Decreased brain zinc availability reduces hippocampal neurogenesis in mice and rats. - sprookjes - yvandenbroek - TriplyDB

Decreased brain zinc availability reduces hippocampal neurogenesis in mice and rats.

Decreased brain zinc availability reduces hippocampal neurogenesis in mice and rats.

http://www.wikidata.org/entity/Q45393764

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Unified theory of Alzheimer's disease (UTAD): implications for prevention and curative therapy Behavioral Abnormality Induced by Enhanced Hypothalamo-Pituitary-Adrenocortical Axis Activity under Dietary Zinc Deficiency and Its Usefulness as a Model Zinc and neurogenesis: making new neurons from development to adulthood Zinc, vitamin A, and glutamine supplementation in Brazilian shantytown children at risk for diarrhea results in sex-specific improvements in verbal learning The Emerging Role for Zinc in Depression and Psychosis.Zinc and the ERK kinases in the developing brain.Biological consequences of zinc deficiency in the pathomechanisms of selected diseases.Regional characterization of energy metabolism in the brain of normal and MPTP-intoxicated mice using new markers of glucose and phosphate transport.Zinc: indications in brain disorders.Zinc chelation reduces hippocampal neurogenesis after pilocarpine-induced seizure.Serum zinc concentrations correlate with mental and physical status of nursing home residents.Dietary zinc reduction, pyruvate supplementation, or zinc transporter 5 knockout attenuates β-cell death in nonobese diabetic mice, islets, and insulinoma cells.Zinc deficiency and neurodevelopment: the case of neurons.Zinc homeostasis and neurodegenerative disorders Zinc in the central nervous system: From molecules to behavior.Chelation of hippocampal zinc enhances long-term potentiation and synaptic tagging/capture in CA1 pyramidal neurons of aged rats: implications to aging and memory.Administration of Zinc plus Cyclo-(His-Pro) Increases Hippocampal Neurogenesis in Rats during the Early Phase of Streptozotocin-Induced Diabetes.Zinc in the Monoaminergic Theory of Depression: Its Relationship to Neural Plasticity.Zinc signaling in the hippocampus and its relation to pathogenesis of depression.Proposed glucocorticoid-mediated zinc signaling in the hippocampus.Nutrition, adult hippocampal neurogenesis and mental health.Limbic system pathologies associated with deficiencies and excesses of the trace elements iron, zinc, copper, and selenium.Synaptic Zn(2+) homeostasis and its significance.Developmental neuroplasticity and the origin of neurodegenerative diseases.Recent advances in the understanding of the role of zinc in ocular tissues.The Physiological, Biochemical, and Molecular Roles of Zinc Transporters in Zinc Homeostasis and Metabolism.Significance of the degree of synaptic Zn²⁺ signaling in cognition.Mapping the dynamics of cortical neuroplasticity of skilled motor learning using micro X-ray fluorescence and histofluorescence imaging of zinc in the rat.GPR39 receptors and actions of trace metals on pancreatic beta cell function and glucose homoeostasis.The Zn2+-sensing receptor, ZnR/GPR39, upregulates colonocytic Cl- absorption, via basolateral KCC1, and reduces fluid loss.Zinc deficiency regulates hippocampal gene expression and impairs neuronal differentiation.Zinc deficiency impairs the renewal of hippocampal neural stem cells in adult rats: involvement of FoxO3a activation and downstream p27(kip1) expression.The cancer chemotherapeutic agent paclitaxel (Taxol) reduces hippocampal neurogenesis via down-regulation of vesicular zinc.Zinc transporter ZnT3 is involved in memory dependent on the hippocampus and perirhinal cortex.Cellular Zinc Homeostasis Contributes to Neuronal Differentiation in Human Induced Pluripotent Stem Cells.A reduced zinc diet or zinc transporter 3 knockout attenuate light induced zinc accumulation and retinal degeneration.Oral zinc supplementation may improve cognitive function in schoolchildren.In vivo fluorescence sensing of the salicylate-induced change of zinc ion concentration in the auditory cortex of rat brain.Differential Effects of Low- and High-dose Zinc Supplementation on Synaptic Plasticity and Neurogenesis in the Hippocampus of Control and High-fat Diet-fed Mice.Neurobehavioral Deficits in a Rat Model of Recurrent Neonatal Seizures Are Prevented by a Ketogenic Diet and Correlate with Hippocampal Zinc/Lipid Transporter Signals.

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P2860

Decreased brain zinc availability reduces hippocampal neurogenesis in mice and rats.

http://www.wikidata.org/entity/Q45393764

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年學術文章

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2009年學術文章

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name

Decreased brain zinc availability reduces hippocampal neurogenesis in mice and rats.

@en

Decreased brain zinc availability reduces hippocampal neurogenesis in mice and rats.

@nl

type

label

Decreased brain zinc availability reduces hippocampal neurogenesis in mice and rats.

@en

Decreased brain zinc availability reduces hippocampal neurogenesis in mice and rats.

@nl

prefLabel

Decreased brain zinc availability reduces hippocampal neurogenesis in mice and rats.

@en

Decreased brain zinc availability reduces hippocampal neurogenesis in mice and rats.

@nl

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Journal of Cerebral Blood Flow & Metabolism

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Decreased brain zinc availability reduces hippocampal neurogenesis in mice and rats.

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P2093

Aaron M Hamby

http://www.w3.org/2001/XMLSchema#string

Atsushi Takeda

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Byung Hoon Yoo

http://www.w3.org/2001/XMLSchema#string

Christian T Sheline

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Haruna Tamano

http://www.w3.org/2001/XMLSchema#string

Jialing Liu

http://www.w3.org/2001/XMLSchema#string

Sang Won Suh

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Seok Joon Won

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Yang Fan

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P2860

ZnT-3, a putative transporter of zinc into synaptic vesicles

Elimination of zinc from synaptic vesicles in the intact mouse brain by disruption of the ZnT3 gene

The utility of Ki-67 and BrdU as proliferative markers of adult neurogenesis

Adult neurogenesis in the mammalian central nervous system

Zinc-specific autometallographic in vivo selenium methods: tracing of zinc-enriched (ZEN) terminals, ZEN pathways, and pools of zinc ions in a multitude of other ZEN cells.

Evidence that the ZNT3 protein controls the total amount of elemental zinc in synaptic vesicles.

Exogenous selenium in the brain. A histochemical technique for light and electron microscopical localization of catalytic selenium bonds.

Hypoglycemia, brain energetics, and hypoglycemic neuronal death.

Hypoglycemic neuronal death is triggered by glucose reperfusion and activation of neuronal NADPH oxidase.

Release of glutamate and GABA in the hippocampus under zinc deficiency.

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1579-1588

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10.1038/JCBFM.2009.80

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9

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29

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2009-06-17T00:00:00Z

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26301029

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19536073

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