts.
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Enzymatic reduction of disulfide bonds in lysosomes: characterization of a gamma-interferon-inducible lysosomal thiol reductase (GILT)HIV-1 Nef impairs MHC class II antigen presentation and surface expressionDeficient peptide loading and MHC class II endosomal sorting in a human genetic immunodeficiency disease: the Chediak-Higashi syndromeAntibody-Based Assays for Phenotyping of Extracellular VesiclesExpression of major histocompatibility complex class II molecules in HeLa cells promotes the recruitment of AP-1 Golgi-specific assembly proteins on Golgi membranesPhagocytic antigen processing and effects of microbial products on antigen processing and T-cell responses.Involvement of clathrin and AP-2 in the trafficking of MHC class II molecules to antigen-processing compartmentsThe IGF axis and hepatocarcinogenesis.Phagocytic processing of antigens for presentation by class II major histocompatibility complex molecules.The export of major histocompatibility complex class I molecules from the endoplasmic reticulum of rat brown adipose cells is acutely stimulated by insulinGlucose transporter (GLUT-4) is targeted to secretory granules in rat atrial cardiomyocytesA dominant-negative clathrin mutant differentially affects trafficking of molecules with distinct sorting motifs in the class II major histocompatibility complex (MHC) pathwayRedundant and distinct functions for dynamin-1 and dynamin-2 isoforms.Early endosomes are required for major histocompatiblity complex class II transport to peptide-loading compartments.Accumulation of major histocompatibility complex class II molecules in mast cell secretory granules and their release upon degranulationSynaptic vesicles form by budding from tubular extensions of sorting endosomes in PC12 cellsImaging and imagination: understanding the endo-lysosomal systemThe Role of Cathepsin D in the Pathogenesis of Human Neurodegenerative Disorders.Extracellular Vesicle (EV) Array: microarray capturing of exosomes and other extracellular vesicles for multiplexed phenotyping.Multiplexed Phenotyping of Small Extracellular Vesicles Using Protein Microarray (EV Array).AP2 clathrin adaptor complex, but not AP1, controls the access of the major histocompatibility complex (MHC) class II to endosomes.Functional early endosomes are required for maturation of major histocompatibility complex class II molecules in human B lymphoblastoid cells.Major histocompatibility complex class II compartments in human and mouse B lymphoblasts represent conventional endocytic compartmentsVacuolization of mucolipidosis type II mouse exocrine gland cells represents accumulation of autolysosomes.Disruption of the Man-6-P targeting pathway in mice impairs osteoclast secretory lysosome biogenesis.Pig kidney legumain: an asparaginyl endopeptidase with restricted specificity.Endogenous ligands of carbohydrate recognition domains of the mannose receptor in murine macrophages, endothelial cells and secretory cells; potential relevance to inflammation and immunity.Transient surface delivery of invariant chain-MHC II complexes via endosomes: a quantitative study.
P2860
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P2860
The biogenesis of the MHC class II compartment in human I-cell disease B lymphoblasts.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
The biogenesis of the MHC class II compartment in human I-cell disease B lymphoblasts.
@en
type
label
The biogenesis of the MHC class II compartment in human I-cell disease B lymphoblasts.
@en
prefLabel
The biogenesis of the MHC class II compartment in human I-cell disease B lymphoblasts.
@en
P2093
P2860
P356
P1476
The biogenesis of the MHC class II compartment in human I-cell disease B lymphoblasts.
@en
P2093
J N Glickman
P A Morton
S Kornfeld
P2860
P304
P356
10.1083/JCB.132.5.769
P407
P577
1996-03-01T00:00:00Z