n-butylidenephthalide treatment prolongs life span and attenuates motor neuron loss in SOD1G93A mouse model of amyotrophic lateral sclerosis.

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Role of Neuroinflammation in Amyotrophic Lateral Sclerosis: Cellular Mechanisms and Therapeutic Implications.Autophagy as a common pathway in amyotrophic lateral sclerosis.

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n-butylidenephthalide treatment prolongs life span and attenuates motor neuron loss in SOD1G93A mouse model of amyotrophic lateral sclerosis.

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http://www.wikidata.org/entity/Q48204504

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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name

n-butylidenephthalide treatmen ...... amyotrophic lateral sclerosis.

@en

n-butylidenephthalide treatmen ...... amyotrophic lateral sclerosis.

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type

Item

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n-butylidenephthalide treatmen ...... amyotrophic lateral sclerosis.

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n-butylidenephthalide treatmen ...... amyotrophic lateral sclerosis.

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n-butylidenephthalide treatmen ...... amyotrophic lateral sclerosis.

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n-butylidenephthalide treatmen ...... amyotrophic lateral sclerosis.

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P1476

n-butylidenephthalide treatmen ...... amyotrophic lateral sclerosis.

@en

P2093

Jing-Jing Zhang

http://www.w3.org/2001/XMLSchema#string

Qin-Ming Zhou

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P2860

n-butylidenephthalide protects against dopaminergic neuron degeneration and α-synuclein accumulation in Caenorhabditis elegans models of Parkinson's disease

The structure of a Bcl-xL/Bim fragment complex: implications for Bim function

The complex molecular biology of amyotrophic lateral sclerosis (ALS)

The roles of intracellular protein-degradation pathways in neurodegeneration

Riluzole prevents morphine-induced apoptosis in rat cerebral cortex

The natural compound n-butylidenephthalide derived from Angelica sinensis inhibits malignant brain tumor growth in vitro and in vivo

Neuronal death in amyotrophic lateral sclerosis is apoptosis: possible contribution of a programmed cell death mechanism.

MTOR-independent, autophagic enhancer trehalose prolongs motor neuron survival and ameliorates the autophagic flux defect in a mouse model of amyotrophic lateral sclerosis.

A review of the neural mechanisms of action and clinical efficiency of riluzole in treating amyotrophic lateral sclerosis: what have we learned in the last decade?

DL-3-n-butylphthalide extends survival by attenuating glial activation in a mouse model of amyotrophic lateral sclerosis.

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375-385

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scholarly article

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10.1111/CNS.12681

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http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Song Li

Shen Chen

Wei-Dong Le

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2017-02-22T00:00:00Z

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28229532

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amyotrophic lateral sclerosis

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6492644

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n-butylidenephthalide treatment prolongs life span and attenuates motor neuron loss in SOD1G93A mouse model of amyotrophic lateral sclerosis.

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P2860

P2860

n-butylidenephthalide treatment prolongs life span and attenuates motor neuron loss in SOD1G93A mouse model of amyotrophic lateral sclerosis.

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