about
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)Apoptosis repressor with caspase recruitment domain protects against cell death by interfering with Bax activationThe Janus Face of Death Receptor Signaling during Tumor ImmunoeditingDevelopment of a PCR-SSOP approach capable of defining the natural killer cell inhibitory receptor (KIR) gene sequence repertoiresResponse to myocardial ischemia/reperfusion injury involves Bnip3 and autophagyER stress contributes to ischemia-induced cardiomyocyte apoptosisMechanisms of ER Stress-Mediated Mitochondrial Membrane Permeabilization.Inhibitor of apoptosis proteins (IAPs) and their antagonists regulate spontaneous and tumor necrosis factor (TNF)-induced proinflammatory cytokine and chemokine production.Disruption of microRNA biogenesis confers resistance to ER stress-induced cell death upstream of the mitochondrion.Deficiency in the mitochondrial apoptotic pathway reveals the toxic potential of autophagy under ER stress conditions.Ischemia/reperfusion injury at the intersection with cell death.Endoplasmic reticulum stress-mediated induction of SESTRIN 2 potentiates cell survival.Stressed to death - mechanisms of ER stress-induced cell death.Caspase-1 promiscuity is counterbalanced by rapid inactivation of processed enzyme.BCL-2 modulates the unfolded protein response by enhancing splicing of X-box binding protein-1.Hypoxia and ischemia induce nuclear condensation and caspase activation in cardiomyocytes.Granzyme B-dependent proteolysis acts as a switch to enhance the proinflammatory activity of IL-1α.Expression, purification and use of recombinant annexin V for the detection of apoptotic cells.Fas/CD95-induced chemokines can serve as "find-me" signals for apoptotic cells.Targeting the angio-proteostasis network: Combining the forces against cancer.Identification of an inhibitor of caspase activation from heart extracts; ATP blocks apoptosome formation.The Unfolded Protein Response in Breast CancerInhibition of IRE1 RNase activity modulates the tumor cell secretome and enhances response to chemotherapyCaspase activation cascades in apoptosisGuidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)Correction: Inhibition of IRE1α RNase activity reduces NLRP3 inflammasome assembly and processing of pro-IL1βInhibition of IRE1α RNase activity reduces NLRP3 inflammasome assembly and processing of pro-IL1β
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description
hulumtuese
@sq
researcher
@en
ricercatrice
@it
wetenschapper
@nl
հետազոտող
@hy
name
Susan Logue
@ast
Susan Logue
@en
Susan Logue
@es
Susan Logue
@nl
Susan Logue
@sl
type
label
Susan Logue
@ast
Susan Logue
@en
Susan Logue
@es
Susan Logue
@nl
Susan Logue
@sl
prefLabel
Susan Logue
@ast
Susan Logue
@en
Susan Logue
@es
Susan Logue
@nl
Susan Logue
@sl
P106
P108
P21
P214
5114156619126128780008
P31
P496
0000-0001-7938-3558