Hypoglycemic neuronal death is triggered by glucose reperfusion and activation of neuronal NADPH oxidase. - sprookjes - yvandenbroek - TriplyDB

Hypoglycemic neuronal death is triggered by glucose reperfusion and activation of neuronal NADPH oxidase.

Hypoglycemic neuronal death is triggered by glucose reperfusion and activation of neuronal NADPH oxidase.

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Hypoglycemia and death in mice following experimental exposure to an extract of Trogia venenata mushrooms Glycemic control and hypoglycemia: is the loser the winner?Migraine Triggers and Oxidative Stress: A Narrative Review and Synthesis Hypothalamic-Pituitary-Adrenal Axis Programming after Recurrent Hypoglycemia during Development Acute hypoglycemia induces retinal cell death in mouse Severe hypoglycemia in a juvenile diabetic rat model: presence and severity of seizures are associated with mortality Pituitary Adenlylate Cyclase Activating Peptide Protects Adult Neural Stem Cells from a Hypoglycaemic milieu Two-dimensional zymography differentiates gelatinase isoforms in stimulated microglial cells and in brain tissues of acute brain injuries Recurrent/moderate hypoglycemia induces hippocampal dendritic injury, microglial activation, and cognitive impairment in diabetic rats.Neurocognitive changes and their neural correlates in patients with type 2 diabetes mellitus Changes in ascorbate, glutathione and α-tocopherol concentrations in the brain regions during normal development and moderate hypoglycemia in rats.Mitochondrial dysfunction induced by nuclear poly(ADP-ribose) polymerase-1: a treatable cause of cell death in stroke.Hypoglycemia aggravates critical illness-induced neurocognitive dysfunction Recurrent moderate hypoglycemia ameliorates brain damage and cognitive dysfunction induced by severe hypoglycemia.Systemic glucose variability predicts cerebral metabolic distress and mortality after subarachnoid hemorrhage: a retrospective observational study.Hypoglycemia induced changes in cholinergic receptor expression in the cerebellum of diabetic rats.Cocaine and human immunodeficiency virus type 1 gp120 mediate neurotoxicity through overlapping signaling pathways.NADPH oxidase 1, a novel molecular source of ROS in hippocampal neuronal death in vascular dementia.Effects of tight computerized glucose control on neurological outcome in severely brain injured patients: a multicenter sub-group analysis of the randomized-controlled open-label CGAO-REA study Targeting NOX enzymes in the central nervous system: therapeutic opportunities.Pharmacologic amelioration of severe hypoglycemia-induced neuronal damage.Oxidative stress in ischemic brain damage: mechanisms of cell death and potential molecular targets for neuroprotection NADPH oxidase-2: linking glucose, acidosis, and excitotoxicity in stroke.Glucose and NADPH oxidase drive neuronal superoxide formation in stroke.Impairment of autophagic flux promotes glucose reperfusion-induced neuro2A cell death after glucose deprivation.Phosphatidylinositol-3-kinase gamma plays a central role in blood-brain barrier dysfunction in acute experimental stroke.Expression of aquaporin 4 and breakdown of the blood-brain barrier after hypoglycemia-induced brain edema in rats.Protection of hypoglycemia-induced neuronal death by β-hydroxybutyrate involves the preservation of energy levels and decreased production of reactive oxygen species White matter hyperintensities in middle-aged adults with childhood-onset type 1 diabetes Effects of Alda-1, an Aldehyde Dehydrogenase-2 Agonist, on Hypoglycemic Neuronal Death Inhibition of NADPH oxidase activation reduces EAE-induced white matter damage in mice.Glycemic control in the pediatric intensive care unit of Leuven: two years of experience.Dynamic optical imaging of metabolic and NADPH oxidase-derived superoxide in live mouse brain using fluorescence lifetime unmixing Hyperglycemia promotes tissue plasminogen activator-induced hemorrhage by Increasing superoxide production Oxidative stress and cell death in the cerebral cortex as a long-term consequence of neonatal hypoglycemia.The 1027th target candidate in stroke: Will NADPH oxidase hold up?Neonatal Glycemia and Neurodevelopmental Outcomes at 2 Years.Evidence that hyperglycemia after recovery from hypoglycemia worsens endothelial function and increases oxidative stress and inflammation in healthy control subjects and subjects with type 1 diabetes.Neonatal hypoglycemic brain injury is a cause of infantile spasms.Antecedent glycemic control reduces severe hypoglycemia-induced neuronal damage in diabetic rats

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Hypoglycemic neuronal death is triggered by glucose reperfusion and activation of neuronal NADPH oxidase.

http://www.wikidata.org/entity/Q42062495

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2007 nî lūn-bûn

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Hypoglycemic neuronal death is ...... ion of neuronal NADPH oxidase.

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Hypoglycemic neuronal death is ...... ion of neuronal NADPH oxidase.

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Hypoglycemic neuronal death is ...... ion of neuronal NADPH oxidase.

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Journal of Clinical Investigation

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Hypoglycemic neuronal death is ...... ion of neuronal NADPH oxidase.

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Aaron M Hamby

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Elizabeth T Gum

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Pak H Chan

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Raymond A Swanson

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Sang Won Suh

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P2860

Activation and assembly of the NADPH oxidase: a structural perspective

Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage

Fluoro-Jade B: a high affinity fluorescent marker for the localization of neuronal degeneration.

Reactive oxygen radicals in signaling and damage in the ischemic brain.

Cell signaling. H2O2, a necessary evil for cell signaling.

Impact of improved glycaemic control on rates of hypoglycaemia in insulin dependent diabetes mellitus.

Synaptic localization of a functional NADPH oxidase in the mouse hippocampus.

The neurobiology of zinc in health and disease.

Regulation and termination of NADPH oxidase activity.

Calcium, mitochondria and oxidative stress in neuronal pathology. Novel aspects of an enduring theme.

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