about
Che-1 phosphorylation by ATM/ATR and Chk2 kinases activates p53 transcription and the G2/M checkpointInhibition of ErbB-2 mitogenic and transforming activity by RALT, a mitogen-induced signal transducer which binds to the ErbB-2 kinase domainThe accumulation of MMS-induced single strand breaks in G1 phase is recombinogenic in DNA polymerase beta defective mammalian cellsA role for oxidized DNA precursors in Huntington's disease-like striatal neurodegenerationThe mammalian mismatch repair protein MSH2 is required for correct MRE11 and RAD51 relocalization and for efficient cell cycle arrest induced by ionizing radiation in G2 phasePeroxynitrite induces tyrosine residue modifications in synaptophysin C-terminal domain, affecting its interaction with srcPhosphorylation on threonine 11 of β-dystrobrevin alters its interaction with kinesin heavy chainThe nebulin SH3 domain is dispensable for normal skeletal muscle structure but is required for effective active load bearing in mouse.Megalencephalic leukoencephalopathy with subcortical cysts protein-1 modulates endosomal pH and protein trafficking in astrocytes: relevance to MLC disease pathogenesisThe main biological determinants of tumor line taxonomy elucidated by a principal component analysis of microarray data.MetaShot: an accurate workflow for taxon classification of host-associated microbiome from shotgun metagenomic data.A pRb-independent mechanism preserves the postmitotic state in terminally differentiated skeletal muscle cells.The Giardia duodenalis 14-3-3 protein is post-translationally modified by phosphorylation and polyglycylation of the C-terminal tail.A simple and effective method to analyze membrane proteins by SDS-PAGE and MALDI mass spectrometry.A cancer-specific transcriptional signature in human neoplasia.An insight into the abundant proteome of 46BR.1G1 fibroblasts deficient of DNA ligase I.HIPK2 sustains apoptotic response by phosphorylating Che-1/AATF and promoting its degradationKnockdown of cyclin-dependent kinase inhibitors induces cardiomyocyte re-entry in the cell cycleNew functions of XPC in the protection of human skin cells from oxidative damageProliferation of Multiple Cell Types in the Skeletal Muscle Tissue Elicited by Acute p21 Suppression.Terminally differentiated muscle cells are defective in base excision DNA repair and hypersensitive to oxygen injury.Critical requirement for cell cycle inhibitors in sustaining nonproliferative states.Differentiation-associated microRNAs antagonize the Rb-E2F pathway to restrict proliferation.Np95 is regulated by E1A during mitotic reactivation of terminally differentiated cells and is essential for S phase entryNon-proliferation as an active state: conceptual and practical implications.The logic and regulation of cell cycle exit and reentry.Levels matter: miR-206 and cyclin D1.Estrogens enhance myoblast differentiation in facioscapulohumeral muscular dystrophy by antagonizing DUX4 activity.Hydra, a versatile model to study the homeostatic and developmental functions of cell death.Robust G2 pausing of adult stem cells in Hydra.Identification of a molecular signature for leukemic promyelocytes and their normal counterparts: Focus on DNA repair genes.The inhibition of cyclin B1 gene transcription in quiescent NIH3T3 cells is mediated by an E-box.Anti-GAPDH Autoantibodies as a Pathogenic Determinant and Potential Biomarker of Neuropsychiatric Diseases.Gaucher disease due to saposin C deficiency is an inherited lysosomal disease caused by rapidly degraded mutant proteins.Trends in tissue repair and regeneration.Phosphorylation of SRSF1 is modulated by replicational stressWild-type p53 induces diverse effects in 32D cells expressing different oncogenes.Expression of E1A in terminally differentiated muscle cells reactivates the cell cycle and suppresses tissue-specific genes by separable mechanismsStreptococcal-vimentin cross-reactive antibodies induce microvascular cardiac endothelial proinflammatory phenotype in rheumatic heart disease.Long-term fate of terminally differentiated skeletal muscle cells following E1A-initiated cell cycle reactivation.
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hulumtues
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հետազոտող
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Marco Crescenzi
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Marco Crescenzi
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Marco Crescenzi
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Marco Crescenzi
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Marco Crescenzi
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Marco Crescenzi
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Marco Crescenzi
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Marco Crescenzi
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Marco Crescenzi
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Marco Crescenzi
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Marco Crescenzi
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Marco Crescenzi
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Marco Crescenzi
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Marco Crescenzi
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Marco Crescenzi
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